A radiosensitizing effect of RAD51 inhibition in glioblastoma stem-like cells (original) (raw)

Cell Cycle Changes after Glioblastoma Stem Cell Irradiation: The Major Role of RAD51

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International Journal of Molecular Sciences

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Glioma stem cells promote radioresistance by preferential activation of the DNA damage response

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HDAC4 and HDAC6 sustain DNA double strand break repair and stem-like phenotype by promoting radioresistance in glioblastoma cells

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Expression Levels of RAD51 Inversely Correlate with Survival of Glioblastoma Patients

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N6-Isopentenyladenosine Enhances the Radiosensitivity of Glioblastoma Cells by Inhibiting the Homologous Recombination Repair Protein RAD51 Expression

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Rad51 inhibition is an effective means of targeting DNA repair in glioma models and CD133+ tumor-derived cells

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HSV-1 amplicon-mediated post-transcriptional inhibition of Rad51 sensitizes human glioma cells to ionizing radiation

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Opposite Effects of the Triple Target (DNA-PK/PI3K/mTOR) Inhibitor PI-103 on the Radiation Sensitivity of Glioblastoma Cell Lines MO59K and MO59J Differing in DNA-PK and ATM Status

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DNA Damage Response Assessments in Human Tumor Samples Provide Functional Biomarkers of Radiosensitivity

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Acquired radioresistance in cancer associated fibroblasts is concomitant with enhanced antioxidant potential and DNA repair capacity

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