Bone Morphogenetic Protein 2 Activates Smad6 Gene Transcription through Bone-specific Transcription Factor Runx2 (original) (raw)

Loss of Smad3-Mediated Negative Regulation of Runx2 Activity Leads to an Alteration in Cell Fate Determination

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Tumor necrosis factor-α enhances the transcription of smad ubiquitination regulatory factor 1 in an activating protein-1- and runx2-dependent manner

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SMAD 8 binding to mice Msx1 basal promoter is required for transcriptional activation

Eliana S. F. W. Abdelhay

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Smad6 Represses Dlx3 Transcriptional Activity through Inhibition of DNA Binding

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CREB Cooperates with BMP-stimulated Smad signaling to enhance transcription of the Smad6 promoter

Edward Schwarz

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Runx2 Is a Common Target of Transforming Growth Factor β1 and Bone Morphogenetic Protein 2, and Cooperation between Runx2 and Smad5 Induces Osteoblast-Specific Gene Expression in the Pluripotent Mesenchymal Precursor Cell Line C2C12

Eung-Gook Kim

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SWI/SNF-Independent Nuclease Hypersensitivity and an Increased Level of Histone Acetylation at the PI Promoter Accompany Active Transcription of the Bone Master Gene Runx2

Berta Henriquez, Fernando Cruzat

Biochemistry, 2009

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SWI/SNF-Independent Nuclease Hypersensitivity and an Increased Level of Histone Acetylation at the P1 Promoter Accompany Active Transcription of the Bone Master Gene Runx2

Alejandro Villagra

Biochemistry, 2009

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Promoting bone morphogenetic protein signaling through negative regulation of inhibitory Smads

Carl-henrik Heldin

The EMBO journal, 2001

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Human Smad3 and Smad4 Are Sequence-Specific Transcription Activators

le Dai

Molecular Cell, 1998

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Bone Morphogenetic Protein-2 Stimulates Runx2 Acetylation

Mi-hye Lee

Journal of Biological Chemistry, 2006

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Regulation of Smad degradation and activity by Smurf2, an E3 ubiquitin ligase

Chenbei Chang

Proceedings of the National Academy of Sciences, 2001

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Targeted disruption in murine cells reveals variable requirement for Smad4 in transforming growth factor β-related signaling

Robert Maxson

2000

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Smad3 activates the Sox9-dependent transcription on chromatin

Takayuki Furumatsu

The International Journal of Biochemistry & Cell Biology, 2009

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Menin is required for bone morphogenetic protein 2- and transforming growth factor β-regulated osteoblastic differentiation through interaction with Smads and Runx2

Lucie Canaff

The Journal of Biological Chemistry, 2004

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Identification of a new human Smad6 splice variant

Lutz Konrad

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Smad proteins function as co-modulators for MEF2 transcriptional regulatory proteins

John McDermott

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Targeted Disruption in Murine Cells Reveals Variable Requirement for Smad4 in Transforming Growth Factor beta -related Signaling

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Smad3 and Smad4 Mediate Transcriptional Activation of the Human Smad7 Promoter by Transforming Growth Factor beta

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Transcriptional regulation of Smad2 is required for enhancement of TGF?/Smad signaling by TGF? inducible early gene

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Functions of mammalian Smad genes as revealed by targeted gene disruption in mice

Xiao Yang

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Regulation of the osteoblast-specific transcription factor, Runx2: Responsiveness to multiple signal transduction pathways

Sharon Dent

Journal of Cellular Biochemistry, 2003

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Smads oppose Hox transcriptional activities

Chenbei Chang

Experimental Cell Research, 2006

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Phosphorylation of Ser465 and Ser467 in the C Terminus of Smad2 Mediates Interaction with Smad4 and Is Required for Transforming Growth Factor-beta Signaling

Serhiy Souchelnytskyi

Journal of Biological Chemistry, 1997

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Identification of mZnf8, a mouse Krüppel-like transcriptional repressor, as a novel nuclear interaction partner of Smad1

Kai Jiao

Molecular and cellular biology, 2002

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Transforming growth factor β inhibits bone morphogenetic protein-induced transcription through novel phosphorylated Smad1/5-Smad3 complexes

Eva Grönroos

Molecular and cellular biology, 2012

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SMAD1/5 signaling in osteoclasts regulates bone formation via coupling factors

Kim Mansky

PLOS ONE, 2018

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Dominant-Negative SMAD-3 Interferes with Transcriptional Activation by Multiple Agonists

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