Lipid Metabolism in Glioblastoma: From De Novo Synthesis to Storage (original) (raw)

Targeting Oncogenic Rewiring of Lipid Metabolism for Glioblastoma Treatment

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Inhibition of SOAT1 suppresses glioblastoma growth via blocking SREBP-1-mediated lipogenesis

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Targeting Cholesterol Metabolism in Glioblastoma: A New Therapeutic Approach in Cancer Therapy

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Sterol regulatory element binding protein-dependent regulation of lipid synthesis supports cell survival and tumor growth

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Cholesterol Derivatives as Promising Anticancer Agents in Glioblastoma Metabolic Therapy

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Lipid metabolism reprogramming and its potential targets in cancer

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GPIHBP1 expression in gliomas promotes utilization of lipoprotein-derived nutrients

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Fatty acid oxidation is required for the respiration and proliferation of malignant glioma cells

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Lipid droplets and ferroptosis as new players in brain cancer glioblastoma progression and therapeutic resistance

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Glioblastoma Cells Counteract PARP Inhibition through Pro-Survival Induction of Lipid Droplets Synthesis and Utilization

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Site-1 protease, a novel metabolic target for glioblastoma

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The Aurora Kinase Inhibitor TAK901 Inhibits Glioblastoma Growth by Blocking SREBP1-Mediated Lipid Metabolism

wenyong Long

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GBM tumors are heterogeneous in their fatty acid metabolism and modulating fatty acid metabolism sensitizes cancer cells derived from recurring GBM tumors to temozolomide

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Brain Fatty Acid-binding Protein and -3/ -6 Fatty Acids: MECHANISTIC INSIGHT INTO MALIGNANT GLIOMA CELL MIGRATION

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Investigating the Potential of Lipids for Use as Biomarkers for Glioblastoma via an Untargeted Lipidomics Approach

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Protect Cancer Cells from Lipotoxicity An Essential Requirement for the SCAP / SREBP Signaling Axis to

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Infiltrative and drug-resistant slow-cycling cells support metabolic heterogeneity in glioblastoma

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An Essential Requirement for the SCAP/SREBP Signaling Axis to Protect Cancer Cells from Lipotoxicity

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Stearoyl CoA Desaturase Is Essential for Regulation of Endoplasmic Reticulum Homeostasis and Tumor Growth in Glioblastoma Cancer Stem Cells

Litia Carvalho

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Selective decrease of the viability and the sterol content of proliferating versus glioma cells exposed to 25-hydroxycholesterol

William Maltese

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Lipidized glioblastoma: Pathological and molecular characteristics

Mircea Iftinca, Amitabh Singh

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Target identification reveals lanosterol synthase as a vulnerability in glioma

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7β-Hydroxycholesterol-induced energy stress leads to sequential opposing signaling responses and to death of c6 glioblastoma cells

Mathilde Schindler, Marcel Mersel

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Could Metabolic Therapy Become a Viable Alternative to the Standard of Care for Managing Glioblastoma?

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Metabolic state of glioma stem cells and nontumorigenic cells

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Lipid Alterations in Glioma: A Systematic Review

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Glucose-Mediated N-glycosylation of SCAP Is Essential for SREBP-1 Activation and Tumor Growth

Chunming Cheng

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7β-Hydroxycholesterol Blocked at C-3-OH Inhibits Growth of Rat Glioblastoma In Vivo: Comparison between 7β-Hydroxycholesteryl-3β (ester)-oleate and 7β- …

Michel Desgeorges

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Lipid Droplets in Cancer: From Composition and Role to Imaging and Therapeutics

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Lipid droplets: platforms with multiple functions in cancer hallmarks

andre cruz

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EGFR Signaling Through an Akt-SREBP-1-Dependent, Rapamycin-Resistant Pathway Sensitizes Glioblastomas to Antilipogenic Therapy

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Fatty acid synthase is a metabolic oncogene targetable in malignant peripheral nerve sheath tumors

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Aberrant lipid metabolism as an emerging therapeutic strategy to target cancer stem cells

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