Activation of nuclear factor-κB by podocytes in the autologous phase of passive Heymann nephritis (original) (raw)
Urinary excretion of C5b-9 reflects disease activity in passive Heymann nephritis
William Couser
Kidney International, 1989
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Increased urinary excretion of C5b-9 distinguishes passive Heymann nephritis in the rat
William Couser
Kidney International, 1989
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Role of T cells in the mediation of Heymann nephritis. ii. Identification of Th1 and cytotoxic cells in glomeruli
Rochelle Boyd
Kidney International, 1997
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A podocyte view of membranous nephropathy: from Heymann nephritis to the childhood human disease
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Pflugers Archiv : European journal of physiology, 2017
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Glomerular complement regulation is overwhelmed in passive Heymann nephritis
Richard Quigg, Guohui Ren
Kidney International, 2001
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Identification of a pathogenic epitope involved in initiation of Heymann nephritis
Dontscho Kerjaschki
Proceedings of the National Academy of Sciences, 1992
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Production of a new model of slowly progressive Heymann nephritis
Rene Lafreniere
International Journal of Experimental Pathology, 2003
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IFN-Inducible Protein-10 Has a Differential Role in Podocyte during Thy 1.1 Glomerulonephritis
Hiroyuki Yoneyama
Journal of the American Society of Nephrology, 2003
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Down-regulation of pathogenic autoantibody response in a slowly progressive Heymann nephritis kidney disease model
Rene Lafreniere
International Journal of Experimental Pathology, 2004
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Glomerular basement membrane expansion in passive Heymann nephritis. Absence of increased synthesis of type IV collagen, laminin, or fibronectin
vijittra leardkamolkarn
The American journal of pathology, 1991
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Nuclear Factor-κB Inhibitors as Potential Novel Anti-Inflammatory Agents for the Treatment of Immune Glomerulonephritis
Jeffrey Kopp
The American Journal of Pathology, 2002
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Induction of B7-1 in podocytes is associated with nephrotic syndrome
Laura Giardino
Journal of Clinical Investigation, 2004
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In Vivo Administration of a Nuclear Transcription Factor-κB Decoy Suppresses Experimental Crescentic Glomerulonephritis
Robert Atkins
Journal of the American Society of Nephrology, 2000
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Limitation of podocyte proliferation improves renal function in experimental crescentic glomerulonephritis
Sian Griffin
Kidney International, 2005
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Regulation of Neph3 gene in podocytes – key roles of transcription factors NF-κB and Sp1
Moin Saleem, Gavin Welsh, Sanna Lehtonen
BMC Molecular Biology, 2009
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Active Heymann nephritis in complement component C6 deficient rats
Bruce Hall
Kidney International, 1995
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Resolution of renal inflammation: a new role for NF- B1 (p50) in inflammatory kidney diseases
DR. X.
AJP: Renal Physiology, 2009
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IL4 Therapy Prevents the Development of Proteinuria in Active Heymann Nephritis by Inhibition of Tc1 Cells1
Giang Tran
2001
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Regulation of Neph3 gene in podocytes � key roles of transcription factors NF-?B and Sp1
Sanna Lehtonen
Bmc Mol Biol, 2009
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An immunopathologic study of a 330-kD protein defined by monoclonal antibodies and reactive with anti-RTE alpha 5 antibodies and kidney eluates from active Heymann nephritis
P. Ronco
The Journal of Immunology
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IL-4 Therapy Prevents the Development of Proteinuria in Active Heymann Nephritis by Inhibition of Tc1 Cells
Rochelle Boyd
The Journal of Immunology, 2001
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Podocytes Populate Cellular Crescents in a Murine Model of Inflammatory Glomerulonephritis
Wilhelm Kriz
Journal of the American Society of Nephrology, 2004
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Inhibition of Complement Regulation Is Key to the Pathogenesis of Active Heymann Nephritis
Jessy Alexander
Journal of Experimental Medicine, 1998
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Activation of the IL-2 Receptor in Podocytes: A Potential Mechanism for Podocyte Injury in Idiopathic Nephrotic Syndrome?
Arnold Zea
PloS one, 2016
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Nephrotic Syndrome and Subepithelial Deposits in a Mouse Model of Immune-Mediated Anti-Podocyte Glomerulonephritis
Catherine Meyer-schwesinger
The Journal of Immunology, 2011
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Overexpression of toll-like receptor 9 correlates with podocyte injury in a murine model of autoimmune membranoproliferative glomerulonephritis
Md. Abdul Masum
Autoimmunity, 2018
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Basic fibroblast growth factor augments podocyte injury and induces glomerulosclerosis in rats with experimental membranous nephropathy
William Couser, Dontscho Kerjaschki
Journal of Clinical Investigation, 1995
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Fn14 in podocytes and proteinuric kidney disease
Maria D Sanchez-Nino
Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease, 2013
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Activation of podocytes by mesangial-derived TNF- : glomerulo-podocytic communication in IgA nephropathy
Fernand Lai
AJP: Renal Physiology, 2008
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Intrinsic proinflammatory signaling in podocytes contributes to podocyte damage and prolonged proteinuria
Heike Goebel
American Journal of Physiology-Renal Physiology, 2012
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NF-κB in Renal Inflammation
Juan Ortiz Moreno
Journal of the American Society of Nephrology, 2010
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