Long-Lasting Synaptic Potentiation Induced by Depolarization Under Conditions That Eliminate Detectable Ca2+ Signals (original) (raw)
Persistent Ca2+Current Contributes to a Prolonged Depolarization inAplysiaBag Cell Neurons
Chris Groten
Journal of Neurophysiology, 2009
View PDFchevron_right
Persistent Ca2+Current Contributes to a Prolonged Depolarization inAplysiaBag Cell Neurons
Chris Groten
Journal of Neurophysiology, 2009
View PDFchevron_right
Ca2+ and synaptic plasticity
graham collingridge
Cell Calcium, 2005
View PDFchevron_right
Enhancement of Synaptic Plasticity through Chronically Reduced Ca2+ Flux during Uncorrelated Activity
Inna Slutsky, Guosong Liu
Neuron, 2004
View PDFchevron_right
Memory-Like Alterations in Aplysia Axons after Nerve Injury or Localized Depolarization
Edgar T Walters
Journal of Neuroscience, 2004
View PDFchevron_right
Synaptic lability after experience-dependent plasticity is not mediated by calcium-permeable AMPARs
Jing Wen
View PDFchevron_right
Retrograde signalling in depolarization-induced suppression of inhibition in rat hippocampal CA1 cells
Bradley Alger
The Journal of Physiology, 1996
View PDFchevron_right
Ca2+-induced Ca2+ release mediates a slow post-spike hyperpolarization in rabbit vagal afferent neurons
Joseph Kao
Journal of neurophysiology, 1998
View PDFchevron_right
Enhancement of sensorimotor connections by conditioning-related stimulation in Aplysia depends upon postsynaptic Ca2+
David Glanzman
Proceedings of the National Academy of Sciences, 1996
View PDFchevron_right
Long-term synaptic facilitation with minimal calcium entry
Lauren Swenarchuk
Brain Research, 1975
View PDFchevron_right
Voltage-sensitive Ca2+ channels in rat striatal synaptosomes : Role on the [Ca2+]i responses to membrane depolarization
Caetana Carvalho
Neurochemistry International, 1996
View PDFchevron_right
Spatiotemporal Distribution of Ca 2+ Following Axotomy and Throughout the Recovery Process of Cultured Aplysia Neurons
Noam Ziv
European Journal of Neuroscience, 1993
View PDFchevron_right
Glutamatergically induced pattern of Ca2+ driving potential as a mechanism of postsynaptic plasticity
L. Savtchenko
Biophysical Journal, 1997
View PDFchevron_right
Effects of membrane depolarization on intracellular calcium in single nerve terminals
Edward Stuenkel
Brain Research, 1990
View PDFchevron_right
Inactivation of acetylcholine release from Torpedo synaptosomes in response to prolonged depolarizations
Francois-Marie Meunier
The Journal of Physiology, 1985
View PDFchevron_right
Ca2+ signalling in postsynaptic dendrites and spines of mammalian neurons in brain slice
John Connor
Journal of Physiology-Paris, 1992
View PDFchevron_right
Development of depolarization-induced calcium transients in insect glial cells is dependent on the presence of afferent axons
Eric S . Tucker
Journal of Neurobiology, 2002
View PDFchevron_right
Depolarization-induced Ca
ALAN D GRINNELL
2010
View PDFchevron_right
Differential time-course of slow afterhyperpolarizations and associated Ca2+ transients in rat CA1 pyramidal neurons: further dissociation by Ca2+ buffer
Peter Pennefather
Neuroscience, 1999
View PDFchevron_right
Conditions Restricting Depolarization-Dependent Calcium Influx in Synaptosomes Reveal a Graded Response of P96 Dephosphorylation and a Transient Dephosphorylation of P65
Pablo Blanco
Journal of Neurochemistry, 1991
View PDFchevron_right
Depolarization-Induced Ca2+ Entry Preferentially Evokes Release of Large Quanta in the Developing Xenopus Neuromuscular Junction
ALAN D GRINNELL
Journal of Neurophysiology, 2010
View PDFchevron_right
Postsynaptic depolarisation enhances transmitter release and causes the appearance of responses at “silent” synapses in rat hippocampus
L. Savtchenko
Neuroscience, 2004
View PDFchevron_right
Increased neuronal excitability during depolarization-induced suppression of inhibition in rat hippocampus
Bradley Alger
The Journal of Physiology, 1996
View PDFchevron_right
Calcium-activated inward spike after-currents in bursting neurone R15 of Aplysia
Darrell Lewis
The Journal of Physiology, 1988
View PDFchevron_right
Depolarization, intracellular calcium and exocytosis in single vertebrate nerve endings
Edward Stuenkel
Biophysical Journal, 1992
View PDFchevron_right
Ca2+ Syntillas, Miniature Ca2+ Release Events in Terminals of Hypothalamic Neurons, Are Increased in Frequency by Depolarization in the Absence of Ca2+ Influx
Kevin E Fogarty
Journal of Neuroscience, 2004
View PDFchevron_right
Presynaptic plasticity: The regulation of Ca2+-dependent transmitter release
Wim E. J. M. Ghijsen
Progress in Neurobiology, 1994
View PDFchevron_right
Synaptic Facilitation and Behavioral Dishabituation in Aplysia: Dependence on Release of Ca2+ from Postsynaptic Intracellular Stores, Postsynaptic Exocytosis, and Modulation of Postsynaptic AMPA Receptor Efficacy
David Glanzman
Journal of Neuroscience, 2005
View PDFchevron_right
Relationship between Membrane Depolarization and Intracellular Free Calcium in Individual Nerve Terminals from the Neurohypophysis
Edward Stuenkel
Annals of the New York Academy of Sciences, 1991
View PDFchevron_right
Quantitative analysis of depolarization-induced ATP release from mouse brain synaptosomes: External calcium dependent and independent processes
Jenny Fiedler
The Journal of Membrane Biology, 1992
View PDFchevron_right
Serotonin Increases Intracellular Ca2+ Transients in Voltage-Clamped Sensory Neurons of Aplysia californica
Marc Klein
Proceedings of the National Academy of Sciences of the United States of America, 1984
View PDFchevron_right
Calcium Channels and Short-term Synaptic Plasticity
karina leal|
Journal of Biological Chemistry, 2013
View PDFchevron_right
Slow depolarizing afterpotentials in neocortical neurons are sodium and calcium dependent
Alon Friedman
Neuroscience Letters, 1992
View PDFchevron_right
Calcium signalling and synaptic plasticity at single hippocampal synapses
A. Fine
View PDFchevron_right
The Number of Components of Enhancement Contributing to Short-Term Synaptic Plasticity at the Neuromuscular Synapse during Patterned Nerve Stimulation Progressively Decreases As Basal Release Probability Is Increased from Low to Normal Levels by Changing Extracellular Ca2+
K. L Magleby
Journal of Neuroscience, 2011
View PDFchevron_right