Triple basepair changes within and adjacent to the conserved YY1 motif upstream of the U3 enhancer repeats of SL3-3 murine leukemia virus cause a small but significant shortening of latency of T-lymphoma induction (original ) (raw )An SL3-3 murine leukemia virus enhancer variant more pathogenic than the wild type obtained by assisted molecular evolution in vivo
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Journal of Virology
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Upstream Conserved Sequences of Mouse Leukemia Viruses Are Important for High Transgene Expression in Lymphoid and Hematopoietic Cells
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Importance of a c-Myb binding site for lymphomagenesis by the retrovirus SL3-3
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Stability of AML1 (core) site enhancer mutations in T lymphomas induced by attenuated SL3-3 murine leukemia virus mutants
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Replication and pathogenicity of primer binding site mutants of SL3-3 murine leukemia viruses
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CBF, Myb, and Ets Binding Sites Are Important for Activity of the Core I Element of the Murine Retrovirus SL3-3 in T Lymphocytes
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Journal of Virology, 1998
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Transcriptional initiation and postinitiation effects of murine leukemia virus long terminal repeat R-region sequences
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The Secondary Structure of the R Region of a Murine Leukemia Virus Is Important for Stimulation of Long Terminal Repeat-Driven Gene Expression
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Journal of Virology, 1998
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Nancy Manley
Journal of Virology, 1989
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The Kissing-Loop Motif Is a Preferred Site of 59 Leader Recombination during Replication of SL3-3 Murine Leukemia Viruses in Mice
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U3 long terminal repeat-mediated induction of intracellular immunity by a murine retrovirus: a novel model of latency for retroviruses
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A transcriptional enhancer with specificity for erythroid cells is located in the long terminal repeat of the Friend murine leukemia virus
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Multistep regulation of enhancer activity of the 21-base-pair element of human T-cell leukemia virus type I
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Upstream Stimulatory Factors Binding to an E Box Motif in the R Region of the Bovine Leukemia Virus Long Terminal Repeat Stimulates Viral Gene Expression
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Suboptimal Enhancer Sequences Are Required for Efficient Bovine Leukemia Virus Propagation In Vivo: Implications for Viral Latency
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Introduction of a cis-acting mutation in the capsid-coding gene of moloney murine leukemia virus extends its leukemogenic properties
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Phenotypes of murine leukemia virus-induced tumors: influence of 3' viral coding sequences
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Enhancers Are Major Targets for Murine Leukemia Virus Vector Integration
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Murine Leukemia Retrovirus Integration Induces the Formation of Transcription Factor Complexes on Palindromic Sequences in the Signal Transducer and Activator of Transcription Factor 5a Gene During the Development of Pre–B Lymphomagenesis
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The American Journal of Pathology, 2011
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Sequences in the U5-gag-pol region influence early and late pathogenic effects of Friend and Moloney murine leukemia viruses
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Relative importance of elements within the SL3-3 virus enhancer for T-cell specificity
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Cis-Acting Sequences Responsible for the Transcriptional Activation of Human T-Cell Leukemia Virus Type I Constitute a Conditional Enhancer
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Thymotropism of murine leukemia virus is conferred by its long terminal repeat
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Substitution of leucine for isoleucine in a sequence highly conserved among retroviral envelope surface glycoproteins attenuates the lytic effect of the Friend murine leukemia virus
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