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Number of items: 23.

2023

Cunningham, Madeleine E. ORCID logoORCID: https://orcid.org/0000-0002-7545-6868, McGonigal, Rhona ORCID logoORCID: https://orcid.org/0000-0001-9571-2526, Barrie, Jennifer A., Campbell, Clare I., Yao, Denggao and Willison, Hugh J. ORCID logoORCID: https://orcid.org/0000-0002-5997-1683(2023)Axolemmal nanoruptures arising from paranodal membrane injury induce secondary axon degeneration in murine Guillain-Barré syndrome. Journal of the Peripheral Nervous System, 28(1), pp. 17-31. (doi: 10.1111/jns.12532) (PMID:36710500) (PMCID:PMC10947354)

2022

Cunningham, Madeleine E. ORCID logoORCID: https://orcid.org/0000-0002-7545-6868, McGonigal, Rhona ORCID logoORCID: https://orcid.org/0000-0001-9571-2526, Barrie, Jennifer A., Yao, Denggao and Willison, Hugh J. ORCID logoORCID: https://orcid.org/0000-0002-5997-1683(2022)Real time imaging of intra-axonal calcium flux in an explant mouse model of axonal Guillain-Barré syndrome. Experimental Neurology, 355, 114127. (doi: 10.1016/j.expneurol.2022.114127) (PMID:35640716) (PMCID:PMC7614209)

McGonigal, Rhona ORCID logoORCID: https://orcid.org/0000-0001-9571-2526, Campbell, Clare I., Barrie, Jennifer A., Yao, Denggao, Cunningham, Madeleine E. ORCID logoORCID: https://orcid.org/0000-0002-7545-6868, Crawford, Colin L., Rinaldi, Simon, Rowan, Edward G. and Willison, Hugh J. ORCID logoORCID: https://orcid.org/0000-0002-5997-1683(2022)Schwann cell nodal membrane disruption triggers bystander axonal degeneration in a Guillain-Barré syndrome mouse model. Journal of Clinical Investigation, 132(14), e158524. (doi: 10.1172/JCI158524) (PMID:35671105) (PMCID:PMC9282931)

Campbell, Clare I., McGonigal, Rhona ORCID logoORCID: https://orcid.org/0000-0001-9571-2526, Barrie, Jennifer A., Delaere, Jolien, Bracke, Laura, Cunningham, Madeleine E. ORCID logoORCID: https://orcid.org/0000-0002-7545-6868, Yao, Denggao, Delahaye, Tim, Van de Walle, Inge and Willison, Hugh J. ORCID logoORCID: https://orcid.org/0000-0002-5997-1683(2022)Complement inhibition prevents glial nodal membrane injury in a GM1 antibody-mediated mouse model. Brain Communications, 4(6), fcac306. (doi: 10.1093/braincomms/fcac306) (PMID:36523267) (PMCID:PMC9746686)

2021

McGonigal, Rhona ORCID logoORCID: https://orcid.org/0000-0001-9571-2526, Barrie, Jennifer A., Yao, Denggao, Black, Lauren E. ORCID logoORCID: https://orcid.org/0000-0001-5459-7939, McLaughlin, Mark ORCID logoORCID: https://orcid.org/0000-0001-5317-2090 and Willison, Hugh J. ORCID logoORCID: https://orcid.org/0000-0002-5997-1683(2021)Neuronally expressed a-series gangliosides are sufficient to prevent the lethal age-dependent phenotype in GM3-only expressing mice. Journal of Neurochemistry, 158(2), pp. 217-232. (doi: 10.1111/jnc.15365) (PMID:33864399)

2020

Cunningham, Madeleine E. ORCID logoORCID: https://orcid.org/0000-0002-7545-6868, Meehan, Gavin R. ORCID logoORCID: https://orcid.org/0000-0001-9855-6565, Robinson, Sophie, Yao, Denggao, McGonigal, Rhona ORCID logoORCID: https://orcid.org/0000-0001-9571-2526 and Willison, Hugh J. ORCID logoORCID: https://orcid.org/0000-0002-5997-1683(2020)Perisynaptic Schwann cells phagocytose nerve terminal debris in a mouse model of Guillain-Barré syndrome. Journal of the Peripheral Nervous System, 25(2), pp. 143-151. (doi: 10.1111/jns.12373) (PMID:32250537)

2019

McGonigal, R. ORCID logoORCID: https://orcid.org/0000-0001-9571-2526, Barrie, J.A., Yao, D., McLaughlin, M. ORCID logoORCID: https://orcid.org/0000-0001-5317-2090, Cunningham, M.E. ORCID logoORCID: https://orcid.org/0000-0002-7545-6868, Rowan, E.G. and Willison, H.J. ORCID logoORCID: https://orcid.org/0000-0002-5997-1683(2019)Glial sulfatides and neuronal complex gangliosides are functionally interdependent in maintaining myelinating axon integrity. Journal of Neuroscience, 39(1), pp. 63-77. (doi: 10.1523/JNEUROSCI.2095-18.2018) (PMID:30446529) (PMCID:PMC6325269)

