Role of Estradiol Receptor-α in Differential Expression of 2,3,7,8-Tetrachlorodibenzo-p-dioxin-Inducible Genes in the RL95-2 and KLE Human Endometrial Cancer Cell Lines (original ) (raw )Transcriptional suppression of estrogen receptor gene expression by 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD)
Michael Gallo
The Journal of Steroid Biochemistry and Molecular Biology, 1998
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Lack of antagonism of 2,3,7,8-tetrachlorodibenzo-p-dioxin's (TCDDs) induction of cytochrome P4501A1 (CYP1A1) by the putative selective aryl hydrocarbon receptor modulator 6-alkyl-1,3,8-trichlorodibenzofuran (6-MCDF) in the mouse hepatoma cell line Hepa-1c1c7
Adrian Fretland
Chemico-Biological Interactions, 2004
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Estradiol enhances and estriol inhibits the expression of CYP1A1 induced by 2,3,7,8-tetrachlorodibenzo-p-dioxin in a mouse ovarian cancer cell line
P. Terranova , Deok-soo Son
Toxicology, 2002
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Antiestrogenic Effects of 2,3,7,8-Tetrachlorodibenzo-p-dioxin Are Mediated by Direct Transcriptional Interference with the Liganded Estrogen Receptor
Paul Cooke
Journal of Biological Chemistry, 1996
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Antiestrogenic action of 2,3,7,8-tetrachlorodibenzo-p-dioxin: Tissue-specific regulation of estrogen receptor in CD1 mice
Michael Gallo
Toxicology and Applied Pharmacology, 1992
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Female Sprague—Dawley Rats Exposed to a Single Oral Dose of 2,3,7,8-Tetrachlorodibenzo- p -dioxin Exhibit Sustained Depletion of Aryl Hydrocarbon Receptor Protein in Liver, Spleen, Thymus, and Lung
Brian Necela
Toxicological Sciences, 1998
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Estrogen receptor α and aryl hydrocarbon receptor cross-talk in a transfected hepatoma cell line (HepG2) exposed to 2,3,7,8-tetrachlorodibenzo-p-dioxin
Marie-christine Chagnon
Toxicology Reports, 2014
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The aryl hydrocarbon receptor-mediated disruption of differentiation status of liver progenitor cells
Alois Kozubík
Toxicology Letters, 2011
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Cross-Talk between 2,3,7,8-Tetrachlorodibenzo-p-dioxin and Testosterone Signal Transduction Pathways in LNCaP Prostate Cancer Cells
Hideko Sone
Biochemical and Biophysical Research Communications, 1999
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Strain Differences in Cytochrome P4501A1 Gene Expression Caused by 2,3,7,8-Tetrachlorodibenzo-p-dioxin in the Rat Liver: Role of the Aryl Hydrocarbon Receptor and Its Nuclear Translocator
Hideko Sone
Biochemical and Biophysical Research Communications, 1998
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Antiestrogenic effects of 2, 3, 7, 8-tetrachlorodibenzo-p-dioxin in mouse uterus: critical role of the aryl hydrocarbon receptor in stromal tissue
Paul Cooke
Toxicological …, 2000
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Estrogen-responsive genes newly found to be modified by TCDD exposure in human cell lines and mouse systems
Hideko Sone
Molecular and cellular endocrinology, 2007
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2,3,7,8-Tetrachlorodibenzo-p-dioxin increases steady-state estrogen receptor-β mRNA levels after CYP1A1 and CYP1B1 induction in rat granulosa cells in vitro11Part of the work communicated in this paper was presented in the 82nd (Toronto, Canada) Annual Meetings of the Endocrine Society. All cDNA ...
