Jun is phosphorylated by several protein kinases at the same sites that are modified in serum-stimulated fibroblasts (original) (raw)
The Activation and Activity Control of AP-1 (Fos/Jun)
A. Cato
Annals of the New York Academy of Sciences, 1993
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The c-fos protein interacts with c-JunAP-1 to stimulate transcription of AP-1 responsive genes
Tony Hunter
Cell, 1988
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A Direct Role for c-fos in AP-1-dependent Gene 1
Robert Distel
2015
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A Direct Role for c-fos in AP1-dependent Gene
Kevin Walton
1990
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Products of the fos and jun Proto-Oncogenes Bind Cooperatively to the AP1 DNA Recognition Sequence
M. Neuberg
Environmental Health Perspectives, 1990
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Positive regulation of jun/AP-1 by E1A
N. Foulkes
Molecular and cellular biology, 1991
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Modulation of the composition of AP-1 complex and its impact on transcriptional activity
B. Pyrzynska
Acta neurobiologiae experimentalis, 2000
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The repertoire of fos and jun proteins expressed during the G1 phase of the cell cycle is determined by the duration of mitogen-activated protein kinase activation
Simon J Cook
1999
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The product of a fos-related gene, fra-1, binds cooperatively to the AP-1 site with Jun: transcription factor AP-1 is comprised of multiple protein complexes
Reiner Gentz
1989
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The duration of ERK1/2 activity determines the activation of c-Fos and Fra-1 and the composition and quantitative transcriptional output of AP-1
Simon J Cook, K. Balmanno
Cellular Signalling, 2007
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Signalling to chromatin and the superinduction of proto-oncogenes
Nikolai Zhelev
Biochemical Society transactions, 1993
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Oncogenes, protein tyrosine kinases, and signal transduction
Lu-Hai Wang
Journal of Biomedical Science, 1994
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An oncogenic mutation uncouples the v-Jun oncoprotein from positive regulation by the SAPK/JNK pathway in vivo
James Woodgett
Current Biology, 1998
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Mechanisms of Oncogene Activation
Oana-Monica Popa
New Aspects in Molecular and Cellular Mechanisms of Human Carcinogenesis, 2016
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T1 is a c-Fos- and FosB-responsive gene which is induced by growth factors through multiple signal transduction pathways
Markus Kalousek
Journal of Biological Chemistry
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Repression of c- fos and c- jun gene expression is not part of AT 2 receptor coupled signal transduction
Monika Stoll
Journal of Molecular Medicine, 1998
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Biphasic increase in c-jun mRNA is required for induction of AP-1-mediated gene transcription: differential effects of muscarinic and thrombin receptor activation
j trejo
Molecular and Cellular Biology, 1992
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Altering the specificity of signal transduction cascades: positive regulation of c-Jun transcriptional activity by protein kinase A
Masahiko Hibi
The EMBO journal, 1994
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The defective transforming phenotype of c-Jun Ala63/73 is rescued by mutation of the C-terminal phosphorylation site
Irene Marchetti
Oncogene, 2001
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A gene activated by growth factors is related to the oncogene v-jun
Lester F Lau
Proceedings of the National Academy of Sciences, 1988
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Extracellular Signal-regulated Kinase 1/2-mediated Phosphorylation of JunD and FosB Is Required for Okadaic Acid-induced Activator Protein 1 Activation
Gerolf Gros
Journal of Biological Chemistry, 1999
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C-Fos Proto-Oncogene Regulation and Function
Jean-Marie Blanchard
Critical Reviews in Oncology/Hematology, 1994
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Presence of distinct AP-1 dimers in normal and transformed rat hepatocytes under basal conditions and after epidermal growth factor stimulation
Mohamed Rahmani
Hepatology, 1997
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Oncogenic Ras activates c-Jun via a separate pathway from the activation of extracellular signal-regulated kinases
Masahiko Hibi
Proceedings of the National Academy of Sciences, 1994
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V-Jun is a Transcriptional Activator, but Not in All Cell-Lines
elias ugarte
Nucleic Acids Research, 1988
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Common DNA binding site for Fos protein complexesand transcription factor AP-1
Robert Distel
Cell, 1988
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Opposing actions of Fos and Jun on transcription of the phosphoenolpyruvate carboxykinase (GTP) gene
Marta Giralt
Journal of Biological Chemistry
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Effects of the inhibition of p38/RK MAP kinase on induction of five fos and jun genes by diverse stimuli
Ana Cuenda
Oncogene, 1997
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Oncogenes and cell proliferation
Michelle Garrett
Current Opinion in Genetics & Development, 1999
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Deciphering AP-1 Function in Tumorigenesis: Fra-ternizing on Target Promoters
M. Yaniv
Cell Cycle, 2007
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AP-1 subunits: quarrel and harmony among siblings
Marina Schorpp-kistner
Journal of Cell Science, 2004
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