Nerve Growth Factor Inhibits Apoptosis in Memory B Lymphocytes via Inactivation of p38 MAPK, Prevention of Bcl-2 Phosphorylation, and Cytochrome c Release (original ) (raw )Nerve Growth Factor-dependent Survival of CESS B Cell Line Is Mediated by Increased Expression and Decreased Degradation of MAPK Phosphatase 1
Maria Gabriella Torcia
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Nerve Growth Factor Is an Autocrine Survival Factor for Memory B Lymphocytes
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Inhibition of nuclear factor kappa B (NFκB) activity induces nerve growth factor‐resistant apoptosis in PC12 cells
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Regulation of Bcl-2 Protein Expression in Human Neuroblastoma SH-SY5Y Cells: Positive and Negative Effects of Protein Kinases C and A, Respectively
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Nerve growth factor (NGF) exerts its pro-apoptotic effect via the P75NTR receptor in a cell cycle-dependent manner
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Nerve growth factor pretreatment attenuates oxygen and glucose deprivation-induced c-Jun amino-terminal kinase 1 and stress-activated kinases p38α and p38β activation and confers neuroprotection in the pheochromocytoma PC12 model
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Nerve growth factor promotes activation of the alpha, beta and gamma isoforms of protein kinase B in PC12 pheochromocytoma cells
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The Cyclic Adenosine Monophosphate-dependent Protein Kinase (PKA) Is Required for the Sustained Activation of Mitogen-activated Kinases and Gene Expression by Nerve Growth Factor
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NGF Withdrawal Induces Apoptosis in CESS B Cell Line through p38 MAPK Activation and Bcl-2 Phosphorylation
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Biochemical and Biophysical Research Communications, 2000
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Pro-apoptotic Bim Induction in Response to Nerve Growth Factor Deprivation Requires Simultaneous Activation of Three Different Death Signaling Pathways
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Mapping of Atypical Protein Kinase C within the Nerve Growth Factor Signaling Cascade: Relationship to Differentiation and Survival of PC12 Cells
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Nerve growth factor signaling through p75 induces apoptosis in Schwann cells via a Bcl-2-independent pathway
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The Journal of neuroscience : the official journal of the Society for Neuroscience, 1999
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Bcl-2 Phosphorylation by p38 MAPK IDENTIFICATION OF TARGET SITES AND BIOLOGIC CONSEQUENCES
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Low K+ Promotes NF B/DNA Binding in Neuronal Apoptosis Induced by K+ Loss
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Molecular and Cellular Biology, 2006
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δ-Protein Kinase C Phosphorylation Parallels Inhibition of Nerve Growth Factor– Induced Differentiation Independent of Changes in Trk A and MAP Kinase Signalling in PC12 Cells
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Nerve growth factor-mediated inhibition of apoptosis post-caspase activation is due to removal of active caspase-3 in a lysosome-dependent manner
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