Triple basepair changes within and adjacent to the conserved YY1 motif upstream of the U3 enhancer repeats of SL3-3 murine leukemia virus cause a small but significant shortening of latency of T-lymphoma induction (original) (raw)

An SL3-3 murine leukemia virus enhancer variant more pathogenic than the wild type obtained by assisted molecular evolution in vivo

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CBF, Myb, and Ets Binding Sites Are Important for Activity of the Core I Element of the Murine Retrovirus SL3-3 in T Lymphocytes

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Transcriptional initiation and postinitiation effects of murine leukemia virus long terminal repeat R-region sequences

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The Secondary Structure of the R Region of a Murine Leukemia Virus Is Important for Stimulation of Long Terminal Repeat-Driven Gene Expression

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Nuclear factors that bind to the enhancer region of nondefective Friend murine leukemia virus

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The Kissing-Loop Motif Is a Preferred Site of 59 Leader Recombination during Replication of SL3-3 Murine Leukemia Viruses in Mice

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Analysis of Wild-Type and Mutant SL3-3 Murine Leukemia Virus Insertions in the c-myc Promoter during Lymphomagenesis Reveals Target Site Hot Spots, Virus-Dependent Patterns, and Frequent Error-Prone Gap Repair

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Differences in activities of murine retroviral long terminal repeats in cytotoxic T lymphocytes and T-lymphoma cells

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Multistep regulation of enhancer activity of the 21-base-pair element of human T-cell leukemia virus type I

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Upstream Stimulatory Factors Binding to an E Box Motif in the R Region of the Bovine Leukemia Virus Long Terminal Repeat Stimulates Viral Gene Expression

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Suboptimal Enhancer Sequences Are Required for Efficient Bovine Leukemia Virus Propagation In Vivo: Implications for Viral Latency

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Enhancers Are Major Targets for Murine Leukemia Virus Vector Integration

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Murine Leukemia Retrovirus Integration Induces the Formation of Transcription Factor Complexes on Palindromic Sequences in the Signal Transducer and Activator of Transcription Factor 5a Gene During the Development of Pre–B Lymphomagenesis

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Sequences in the U5-gag-pol region influence early and late pathogenic effects of Friend and Moloney murine leukemia viruses

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Relative importance of elements within the SL3-3 virus enhancer for T-cell specificity

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Cis-Acting Sequences Responsible for the Transcriptional Activation of Human T-Cell Leukemia Virus Type I Constitute a Conditional Enhancer

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Thymotropism of murine leukemia virus is conferred by its long terminal repeat

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Substitution of leucine for isoleucine in a sequence highly conserved among retroviral envelope surface glycoproteins attenuates the lytic effect of the Friend murine leukemia virus

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