Postmortem evidence of structural brain changes in schizophrenia. Differences in brain weight, temporal horn area, and parahippocampal gyrus compared with affective disorder - PubMed (original) (raw)
Postmortem evidence of structural brain changes in schizophrenia. Differences in brain weight, temporal horn area, and parahippocampal gyrus compared with affective disorder
R Brown et al. Arch Gen Psychiatry. 1986 Jan.
Abstract
The brains of 232 patients with a case-note diagnosis of schizophrenia or affective disorder who died in one mental hospital over a period of 22 years were weighed, and were assessed in a coronal section at the level of the interventricular foramina. From this sample were eliminated the brains of patients whose illnesses did not meet the Washington University criteria for a diagnosis of definite schizophrenia or primary affective disorder and those brains that showed significant histopathologic evidence of Alzheimer's-type change or cerebrovascular disease. This left a sample of 41 patients with schizophrenia and 29 patients with affective disorder. With age, sex, and year of birth controlled for, the brains of the patients with schizophrenia were 6% lighter, had lateral ventricles that were larger in the anterior (by 19%), and particularly in the temporal, (by 97%) horn cross section, and had significantly thinner parahippocampal cortices (by 11%). The findings provide postmortem confirmation of reports of ventricular enlargement in radiological studies and suggest that such enlargement is associated with tissue loss in the temporal lobe. The changes in schizophrenia were of a lesser degree than those seen in a sample of brains of patients with Alzheimer's-type dementia and Huntington's chorea.
Similar articles
- Temporal horn enlargement is present in schizophrenia and bipolar disorder.
Roy PD, Zipursky RB, Saint-Cyr JA, Bury A, Langevin R, Seeman MV. Roy PD, et al. Biol Psychiatry. 1998 Sep 15;44(6):418-22. doi: 10.1016/s0006-3223(98)00105-x. Biol Psychiatry. 1998. PMID: 9777171 Clinical Trial. - Schizophrenia as an anomaly of development of cerebral asymmetry. A postmortem study and a proposal concerning the genetic basis of the disease.
Crow TJ, Ball J, Bloom SR, Brown R, Bruton CJ, Colter N, Frith CD, Johnstone EC, Owens DG, Roberts GW. Crow TJ, et al. Arch Gen Psychiatry. 1989 Dec;46(12):1145-50. doi: 10.1001/archpsyc.1989.01810120087013. Arch Gen Psychiatry. 1989. PMID: 2589928 - Temporal lobe structure as determined by nuclear magnetic resonance in schizophrenia and bipolar affective disorder.
Johnstone EC, Owens DG, Crow TJ, Frith CD, Alexandropolis K, Bydder G, Colter N. Johnstone EC, et al. J Neurol Neurosurg Psychiatry. 1989 Jun;52(6):736-41. doi: 10.1136/jnnp.52.6.736. J Neurol Neurosurg Psychiatry. 1989. PMID: 2746266 Free PMC article. - The relationship between structural brain imaging and histopathologic findings in schizophrenia research.
Benes FM. Benes FM. Harv Rev Psychiatry. 1993 Jul-Aug;1(2):100-9. doi: 10.3109/10673229309017065. Harv Rev Psychiatry. 1993. PMID: 9384836 Review. - The continuum of psychosis and its genetic origins. The sixty-fifth Maudsley lecture.
Crow TJ. Crow TJ. Br J Psychiatry. 1990 Jun;156:788-97. doi: 10.1192/bjp.156.6.788. Br J Psychiatry. 1990. PMID: 2207509 Review.
Cited by
- N-methyl-D-aspartate receptor hypofunction as a potential contributor to the progression and manifestation of many neurological disorders.
Dong B, Yue Y, Dong H, Wang Y. Dong B, et al. Front Mol Neurosci. 2023 Jun 15;16:1174738. doi: 10.3389/fnmol.2023.1174738. eCollection 2023. Front Mol Neurosci. 2023. PMID: 37396784 Free PMC article. Review. - The synaptic hypothesis of schizophrenia version III: a master mechanism.
Howes OD, Onwordi EC. Howes OD, et al. Mol Psychiatry. 2023 May;28(5):1843-1856. doi: 10.1038/s41380-023-02043-w. Epub 2023 Apr 11. Mol Psychiatry. 2023. PMID: 37041418 Free PMC article. Review. - Gyral peaks and patterns in human brains.
Zhang S, Zhang T, He Z, Li X, Zhang L, Zhu D, Jiang X, Liu T, Han J, Guo L. Zhang S, et al. Cereb Cortex. 2023 May 24;33(11):6708-6722. doi: 10.1093/cercor/bhac537. Cereb Cortex. 2023. PMID: 36646465 Free PMC article. - The neurochemical pathology of schizophrenia: post-mortem studies from dopamine to parvalbumin.
Reynolds GP. Reynolds GP. J Neural Transm (Vienna). 2022 Jun;129(5-6):643-647. doi: 10.1007/s00702-021-02453-6. Epub 2021 Dec 21. J Neural Transm (Vienna). 2022. PMID: 34935080 Free PMC article. Review. - The Perspectives of Early Diagnosis of Schizophrenia Through the Detection of Epigenomics-Based Biomarkers in iPSC-Derived Neurons.
Lee D, Seo J, Jeong HC, Lee H, Lee SB. Lee D, et al. Front Mol Neurosci. 2021 Nov 12;14:756613. doi: 10.3389/fnmol.2021.756613. eCollection 2021. Front Mol Neurosci. 2021. PMID: 34867186 Free PMC article.
MeSH terms
LinkOut - more resources
Full Text Sources
Medical