Innate Signaling in Otitis Media: Pathogenesis and Recovery (original) (raw)

Differences in innate immune response gene regulation in the middle ear of children who are otitis prone and in those not otitis prone

Ravinder Kaur

American journal of rhinology & allergy, 2016

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Role of innate immunity in the pathogenesis of otitis media

Kalai Mathee

International Journal of Infectious Diseases, 2014

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Cytokine, chemokine, and toll-like receptor expression in middle ear fluids of children with acute otitis media

Ravinder Kaur

The Laryngoscope, 2015

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The Toll-Like receptor adaptor TRIF contributes to otitis media pathogenesis and recovery

Michelle Hernandez

BMC Immunology, 2009

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Polymorphisms in Toll-like receptors 2 and 4 genes and their expression in chronic suppurative otitis media

Ana Jotic

Auris Nasus Larynx, 2015

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The role of DNA sensing and innate immune receptor TLR9 in otitis media

Michelle Hernandez

Innate Immunity, 2012

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Expression of toll-like receptors in chronic otitis media and cholesteatoma

Shin Kariya

International journal of pediatric otorhinolaryngology, 2013

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Expression of Toll-Like Receptors 2, 4 and Nuclear Factor Kappa B in Mucosal Lesions of Human Otitis: Pattern and Relationship in a Clinical Immunohistochemical Study

Ana Jotic

Annals of Otology, Rhinology & Laryngology, 2014

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Predisposition to Childhood Otitis Media and Genetic Polymorphisms within the Toll-Like Receptor 4 (TLR4) Locus

Eira Leinonen

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The Role of Inflammatory Mediators in the Pathogenesis of Otitis Media and Sequelae

Tina huang

Clinical and Experimental Otorhinolaryngology, 2008

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Panel 4: Recent Advances in Otitis Media in Molecular Biology, Biochemistry, Genetics, and Animal Models

Ann Hermansson

Otolaryngology -- Head and Neck Surgery, 2013

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The immunoregulatory and allergy-associated cytokines in the aetiology of the otitis media with effusion

jeffrey Pearson

Mediators of Inflammation, 2004

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Chronic suppurative otitis media causes macrophage-associated sensorineural hearing loss

brian bacacao

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Probing Immune-Mediated Clearance of Acute Middle Ear Infection in Mice

Longhuan Ma

Frontiers in Cellular and Infection Microbiology, 2022

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Genetic Polymorphisms in Immunoresponse GenesTNFA,IL6,IL10, andTLR4Are Associated With Recurrent Acute Otitis Media

L. Sanders

Pediatrics, 2007

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A model of chronic, transmissible Otitis Media in mice

Longhuan Ma

PLOS Pathogens, 2019

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High concentrations of middle ear antimicrobial peptides and proteins and proinflammatory cytokines are associated with detection of middle ear pathogens in children with recurrent acute otitis media

Shyan Vijayasekaran

PLOS ONE, 2019

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Niche- and Gender-Dependent Immune Reactions in Relation to the Microbiota Profile in Pediatric Patients with Otitis Media with Effusion

Fredrik Tjernström

Infection and Immunity, 2020

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Polymorphisms of Immunity Genes and Susceptibility to Otitis Media in Children

Reuben Matalon

PLoS ONE, 2014

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Toll-Like Receptor 2Dependent NF B Activation Is Involved in Nontypeable Haemophilus influenzae-Induced Monocyte Chemotactic Protein 1 Up-Regulation in the Spiral Ligament Fibrocytes of the Inner Ear

Jeong-Im Woo

Infection and Immunity, 2007

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NOD1/NOD2-mediated recognition of non-typeableHaemophilus influenzaeactivates innate immunity during otitis media

Jasmine Lee

Innate Immunity, 2019

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Evidence of functional cell-mediated immune responses to nontypeable Haemophilus influenzae in otitis-prone children

Dino Tan

PLOS ONE

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Role of Phagocytes in Antimicrobial Defence of the Middle Ear

Hideyuki Kawauchi

Auris Nasus Larynx, 1991

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New Paradigms in the Pathogenesis of Otitis Media in Children

James Coticchia

Frontiers in Pediatrics, 2013

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Mouse models for human otitis media

Qing Zheng

Brain Research, 2009

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Acute otitis media severity: Association with cytokine gene polymorphisms and other risk factors

Reuben Matalon

International Journal of Pediatric Otorhinolaryngology, 2011

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Antibodies Mediate Formation of Neutrophil Extracellular Traps in the Middle Ear and Facilitate Secondary Pneumococcal Otitis Media

Emma Job

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