OAZ Regulates Bone Morphogenetic Protein Signaling through Smad6 Activation (original) (raw)

Promoting bone morphogenetic protein signaling through negative regulation of inhibitory Smads

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The EMBO journal, 2001

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Protein associated with SMAD1 (PAWS1/FAM83G) is a substrate for type I bone morphogenetic protein receptors and modulates bone morphogenetic protein signalling

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Retinoic acid regulates bone morphogenic protein signal duration by promoting the degradation of phosphorylated Smad1

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Transforming growth factor β inhibits bone morphogenetic protein-induced transcription through novel phosphorylated Smad1/5-Smad3 complexes

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Dual Roles of Smad Proteins in the Conversion from Myoblasts to Osteoblastic Cells by Bone Morphogenetic Proteins

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2009

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Mouse Smad8 Phosphorylation Downstream of BMP Receptors ALK-2, ALK-3, and ALK-6 Induces Its Association with Smad4 and Transcriptional Activity

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SANE, a Novel LEM Domain Protein, Regulates Bone Morphogenetic Protein Signaling through Interaction with Smad1

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Physical and Functional Interaction of Murine and Xenopus Smad7 with Bone Morphogenetic Protein Receptors and Transforming Growth Factor-beta Receptors

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Smad6 is essential to limit BMP signaling during cartilage development

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Diverse Biological Effect and Smad Signaling of Bone Morphogenetic Protein 7 in Prostate Tumor Cells

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BMP canonical Smad signaling through Smad1 and Smad5 is required for endochondral bone formation

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Context-Dependent Proangiogenic Function of Bone Morphogenetic Protein Signaling Is Mediated by Disabled Homolog 2

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Autogenous regulation of a network of bone morphogenetic proteins (BMPs) mediates the osteogenic differentiation in murine marrow stromal cells

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CpG-Oligodeoxynucleotide Inhibits Smad-Dependent Bone Morphogenetic Protein Signaling: Effects on Myeloma Cell Apoptosis and In Vitro Osteoblastogenesis

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Inhibitory effects and target genes of bone morphogenetic protein 6 in Jurkat TAg cells

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Tob proteins enhance inhibitory Smad-receptor interactions to repress BMP signaling

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Smad8 is expressed in the anterior necrotic zone: Evidence for a role of bone morphogenetic proteins/SMAD signaling in the activation of a molecular cascade that culminates in cell death

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