Mitogen-Activated Protein Kinases in the Intensive Care Unit: Prognostic Potential (original) (raw)
Plasma receptor interacting protein kinase-3 levels are associated with acute respiratory distress syndrome in sepsis and trauma: a cohort study
Paul Lanken
Critical Care, 2019
View PDFchevron_right
Mitogen-Activated Protein Kinase Inhibition in Traumatic Brain Injury:In Vitro and In Vivo Effects
Elizabeth Fini
Journal of Cerebral Blood Flow & Metabolism, 2002
View PDFchevron_right
Adenosine monophosphate-activated protein kinase activation protects against sepsis-induced organ injury and inflammation
Hernando Gomez
Journal of Surgical Research, 2015
View PDFchevron_right
Thirty-Eight-Negative Kinase 1 Is a Mediator of Acute Kidney Injury in Experimental and Clinical Traumatic Hemorrhagic Shock
Milena Armacki
Frontiers in Immunology, 2020
View PDFchevron_right
Role of Mitogen- and Stress-Activated Kinases in Ischemic Injury
Elaine Irving
Journal of Cerebral Blood Flow & Metabolism, 2002
View PDFchevron_right
p38-alpha MAPK couples inflammatory response and proliferation of hepatocytes during acute liver injury
Jean-pierre Couty
Journal of Hepatology, 2017
View PDFchevron_right
Sustained Activation of p38 Mitogen-Activated Protein Kinase Contributes to the Vascular Response to Injury
Robert Willette
Journal of Pharmacology and Experimental Therapeutics, 2002
View PDFchevron_right
Inhibition of p38 Mitogen-Activated Protein Kinase Reduces Inflammation After Coronary Vascular Injury in Humans
Mark Hurle
Arteriosclerosis Thrombosis and Vascular Biology, 2010
View PDFchevron_right
Mitogen-Activated Protein Kinases Regulate Susceptibility to Ventilator-Induced Lung Injury
Tamas Dolinay
PLoS ONE, 2008
View PDFchevron_right
Lung, Spleen, and Kidney Are the Major Places for Inducible Nitric Oxide Synthase Expression in Endotoxic Shock: Role of P38 Mitogen-Activated Protein Kinase in Signal Transduction of Inducible Nitric Oxide Synthase Expression
Qiaobing Huang
Shock, 2004
View PDFchevron_right
Early Cytokine Production Risk Stratifies Trauma Patients for Multiple Organ Failure
Frederick A Moore
Journal of the American College of Surgeons, 2009
View PDFchevron_right
Inhibition of IκB Kinase Attenuates the Organ Injury and Dysfunction Associated With Hemorrhagic Shock
Karim Brohi
Molecular medicine (Cambridge, Mass.), 2015
View PDFchevron_right
Predictors of Multiple Organ Dysfunction Syndrome in Patients with Major Trauma
Orapan Thosingha
2017
View PDFchevron_right
Multiple organ dysfunction syndrome (MODS) prediction score in multi-trauma patients
Laurens Kalesaran
International Journal of Surgery Open, 2017
View PDFchevron_right
p38 Mitogen-Activated Protein Kinase-Dependent Chemokine Production, Leukocyte Recruitment, and Hepatocellular Apoptosis in Endotoxemic Liver Injury
Robert Roth
Annals of Surgery, 2005
View PDFchevron_right
Role of p38 mitogen-activated protein kinase pathway in estrogen-mediated cardioprotection following trauma-hemorrhage
Ya-Ching Hsieh
AJP: Heart and Circulatory Physiology, 2007
View PDFchevron_right
Pathophysiology of Acute Illness and Injury
Sergio Arlati
Operative Techniques and Recent Advances in Acute Care and Emergency Surgery, 2018
View PDFchevron_right
Mitogen Activated Protein Kinases in Steatotic and Non-Steatotic Livers Submitted to Ischemia-Reperfusion
Araní Casillas Ramírez
International Journal of Molecular Sciences, 2019
View PDFchevron_right
Signatures of inflammation and impending multiple organ dysfunction in the hyperacute phase of trauma: A prospective cohort study
Mimoza Hoti, Miederer I, Minal Patel, Claudia Cabrera, David Watson
PLOS Medicine, 2017
View PDFchevron_right
The Potential of Adipokines in Identifying Multiple Trauma Patients at Risk of Developing Multiple Organ Dysfunction Syndrome
Martijn van Griensven
Research Square (Research Square), 2020
View PDFchevron_right
CAN A P38???? KINASE INHIBITOR REVERSE ABERRANT MONOCYTE TO IMMATURE DENDRITIC CELL DIFFERENTIATION (M?????iDC) IN TRAUMA PATIENTS?
Carol Miller-graziano
Critical Care Medicine, 2005
View PDFchevron_right
Multiple Organ Dysfunction Syndrome (MODS): Is It Preventable or Inevitable?
Rifat Latifi, Ayman El-Menyar, Ruben Peralta
International Journal of Clinical Medicine, 2012
View PDFchevron_right
Role of p38 mitogen-activated protein kinase in cardiac myocyte secretion of the inflammatory cytokine TNF-α
Cherry Ballard-Croft
American Journal of Physiology-Heart and Circulatory Physiology, 2001
View PDFchevron_right
Aberrant Cellular Signaling in Multiple Organ Failure: Mechanism, Consequences and Therapeutic Applications
KOMAL SHARMA
Journal of Drug Delivery and Therapeutics
View PDFchevron_right
Early modifications in the expression of mitogen-activated protein kinase (MAPK/ERK), stress-activated kinases SAPK/JNK and p38, and their phosphorylated substrates following focal cerebral ischemia
Esther Dalfo
Acta neuropathologica, 2003
View PDFchevron_right
The role of MAP2 kinases and p38 kinase in acute murine liver injury models
Neil Kaplowitz
Cell death & disease, 2017
View PDFchevron_right
The stress-activated protein kinases are major c-Jun amino-terminal kinases activated by ischemia and reperfusion
James Woodgett
Journal of Biological Chemistry, 1994
View PDFchevron_right
SB 239063, a second-generation p38 mitogen-activated protein kinase inhibitor, reduces brain injury and neurological deficits in cerebral focal ischemia
S. Kassis
The Journal of pharmacology and experimental therapeutics, 2001
View PDFchevron_right
SB 239063, a second-generation p38 mitogen-activated protein kinase inhibitor, reduces brain injury and neurological deficits in cerebral focal ischemia. J Pharmacol Exp Ther
Frank Barone
Journal of Pharmacology and Experimental Therapeutics
View PDFchevron_right