Orthostatic tremor: diagnostic entity or variant of essential tremor? (original) (raw)

1989, Journal of Neurology, Neurosurgery & Psychiatry

SP, et al. earlier, over a week he developed severe Single photon emission computed tomograprogressive ataxia, complete external and phy in Alzheimer's disease. Arch Neurol internal ophthalmoplegia, the eyes remain-1988;45:392-6. ing in neutral position ptotic but without 13 Foster NL, Chase TN, Fedio P, et al Alz-dingin ane posiion ptotic butwitout heimer's disease: focal cortical changes diplopia, and general arreflexia. Consclousshown by positron emission tomography. ness was normal. CSF showed 0 cells, Neurology 1983;33:961-5. glucose 0-68 g/l and protein 0-63 g/l. In the 14 Taboeda E, Dickson D, Horoupian D, Davies next few days, transitory breathing and P. Clinicopathologic and neurochemical swallowing difficulties developed, as well as studies of one case of dysphasic dementia. J mild weakness of the facial musculature. In Neuropathol Exp Neurol 1986;45:323. this situation of complete ocular paralysis, 15 Morris JC, Cole M, Banker BQ, Wright D. the patient made constant gesticulation due Hereditary dysphasic dementia and the Pick-.. o Alzheimer spectrum. Ann Neurol to frequent, occasionallytsustained, bilateral 1984;16:455-66. blepharospasm attacks. This picture regres-16 Tissot R, Constantinidis J, Richard J. Picks sed to the previous situation after the ophdisease. In: Vinken PJ, Bruyn GW, Klawans thalmoplegia resolved some months later. HL, Fredriks JAM, eds. Handbook of Clin-The severe aggravation of facial spasms in ical Neurology Neurobehavioural disorders. our case was striking, well in excess of what Amsterdam, 1985;46:233-46. could have been expected from the emotional 17 Hudson AJ. Amyotrophic lateral sclerosis and stress of hospitalisation or appearance of