Forma paralitica de un caso de rabia humana trasmitida por murciélago, simulando Síndrome de Guillain Barre: Presentación de un caso de autopsia (original) (raw)
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Journal of Human Growth and Development, 2020
Introduction: Guillain-Barre Syndrome (GBS) is the most frequent cause of acute and sub-acute flaccid paralysis after polio eradication. Although rare, it is recognized as the leading cause of flaccid paralysis among the admissions to pediatric intensive care for acute neuromuscular diseases. Objective: To report the case of a 14-month-old male patient with a probable diagnosis of GBS with acute, myelinated motor sensitive neuropathy, with probable secondary axonal involvement, with rapid clinical recovery. Case Report: A male patient admitted in a reference hospital in the Federal District, Brazil, residing in the Integrated Development Region of the Federal District and Surroundings. The child was 14 months old and 8.6 kg, with an updated vaccination status and neuropsychomotor development appropriate for his age, with a condition of paresis in the lower limbs, without cognitive changes. After 14 hours of admission, due to the worsening of his clinical situation and the albumino-c...
Guillain-Barré Syndrome Masquerading as Brain Death
Bangladesh Critical Care Journal, 2013
Brain death is irreversible loss of function of the brain including the brain stem. Many clinical conditions may mimic brain death. This is a case review of a 27 year old Bangladeshi male who complaints heaviness of tongue and slurring of speech and was diagnosed as a case of bulbar palsy and found to develop ascending flaccid paralysis of limbs, subsequently he developed respiratory failure was placed on mechanical ventilator (MV) with GCS -3 and brain stem and all spinal reflexes were absent. The MRI & CSF studies were unremarkble. He was on MV and had no spontaneous breathing. A provisional diagnosis of GBS was made with a dfferential diagnosis of Brain stem death. Patient recieved 5 cycles of plasma pheresis without any clinical change. EEG showed cerebral reactivity on tactile stimulation and external noise stimulation. 31ist day of his illness patient developed involuntary tongue movement with pupils weakly reacting to light. 37th day of his illness he opened his eyes with voc...
GuillainâBarré syndrome mimicking brain death
Journal of The Peripheral Nervous System, 2009
Dear Editor, Fulminant Guillain-Barré syndrome (GBS) is a rapidly progressive variant of acute inflammatory polyradiculoneuropathy in which the patient develops flaccid quadriplegia and loss of brainstem function. This entity is very rare and the diagnosis can be challenging. We report the case of a 61-year-old woman and review the literature on this topic.
A unicenter, prospective study of Guillain‐Barré syndrome in Spain
Acta Neurologica Scandinavica, 2019
Objective: We report a prospective study analyzing clinical characteristics, sub-typing and prognosis in Guillain-Barré syndrome (GBS). Method: The study was based on consecutive GBS patients admitted between 2009 and 2017. Disability was serially assessed using the GBS disability scale. Results: Fifty-six GBS patients were identified with an average age of 55 years (range, 5-86 years) and a male/female ratio of 2.1. The interval to nadir was <7 days in 59% of cases, and 7 to 28 days in the remainder; at nadir, 35.5% of patients were able to walk unaided, and 64.5% did not. Mechanical ventilation was needed in 20% of cases. There were 2 fatal cases. Clinical variants included paraparetic GBS 7 cases, Miller Fisher syndrome 1 case, and acute sensory ataxic neuropathy (ASAN) 1 case. Serial electrophysiology showed a demyelinating pattern in 62.5% of cases, axonal in 28.5%, inexcitable in 1.8%, equivocal in 1.8%, and normal in 5.4%. Very early (1 to 4 days after onset) electrophysiology was done in 18 patients; equivocal or normal features in 6 of them evolved into an axonal pattern in 4. Reversible conduction failure of sensitive nerves occurred in ASAN. Antiganglioside antibodies were only detected in axonal GBS. At 24-month follow-up, functional outcome did not differ between demyelinating and axonal GBS. Clinico-pathological correlation in an early fatal case is reported.
Fulminant Guillain-Barre Syndrome Mimicking Clinical Brain Death: A Rare Condition With Bad Outcomes
Journal of Medical Cases, 2015
Guillain-Barre syndrome (GBS) is a disease of the peripheral nervous system characterized by acute areflexic paralysis and albuminocytological dissociation on cerebrospinal fluid (CSF). Fulminant GBS mimicking clinical brain death has been described in the literature and has overall poor prognosis since patients either die or are left with severe disability. We describe a case of fulminant GBS mimicking clinical brain death where patient died of unexplained asystolic cardiac arrest related to severe dysautonomia. Dysautonomia is a marker of poor prognosis in this patient population.
Guillain-Barré Syndrome (GBS)- A Review
2019
Guillain-Barré syndrome (GBS), a rare neurological disorder which attack self-immune system especially peripheral nervous system (PNS)and its network of nerves located outside of the brain and spinal cord. GBS has a number of recognized subtypes that have differing clinical and pathophysiological features. Most of the incidence rates of GBS reported was between 1.1/100,000/year and 1.8/100,000/year with lower rates reported in children (less than 16 years) of 0.4/100,000/year to 1.4/100,000/year. The reason for GBS though unknown but according to the Centre for Disease Control and Prevention (CDC), about two-thirds of people developed GBS post to gastrointestinal or a respiratory infection. Infectives include Campylobacter jejuni, Zika virus and herpes virus besides pesticides accumulation. Nerves innervating various muscles are affecting showing difficulty in response to the brain signals is one major symptom. Electrodiagnostic study and cerebrospinal liquid (CSF) estimations are t...
Guillain-Barré Syn drome: A Ret ro spec tive, Hos pi tal-based Study
G uillain-Barré syn drome (GBS), the most common cause of acute flac cid pa ral y sis since the elim i na tion of po lio my eli tis world wide, is char ac terized by acute as cend ing weak ness, areflexia and albuminocytologic dis so ci a tion of the cerebrospinal fluid (CSF). 1 The clin i cal fea tures of this acute as cending weak ness with out amyotrophy were de scribed by Lan dry in 1859. Albuminocytologic dis so ci a tion, how -ever, was not in tro duced un til 1916 by Guillain, Barré, and Strohl, 2 and then it be came known in ter na tion ally as Guillain-Barré syn drome.