IL-17 contributes to autoimmune hepatitis (original) (raw)
References
Lan RY, Salunga TL, Tsuneyama K, et al. Hepatic IL-17 responses in human and murine primary biliary cirrhosis. J Autoimmun, 2009,32(1):43–51 ArticleCASPubMed Google Scholar
Lleo A, Battezzati PM, Selmi C, et al. Is autoimmunity a matter of sex? Autoimmun Rev, 2008,7(8):626–630 ArticleCASPubMed Google Scholar
Gershwin ME, Mackay IR, Sturgess A, et al. Identification and specificity of a cDNA encoding the 70 kd mitochondrial antigen recognized in primary biliary cirrhosis. J Immunol, 1987,138(10):3525–3531 CASPubMed Google Scholar
Mackay IR, Whittingham S, Fida S, et al. The peculiar autoimmunity of primary biliary cirrhosis. Immunol Rev, 2000,174(1):226–237 ArticleCASPubMed Google Scholar
Shimoda S, Miyakawa H, Nakamura M, et al. CD4 T-cell autoreactivity to the mitochondrial autoantigen PDC-E2 in AMA-negative primary biliary cirrhosis. J Autoimmun, 2008,31(2):110–115 ArticleCASPubMed Google Scholar
Aarvak T, Chabaud M, Miossec P, et al. IL-17 is produced by some proinflammatory Th1/Th0 cells but not by Th2 cells. J Immunol, 1999,162(3):1246–1251 CASPubMed Google Scholar
Albanesi C, Scarponi C, Cavani A, et al. Interleukin-17 is produced by both Th1 and Th2 lymphocytes, and modulates interferon-gamma- and interleukin-4-induced activation of human keratinocytes. J Invest Dermatol, 2000, 115(1):81–87 ArticleCASPubMed Google Scholar
Infante-Duarte C, Horton HF, Byrne MC, et al. Microbial lipopeptides induce the production of IL-17 in Th cells. J Immunol, 2000,165(11):6107–6115 CASPubMed Google Scholar
Molet S, Hamid Q, Davoine F, et al. IL-17 is increased in asthmatic airways and induces human bronchial fibroblasts to produce cytokines. J Allergy Clin Immunol, 2001,108(3):430–438 ArticleCASPubMed Google Scholar
Ferretti S, Bonneau O, Dubois GR, et al. IL-17, produced by lymphocytes and neutrophils, is necessary for lipopolysaccharide-induced airway neutrophilia: IL-15 as a possible trigger. J Immunol, 2003,170(4):2106–2112 CASPubMed Google Scholar
Happel KI, Zheng M, Young E, et al. Roles of Toll-like receptor 4 and IL-23 in IL-17 expression in response to Klebsiella pneumoniae infection. J Immunol, 2003,170(9): 4432–4436 CASPubMed Google Scholar
Moseley TA, Haudenschild DR, Rose L, et al. Interleukin-17 family and IL-17 receptors. Cytokine Growth Factor Rev, 2003,14(2):155–174 ArticleCASPubMed Google Scholar
Wong CK, Ho CY, Li EK, et al. Elevation of proinflammatory cytokine (IL-18, IL-17, IL-12) and Th2 cytokine (IL-4) concentrations in patients with systemic lupus erythematosus. Lupus, 2000,9(8):589–593 ArticleCASPubMed Google Scholar
Hamzaoui K, Hamzaoui A, Guemira F, et al. Cytokine profile in Behcet’s disease patients: relationship with disease activity. Scand J Rheumatol, 2002,31(4):205–210 ArticlePubMed Google Scholar
Antonysamy MA, Fanslow WC, Fu F, et al. Evidence for a role of IL-17 in organ allograft rejection: IL-17 promotes the functional differentiation of dendritic cell progenitors. J Immunol, 1999,162:577–584 CASPubMed Google Scholar
Matsumoto K, Kanmatsuse K. Increased urinary excretion of interleukin-17 in nephrotic patients. Nephron, 2002, 91(2):243–249 ArticleCASPubMed Google Scholar
Wong CK, Ho CY, Ko FW. Proinflammatory cytokines (IL-17, IL-6, IL-18 and IL-12) and Th cytokines (IFN-γ, IL-4, IL-10 and IL-13) in patients with allergic asthma. Clin Exp Immunol, 2001,125(2):177–183 ArticleCASPubMed Google Scholar
Lock C, Hermans G, Pedotti R, et al. Gene-microarray analysis of multiple sclerosis lesions yields new targets validated in autoimmune encephalomyelitis. Nat Med, 2002,8:500–508 ArticleCASPubMed Google Scholar
Ye PF, Rodriguez H, Kanaly S, et al. Requirement of interleukin 17 receptor signaling for lung CXC chemokine and granulocyte colony-stimulating factor expression, neutrophil recruitment, and host defense. J Exp Med, 2001,194(4):519–528 ArticleCASPubMed Google Scholar
Ma X, Jia YT, Qiu DK. Inhibition of p38 mitogen-activated protein kinase attenuates experimental autoimmune hepatitis: Involvement of nuclear factor kappa B. World J Gastroenterol, 2007,13(31):4249–4254 CASPubMed Google Scholar
Nakae S, Komiyama Y, Nambu A, et al. Antigen-specific T cell sensitization is impaired in IL-17-deficient mice, causing suppression of allergic cellular and humoral responses. Immunity, 2002,17(3):375–387 ArticleCASPubMed Google Scholar
Alvarez F, Berg PA, Bianchi FB, et al. International autoimmune hepatitis group report: review of criteria for diagnosis of autoimmune hepatitis. J Hepatol, 1999,31(5): 929–938 ArticleCASPubMed Google Scholar
Kolls JK, Linde A. Interleukin-17 family members and inflammation. Immunity, 2004,21(4):467–476 ArticleCASPubMed Google Scholar
Matsuzaki G, Umemura M. Interleukin-17 as an effector molecule of innate and acquired immunity against infections. Microbiol Immunol, 2007,51(12):1139–1147 CASPubMed Google Scholar
Acosta-Rodriguez EV, Napolitani G, Lanzavecchia A, et al. Interleukin 1 and 6 but not transforming growth factor are essential for the differentiation of interleukin 17-producing human T helper cells. Nat Immunol, 2007,8(9):942–949 ArticleCASPubMed Google Scholar
Weaver CT, Hatton RD, Mangan PR. IL-17 family cytokines and the expanding diversity of effector T cell lineages. Annu Rev Immunol, 2007,25:821–852 ArticleCASPubMed Google Scholar
Amadi-Obi A, Yu CH, Liu X, et al. Th17 cells contribute to uveitis and scleritis and are expanded by IL-2 and inhibited by IL-27/STAT1. Nat Med, 2007,13(6):711–718 ArticleCASPubMed Google Scholar
Zhang JY, Zhang Z, Lin F. Interleukin-17-producing CD4+ T cells increase with severity of liver damage in patients with chronic hepatitis B. Hepatology, 2010,51(1):81–91 CASPubMed Google Scholar
Lemmers A, Moreno C, Gustot T, et al. The interleukin-17 pathway is involved in human alcoholic liver disease. Hepatology, 2009,49(2):646–657 ArticleCASPubMed Google Scholar
Ruth YZ, Lan A, Thucydides L, et al. Hepatic IL-17 responses in human and murine primary biliary cirrhosis. J Autoimmunol, 2009,32(1):43–51 Article Google Scholar
Silvia SC, Dieudonnée T, Isabelle C. Interleukin-17 is a negative regulator of established allergic asthma. J Exp Med, 2007,203(12):2715–2725 Google Scholar