Microsized inflammaging protects stem cells - PubMed (original) (raw)
Comment
Microsized inflammaging protects stem cells
Ryan M O'Connell et al. Blood. 2020.
Abstract
In this issue of Blood, Grants et al have identified connections between reduced expression of microRNA-146a (miR-146a), increased inflammation, impaired hematopoietic stem cell (HSC) quiescence, and a poor prognosis for acute myeloid leukemia (AML). miR-146a was originally identified as an anti-inflammatory microRNA that targets signaling proteins, which mediate inflammatory responses., Deletion of miR-146a in mice phenocopies many aspects of age-dependent inflammation,, termed “inflammaging.”
Conflict of interest statement
Conflict-of-interest disclosure: The authors declare no competing financial interests.
Conflict-of-interest disclosure: The authors declare no competing financial interests.
Figures
Proposed model: as a consequence of inflammaging, miR-146a levels are reduced, and inflammatory cytokine production is increased in hematopoietic cells, including macrophages. Consequently, these cytokines promote HSC proliferation and differentiation into stem cells with a myeloid and, potentially, leukemic bias. This state contributes to malignant transformation by some cell clones. Upon transformation to AML, miR-146a expression negatively correlates with prognosis.
Comment on
- Altered microRNA expression links IL6 and TNF-induced inflammaging with myeloid malignancy in humans and mice.
Grants JM, Wegrzyn J, Hui T, O'Neill K, Shadbolt M, Knapp DJHF, Parker J, Deng Y, Gopal A, Docking TR, Fuller M, Li J, Boldin M, Eaves CJ, Hirst M, Karsan A. Grants JM, et al. Blood. 2020 Jun 18;135(25):2235-2251. doi: 10.1182/blood.2019003105. Blood. 2020. PMID: 32384151
References
- Grants J, Wegrzyn J, Hui T, et al. Altered microRNA expression links IL6 and TNF-induced inflammaging with myeloid malignancy in humans and mice. Blood. 2020;135(25):2235-2251. -PubMed
- Starczynowski DT, Kuchenbauer F, Argiropoulos B, et al. Identification of miR-145 and miR-146a as mediators of the 5q- syndrome phenotype. Nat Med. 2010;16(1):49-58. -PubMed
- Franceschi C, Campisi J. Chronic inflammation (inflammaging) and its potential contribution to age-associated diseases. J Gerontol A Biol Sci Med Sci. 2014;69(suppl 1):S4-S9. -PubMed
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