The Adaptor Protein AP-3 Is Required for CD1d-Mediated Antigen Presentation of Glycosphingolipids and Development of V 14i NKT Cells
Emanual Maverakis
Journal of Experimental Medicine, 2003
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Diverse Endogenous Antigens for Mouse NKT Cells: Self-Antigens That Are Not Glycosphingolipids
Mitchell Kronenberg
The Journal of Immunology, 2010
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Selective Ability of Mouse CD1 to Present Glycolipids: α-Galactosylceramide Specifically Stimulates Vα14+ NK T Lymphocytes
Kyoko Hayakawa
The Journal of Immunology, 1998
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Glycolipid/CD1d Complex Exhibits High-Affinity Binding to a The V{alpha}14 NKT Cell TCR
Nicholas Gascoigne
2000
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Cutting Edge: Structural Basis for the Recognition of -Linked Glycolipid Antigens by Invariant NKT Cells
Dirk Zajonc
The Journal of Immunology, 2011
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Selective Ability of Mouse CD1 to Present Glycolipids: a-Galactosylceramide Specifically Stimulates Va141 NK T Lymphocytes1
Masaru Taniguchi
The Journal of Immunology, 1998
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Lysosomal Localization of Murine CD1d Mediated by AP-3 Is Necessary for NK T Cell Development
Jenny Gumperz
The Journal of Immunology, 2003
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The T cell antigen receptor expressed by V 14i NKT cells has a unique mode of glycosphingolipid antigen recognition
Rachel Ndonye
Proceedings of the National Academy of Sciences, 2004
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V 2 natural killer T cell antigen receptor-mediated recognition of CD1d-glycolipid antigen
Melissa Honeydew
Proceedings of the National Academy of Sciences, 2011
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Glycolipid Antigen Drives Rapid Expansion and Sustained Cytokine Production by NK T Cells1
Rachael Keating
2003
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The Mouse CD1d Cytoplasmic Tail Mediates CD1d Trafficking and Antigen Presentation by Adaptor Protein 3-Dependent and -Independent Mechanisms
Oddmund Bakke
The Journal of Immunology, 2005
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The Role of Adaptor Proteins in the Biology of Natural Killer T (NKT) Cells
Jochen Mattner
Frontiers in Immunology, 2019
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Vβ2 natural killer T cell antigen receptor-mediated recognition of CD1d-glycolipid antigen
T. Mallevaey
Proceedings of the National Academy of Sciences, 2011
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A semi-invariant Vα10+ T cell antigen receptor defines a population of natural killer T cells with distinct glycolipid antigen–recognition properties
Mark Smyth
Nature Immunology, 2011
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Recycling CD1d1 Molecules Present Endogenous Antigens Processed in an Endocytic Compartment to NKT Cells
Kyoko Hayakawa, Tonya Webb
The Journal of Immunology, 2002
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Selective ability of mouse CD1 to present glycolipids: alpha-galactosylceramide specifically stimulates V alpha 14+ NK T lymphocytes
Patrick Mitchell, Kyoko Hayakawa
Journal of immunology (Baltimore, Md. : 1950), 1998
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A structural basis for selection and cross-species reactivity of the semi-invariant NKT cell receptor in CD1d/glycolipid recognition
James McCluskey
Journal of Experimental Medicine, 2006
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Crucial amino acid residues of mouse CD1d for glycolipid ligand presentation to Vα14 NKT cells
Mitchell Kronenberg
International Immunology, 2001
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Germline-encoded recognition of diverse glycolipids by natural killer T cells
Natalie Borg
Nature Immunology, 2007
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The response of natural killer T cells to glycolipid antigens is characterized by surface receptor down-modulation and expansion
Mi-young Son, Sebastian Joyce
Proceedings of the National Academy of Sciences, 2003
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Crucial amino acid residues of mouse CD1d for glycolipid ligand presentation to Valpha14 NKT cells
Masaru Taniguchi
International Immunology, 2001
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Glycolipid Antigen Drives Rapid Expansion and Sustained Cytokine Production by NK T Cells
Mark Smyth
The Journal of Immunology, 2003
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Cutting Edge: Structural Basis for the Recognition of β-Linked Glycolipid Antigens by Invariant NKT Cells
Dirk Zajonc
The Journal of Immunology, 2011
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Glycolipid presentation to natural killer T cells differs in an organ-dependent fashion
Moriya Tsuji
Proceedings of the National Academy of Sciences, 2005
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Interplay between carbohydrate and lipid in recognition of glycolipid antigens by natural killer T cells
Dirk Zajonc
2012
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The T cell antigen receptor expressed by Vα14iNKT cells has a unique mode of glycosphingolipid antigen recognition
Amy Howell
Proceedings of the National Academy of Sciences, 2004
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