Bcl10 activates the NF-κB pathway through ubiquitination of NEMO (original) (raw)
- Letter
- Published: 24 December 2003
- Ingrid Wertz1,4,
- Karen O'Rourke1,
- Mark Ultsch2,
- Somasekar Seshagiri3,
- Michael Eby3,
- Wei Xiao5 &
- …
- Vishva M. Dixit1
Nature volume 427, pages 167–171 (2004)Cite this article
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Abstract
The NF-κB family of transcription factors is activated in response to many stimuli, including pro-inflammatory cytokines, environmental stresses and, in the case of B and T lymphocytes, by antigenic stimulation1,2. Bcl10 is essential for NF-κB activation by T- and B-cell receptors. T and B lymphocytes from Bcl10-deficient mice fail to activate NF-κB in response to antigen-receptor stimulation and, as a consequence, are unable to proliferate3. Bcl10 overexpression is sufficient to activate NF-κB, a process that requires the NF-κB essential modulator NEMO (also known as IKK-γ), which is the regulatory subunit of the IκB kinase complex4. However, the cellular mechanism by which Bcl10 activates the NF-κB pathway remains unclear. Here we show that Bcl10 targets NEMO for lysine-63-linked ubiquitination. Notably, a mutant form of NEMO that cannot be ubiquitinated inhibited Bcl10-induced NF-κB activation. Paracaspase and a ubiquitin-conjugating enzyme (UBC13) were both required for Bcl10-induced NEMO ubiquitination and subsequent NF-κB activation. Furthermore, short interfering RNAs that reduced the expression of paracaspase and UBC13 abrogated the effects of Bcl10. Thus, the adaptor protein Bcl10 promotes activation of NF-κB transcription factors through paracaspase- and UBC13-dependent ubiquitination of NEMO.
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Acknowledgements
We thank members of the Dixit laboratory for discussions and support; K. Newton for critical reading of the manuscript; S. Sun for the JM4.5.2 cell line; S. Yamaoka for the 5R cell line; R. Baker for Usp2; M. Yan for experimental advice; Z. Zhang for bioinformatics analysis; P. Andersen for antibody preparation; P. Yin for protein purification; and the Genentech Sequencing Core. I.E.W. is funded by a PSTP fellowship from the University of California at Davis.
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Authors and Affiliations
- Department of Molecular Oncology, Genentech Inc. 1 DNA Way, South San Francisco, California, 94080, USA
Honglin Zhou, Ingrid Wertz, Karen O'Rourke & Vishva M. Dixit - Department of Protein Engineering, Genentech Inc. 1 DNA Way, South San Francisco, California, 94080, USA
Mark Ultsch - Department of Molecular Biology, Genentech Inc. 1 DNA Way, South San Francisco, California, 94080, USA
Somasekar Seshagiri & Michael Eby - Department of Biological Chemistry, School of Medicine, University of California at Davis, Davis, California, 95616, USA
Ingrid Wertz - Department of Microbiology and Immunology, University of Saskatchewan, Saskatoon, Saskatchewan, S7N 5E5, Canada
Wei Xiao
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Zhou, H., Wertz, I., O'Rourke, K. et al. Bcl10 activates the NF-κB pathway through ubiquitination of NEMO.Nature 427, 167–171 (2004). https://doi.org/10.1038/nature02273
- Received: 20 October 2003
- Accepted: 02 December 2003
- Published: 24 December 2003
- Issue Date: 08 January 2004
- DOI: https://doi.org/10.1038/nature02273