Release of Matrix Metalloproteinases-2 and 9 by S-Nitrosylated Caveolin-1 Contributes to Degradation of Extracellular Matrix in tPA-Treated Hypoxic Endothelial Cells (original) (raw)
Fig 6
Scavenge of NO with C-PTIO (A) and inhibition of ERK signal pathway with U0126 (B) recover the same levels of fibronectin (Fb) and laminin β-1(LMN β-1) in the cells treated with control (Ctrl), OGD, tPA, and OGD+tPA (O+t).
Upper panel: representative immunoblots, β-actin served as loading control; Bottom panel: band intensity was quantitated after normalization to β-actin and the data were expressed as mean ± SD, ANOVA, N = 4.