original ) (raw )Expression of FGF23 is correlated with serum phosphate level in isolated fibrous dysplasia
Yoshiki Nishizawa
Life Sciences, 2006
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Fibroblast Growth Factor 23: A Phosphatonin Regulating Phosphate Homeostasis?
Colin Dunstan
Endocrinology, 2004
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A new missense mutation in FGF23 gene in a male with hyperostosis–hyperphosphatemia syndrome (HHS)
Soudeh Ghafouri-Fard
Gene, 2014
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Is FGF23 the long sought after phosphaturic factor phosphatonin
Laurent Beck
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A Case of Hyperphosphatemia and Elevated Fibroblast Growth Factor 23: A Brief Review of Hyperphosphatemia and Fibroblast Growth Factor 23 Pathway
Virginia Vega-warner
Kidney International Reports
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Homozygous ablation of fibroblast growth factor-23 results in hyperphosphatemia and impaired skeletogenesis, and reverses hypophosphatemia in Phex-deficient mice
Reinhold Erben
Matrix Biology, 2004
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Molecular bases of diseases characterized by hypophosphatemia and phosphaturia: new understanding
Takehisa Yamamoto
Clinical pediatric endocrinology : case reports and clinical investigations : official journal of the Japanese Society for Pediatric Endocrinology, 2006
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Hyperphosphatemic tumoral calcinosis caused by FGF23 compound heterozygous mutations: what are the therapeutic options for a better control of phosphatemia?
Frédérique Dijoud
Pediatric Nephrology, 2018
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Interdisciplinary management of FGF23-related phosphate wasting syndromes: a Consensus Statement on the evaluation, diagnosis and care of patients with X-linked hypophosphataemia
Pol Harvengt
Nature Reviews Endocrinology
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Conditional Deletion of Murine Fgf23: Interruption of the Normal Skeletal Responses to Phosphate Challenge and Rescue of Genetic Hypophosphatemia
Julia Hum
Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research, 2016
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Oncogenic osteomalacia illustrating the effect of fibroblast growth factor 23 on phosphate homeostasis
Jaak Billen
Clinical kidney journal, 2012
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Renal Phosphate Wasting in Fibrous Dysplasia of Bone Is Part of a Generalized Renal Tubular Dysfunction Similar to That Seen in Tumor-Induced Osteomalacia
Shlomo Wientroub , Caroline Chebli
Journal of Bone and Mineral Research, 2001
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FGF23 and its role in X-linked hypophosphatemia-related morbidity
Dirk Schnabel
Orphanet Journal of Rare Diseases, 2019
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Long-term deficiency of the osteocytic hormone Fgf- 23 induces hyperparathyroidism, but not premature aging or altered insulin sensitivity in mice
Reinhold Erben
2011
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Genetic Evidence of Serum Phosphate-Independent Functions of FGF-23 on Bone
Reinhold Erben
PLoS Genetics, 2008
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Hypophosphatemia with Elevations in Serum Fibroblast Growth Factor 23 in a Child with Jansen’s Metaphyseal Chondrodysplasia
Craig Langman
The Journal of Clinical Endocrinology & Metabolism, 2009
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Identification of a novel loss-of-function PHEX mutation, Ala720Ser, in a sporadic case of adult-onset hypophosphatemic osteomalacia
William Fraser
Bone, 2018
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Regulation of renal phosphate transport by FGF23 is mediated by FGFR1 and FGFR4
Carlton Bates
American Journal of Physiology-Renal Physiology, 2013
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Severe vascular calcification and tumoral calcinosis in a family with hyperphosphatemia: a fibroblast growth factor 23 mutation identified by exome sequencing
Barbara Truitt
Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association, 2014
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The Autosomal Dominant Hypophosphatemic Rickets R176Q Mutation in Fibroblast Growth Factor 23 Resists Proteolytic Cleavage and Enhances in Vivo Biological Potency
Geeta Hampson
Journal of Biological Chemistry, 2003
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Hyperphosphatemia with low FGF7 and normal FGF23 and sFRP4 levels in the circulation characterizes pediatric hypophosphatasia
Deborah Wenkert
Bone, 2020
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Oncogenic osteomalacia: A reversible metabolic bone disorder
Dineshkumar Dhanwal
Indian Journal of Orthopaedics, 2010
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Acute effect of oral phosphate loading on serum fibroblast growth factor 23 levels in healthy men
Fumiaki Imamura
Kidney International, 2006
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The FGF23–Klotho axis: endocrine regulation of phosphate homeostasis
Mohammed Abdul Razzaque
Nature Reviews Endocrinology, 2009
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A G protein–coupled, IP3/protein kinase C pathway controlling the synthesis of phosphaturic hormone FGF23
Jordan Spatz
JCI Insight, 2019
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Hypophosphatemic rickets and osteomalacia
Durval Damiani , Luiz Claudio G. Castro
Arquivos Brasileiros de Endocrinologia & Metabologia, 2006
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Hyperostosis?Hyperphosphatemia Syndrome: A Congenital Disorder of O Glycosylation Associated With Augmented Processing of Fibroblast Growth Factor 23
Sofia Cohen
Journal of Bone and Mineral Research, 2007
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Osteocyte regulation of phosphate homeostasis and bone mineralization underlies the pathophysiology of the heritable disorders of rickets and osteomalacia
Baozhi Yuan
Bone, 2013
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Disordered Secretion of FGF23 Links Three Disorders of Phosphate Transport
gordon strewler
International Bone and Mineral Society Knowledge Environment, 2006
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Identifying the culprit lesion in tumor induced hypophosphatemia, the solution of a clinical enigma
Neveen Hamdy
Endocrine, 2016
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Fibroblast Growth Factor (FGF)-23 Inhibits Renal Phosphate Reabsorption by Activation of the Mitogen-activated Protein Kinase Pathway
Ken-Ichi Inui
Journal of Biological Chemistry, 2002
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Antibody-Mediated Activation of FGFR1 Induces FGF23 Production and Hypophosphatemia
Jose Zavala-solorio
PLoS ONE, 2013
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