Asok Dasmahapatra
Molecular and Cellular Endocrinology, 2001
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Inhibition of Prolactin Receptor Gene Expression by 2,3,7,8-Tetrachlorodibenzo-p-dioxin in MCF-7 Human Breast Cancer Cells
Xiaohong Wang
Archives of Biochemistry and Biophysics, 1996
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2,3,7,8-Tetrachlorodibenzo-p-dioxin causes an extensive alteration of 17 beta-estradiol metabolism in MCF-7 breast tumor cells
john gierthy
Proceedings of the National Academy of Sciences, 1990
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Aryl-hydrocarbon Receptor-Deficient Mice Are Resistant to 2,3,7,8-Tetrachlorodibenzo-p-dioxin-Induced Toxicity
Pedro Fernandez-salguero
Toxicology and Applied Pharmacology, 1996
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Effects of the environmental contaminants DEHP and TCDD on estradiol synthesis and aryl hydrocarbon receptor and peroxisome proliferator-activated receptor signalling in the human granulosa cell line KGN
Ronald Biemann
Molecular Human Reproduction, 2014
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Aryl Hydrocarbon Receptor-Mediated Gene Expression by Chlorinated Polycyclic Aromatic Hydrocarbons and Cross-Talk with Estrogen Receptors
Takeshi Ohura
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Effects of 2,3,7,8-tetrachlorodibenzo-p-dioxin and related compounds on the occupied nuclear estrogen receptor in MCF-7 human breast cancer cells
S. Safe
Cancer research, 1990
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Studies on the Relationship between Estrogen Receptor Content, GlutathioneS-Transferase π Expression, and Induction by 2,3,7,8-Tetrachlorodibenzo-p-dioxin and Drug Resistance in Human Breast Cancer Cells
Rola Barhoumi
Archives of Biochemistry and Biophysics, 1997
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Inhibition of estrogen-mediated uterine gene expression responses by dioxin
Kj Williams
Molecular Pharmacology, 2008
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In vivo up-regulation of aryl hydrocarbon receptor expression by 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) in a dioxin-resistant rat model
Raimo Pohjanvirta
Biochemical Pharmacology, 2001
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The aryl hydrocarbon receptor ligands 2,3,7,8-tetrachlorodibenzo-p-dioxin and 3-methylcholanthrene regulate distinct genetic networks
Ingemar Pongratz
Molecular and cellular endocrinology, 2012
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Estrogenic status modulates aryl hydrocarbon receptor--mediated hepatic gene expression and carcinogenicity
Martin Ronis
Carcinogenesis, 2008
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Estrogen and aryl hydrocarbon receptor expression and crosstalk in human Ishikawa endometrial cancer cells
ichen chen
The Journal of Steroid Biochemistry and Molecular Biology, 2000
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Activation of transcription factors activator protein-1 and nuclear factor-κB by 2,3,7,8-Tetrachlorodibenzo-p-dioxin
Howard Shertzer
Biochemical Pharmacology, 2000
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2,3,7,8-Tetrachlorodibenzo-p-dioxin Blocks Androgen-Dependent Cell Proliferation of LNCaP Cells through Modulation of pRB Phosphorylation
Alvaro Puga
Molecular Pharmacology, 2004
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Repression of Aryl Hydrocarbon Receptor Transcriptional Activity by Epidermal Growth Factor
O. Hankinson
Molecular Interventions, 2009
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Responsiveness of the Adult Male Rat Reproductive Tract to 2,3,7,8-Tetrachlorodibenzo-p-dioxin Exposure: Ah Receptor and ARNT Expression, CYP1A1 Induction, and Ah Receptor Down-Regulation
R. Pollenz
Toxicology and Applied Pharmacology, 1998
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Interactive effects of estradiol and 2,3,7,8-tetrachloro-dibenzo-p-dioxin on hepatic cytochrome P-450 and mouse uterus
Michael Gallo
Toxicology Letters, 1986
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The Transcription Factor Aryl Hydrocarbon Receptor Nuclear Translocator Functions as an Estrogen Receptor Selective Coactivator, and Its Recruitment to Alternative Pathways Mediates Antiestrogenic Effects of Dioxin
Ingemar Pongratz , Joëlle Rüegg
Molecular Endocrinology, 2007
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The Transcription Factor Aryl Hydrocarbon Receptor Nuclear Translocator Functions as an Estrogen Receptor β-Selective Coactivator, and Its Recruitment to Alternative Pathways Mediates Antiestrogenic Effects of Dioxin
Elin Swedenborg , Ingemar Pongratz , Joëlle Rüegg
Molecular Endocrinology, 2008
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The transcription factor aryl hydrocarbon receptor nuclear translocator functions as an estrogen receptor beta-selective coactivator, and its recruitment to alternative pathways mediates antiestrogenic effects of dioxin
Katarina Pettersson , Joëlle Rüegg , Ingemar Pongratz
Molecular endocrinology (Baltimore, Md.), 2008
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Structure-dependent induction of aryl hydrocarbon hydroxylase activity in C57BL6 mice by 2,3,7,8-tetrachlorodibenzo-p-dioxin and related congeners: Mechanistic studies
Jadwiga Piskorska-Pliszczynska
Toxicology and Applied Pharmacology, 1990
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Direct and indirect impact of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) on adult mouse Leydig cells: An in vitro study
Nathalie Montenegro Vega
Toxicology Letters, 2011
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