Biliary Obstruction From a Bile Duct Mass (original) (raw)

Hepatoma presenting as extrahepatic biliary obstruction

The American Journal of Digestive Diseases, 1969

EPaTOMA may present in many unusual ways, inchtding fever of unknown origin? portal vein thrombosis,-" hypoglycemia, '~ erythyrocytosis,~ and hemorrhagic peritonitis, s Jaundice occasionally occurs and is usually due to the infiltrating hepatoma or to underlying cirrhosis, and less commonly to extension of the tumor into the major hepatic ducts. 6 s In the present patient, jaundice was caused by extrahepatic biliary obstruction, a mechanism rarely observed in patients with hepatoma. TM

An unexpected cause of jaundice

Journal of Paediatrics and Child Health, 2002

A 10-year-old girl presented with jaundice. She was born in Bosnia, but in 1992 escaped during the Bosnian war to a refugee camp in Austria for 3 years, where her father describes conditions as having been unhygienic. For the last 4 years they have been resident in Australia.

Unusual cause of cholestatic jaundice in a young immunocompetent male

Journal of Clinical and Experimental Hepatology, 2016

A 18-year-old boy with no previous comorbidities presented with jaundice, fever, and loss of weight and appetite of one-month duration. He had no previous history of long-standing fatigue, jaundice, ascites, upper gastrointestinal bleeding, or encephalopathy. Based on chest skiagram findings of nonhomogenous opacities in right upper lobe ([ 2 8 _ T D $ D I F F ] Figure 1; arrow), he was started on antitubercular therapy (ATT) in an outside medical facility. Following this[ 2 9 _ T D $ D I F F ] , he had worsening of jaundice and four weeks later developed progressively increasing abdominal distension[ 3 0 _ T D $ D I F F ]. On examination, he was icteric, and had pallor and firm palpable lymph nodes in cervical and axillary region along with firm hepatomegaly and ascites. Investigations revealed normochromic, normocytic anemia with [ 3 1 _ T D $ D I F F ] hemoglobin of 9.9 g/dl and elevated erythrocytic sedimentation rate of 55 in the first hour. The total leukocyte count [ 3 2 _ T D $ D I F F ] (17,600 cells/mm 3 [ 1 0 _ T D $ D I F F ]) was raised with an absolute eosinophil count of 5456 cells/mm 3 [ 3 3 _ T D $ D I F F ]. The platelet count was normal. Stool routine microscopy did not show any parasite on consecutive three stool samples. Acid[ 3 4 _ T D $ D I F F ]-fast bacilli (AFB) were not detected in the sputum examination[ 3 5 _ T D $ D I F F ]. Liver [ 2 _ T D $ D I F F ] Figure 2 CECT abdomen: enlarged liver that shows heterogenous attenuation, multiple hypodense lesions (white arrows[ 3 _ T D $ D I F F ]), and dilated intrahepatic biliary radicles (black arrows). Figure 1 Chest X-ray: nonhomogenous opacities in right upper lobe (arrows).