Blood–brain barrier disruption and enhanced vascular permeability in the multiple sclerosis model EAE (original) (raw)

Blood-brain barrier disruption in multiple sclerosis

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Multiple Sclerosis, 2003

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Role of the Blood-Brain Barrier in Multiple Sclerosis

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The effects of blood–brain barrier disruption on glial cell function in multiple sclerosis

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Reviewing the Significance of Blood–Brain Barrier Disruption in Multiple Sclerosis Pathology and Treatment

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2021

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Abnormal Endothelial Tight Junctions in Active Lesions and Normal-appearing White Matter in Multiple Sclerosis

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Inflammation-induced endothelial to mesenchymal transition promotes brain endothelial cell dysfunction and occurs during multiple sclerosis pathophysiology

Helga De Vries

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Sera from Patients with Multiple Sclerosis in Relapse or Remission Affect the Blood-Brain Barrier Differently: An In vitro Study

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Tight junctional abnormality in multiple sclerosis white matter affects all calibres of vessel and is associated with blood–brain barrier leakage and active demyelination

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Emerging roles of endothelial cells in multiple sclerosis pathophysiology and therapy

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Blood-Brain Barrier Alterations in the Cerebral Cortex in Experimental Autoimmune Encephalomyelitis

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Sera from Remitting and Secondary Progressive Multiple Sclerosis Patients Disrupt the Blood-Brain Barrier

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Multiple Sclerosis and the Blood-Central Nervous System Barrier

Alan Palmer

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Contribution of Tissue Inflammation and Blood-Brain Barrier Disruption to Brain Softening in a Mouse Model of Multiple Sclerosis

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The interplay between T helper cells and brain barriers in the pathogenesis of multiple sclerosis

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Multiple sclerosis and cerebral endothelial dysfunction: Mechanisms

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Evidence of persistent blood-brain barrier abnormalities in chronic-progressive multiple sclerosis

Luz Claudio

Acta Neuropathologica, 1995

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Neuronal and BBB damage induced by sera from patients with secondary progressive multiple sclerosis

Giuseppe Salemi

International Journal of Molecular Medicine, 2009

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Focal disturbances in the blood-brain barrier are associated with formation of neuroinflammatory lesions

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Neurobiology of disease, 2014

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Blood–brain barrier permeability and monocyte infiltration in experimental allergic encephalomyelitis

I. Schadee-Eestermans

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Claudin-1 induced sealing of blood–brain barrier tight junctions ameliorates chronic experimental autoimmune encephalomyelitis

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Identification of an essential endogenous regulator of blood-brain barrier integrity, and its pathological and therapeutic implications

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Proceedings of the National Academy of Sciences, 2013

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Multiple sclerosis as a vascular disease

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Neurological Research, 2006

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Neurofilament levels are associated with blood–brain barrier integrity, lymphocyte extravasation, and risk factors following the first demyelinating event in multiple sclerosis

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Adhesion molecules on endothelial cells in the central nervous system: An emerging area in the neuroimmunology of multiple sclerosis

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Blood-brain barrier permeability and monocyte infiltration in experimental allergic encephalomyelitis: A quantitative MRI study

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Blood-Brain Barrier Disruption as a Key Role Prob-lem in Multiple Sclerosis: A Novel Primary Prevention Strategy

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Biomedical Journal of Scientific & Technical Research, 2020

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Sclerosis an Animal Model of Multiple Attenuates Clinical Signs of Disease in the Central Nervous System and Prevents Leukocyte Recruitment to Protein Prenyltransferase Inhibitors Inhibition of Rho GTPases with

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Inflammatory events at blood-brain barrier in neuroinflammatory and neurodegenerative disorders: Implications for clinical disease

Damir Janigro

Epilepsia, 2012

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Endothelial microparticles (EMP) bind and activate monocytes: elevated EMP-monocyte conjugates in multiple sclerosis

alireza minagar

Frontiers in Bioscience, 2004

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Serum from patients with multiple sclerosis downregulates occludin and VE-cadherin expression in cultured endothelial cells

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Multiple Sclerosis Journal, 2003

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In vivo relationship of interleukin-2 and soluble IL-2 receptor to blood-brain barrier impairment in patients with active multiple sclerosis

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