2018

Meehan, Gavin R. ORCID logoORCID: https://orcid.org/0000-0001-9855-6565, McGonigal, Rhona ORCID logoORCID: https://orcid.org/0000-0001-9571-2526, Cunningham, Madeleine E. ORCID logoORCID: https://orcid.org/0000-0002-7545-6868, Wang, Yuzhong, Barrie, Jennifer A., Halstead, Susan K. ORCID logoORCID: https://orcid.org/0000-0001-7396-8969, Gourlay, Dawn, Yao, Denggao and Willison, Hugh J. ORCID logoORCID: https://orcid.org/0000-0002-5997-1683(2018)Differential binding patterns of anti-sulfatide antibodies to glial membranes. Journal of Neuroimmunology, 323, pp. 28-35. (doi: 10.1016/j.jneuroim.2018.07.004) (PMID:30196830) (PMCID:PMC6134133)

2016

Cunningham, Madeleine ORCID logoORCID: https://orcid.org/0000-0002-7545-6868, McGonigal, Rhona ORCID logoORCID: https://orcid.org/0000-0001-9571-2526, Meehan, Gavin R. ORCID logoORCID: https://orcid.org/0000-0001-9855-6565, Barrie, Jennifer A., Yao, Denggao, Halstead, Susan K. ORCID logoORCID: https://orcid.org/0000-0001-7396-8969 and Willison, Hugh J. ORCID logoORCID: https://orcid.org/0000-0002-5997-1683(2016)Anti-ganglioside antibodies are removed from circulation in mice by neuronal endocytosis. Brain, 139(6), pp. 1657-1665. (doi: 10.1093/brain/aww056) (PMID:27017187) (PMCID:PMC4892750)

McGonigal, Rhona ORCID logoORCID: https://orcid.org/0000-0001-9571-2526, Cunningham, Madeleine E. ORCID logoORCID: https://orcid.org/0000-0002-7545-6868, Yao, Denggao, Barrie, Jennifer A., Sankaranarayanan, Sethu, Fewou, Simon N., Furukawa, Koichi, Yednock, Ted A. and Willison, Hugh J. ORCID logoORCID: https://orcid.org/0000-0002-5997-1683(2016)C1q-targeted inhibition of the classical complement pathway prevents injury in a novel mouse model of acute motor axonal neuropathy. Acta Neuropathologica Communications, 4, 23. (doi: 10.1186/s40478-016-0291-x) (PMID:26936605) (PMCID:PMC4776408)

2015

Delmont, Emilien, Robb, Henry, Davidson, Amy, Halstead, Susan ORCID logoORCID: https://orcid.org/0000-0001-7396-8969, Yao, Denggao, Meehan, Gavin R. ORCID logoORCID: https://orcid.org/0000-0001-9855-6565 and Willison, Hugh ORCID logoORCID: https://orcid.org/0000-0002-5997-1683(2015)Prospective study comparing enzyme-linked immunosorbent assay and glycoarray assay to detect antiglycolipid antibodies in a routine diagnostic neuroimmunology laboratory setting. Clinical and Experimental Neuroimmunology, 6(2), pp. 175-182. (doi: 10.1111/cen3.12193)

Delmont, Emilien, Halstead, Susan ORCID logoORCID: https://orcid.org/0000-0001-7396-8969, Galban-Horcajo, Francesc, Yao, Denggao, Desnuelle, Claude and Willison, Hugh ORCID logoORCID: https://orcid.org/0000-0002-5997-1683(2015)Improving the detection of IgM antibodies against glycolipids complexes of GM1 and galactocerebroside in multifocal motor neuropathy using glycoarray and ELISA assays. Journal of Neuroimmunology, 278, pp. 159-161. (doi: 10.1016/j.jneuroim.2014.11.001) (PMID:25468269)

2014

Macdonald, FH, Yao, D, Quinn, JA ORCID logoORCID: https://orcid.org/0000-0002-8267-0993 and Greenhalgh, DA ORCID logoORCID: https://orcid.org/0000-0002-6737-2744(2014)PTEN ablation in RasHa/Fos skin carcinogenesis invokes p53-dependent p21 to delay conversion while p53-independent p21 limits progression via cyclin D1/E2 inhibition. Oncogene, 33(32), pp. 4132-4143. (doi: 10.1038/onc.2013.372)

Yao, D. et al. (2014)Neuronal expression of galnac transferase is sufficient to prevent the age-related neurodegenerative phenotype of complex ganglioside-deficient mice. Journal of Neuroscience, 34(3), pp. 880-891. (doi: 10.1523/JNEUROSCI.3996-13.2014)

2013

Rupp, Angie, Cunningham, Madeleine E. ORCID logoORCID: https://orcid.org/0000-0002-7545-6868, Yao, Denggao, Furukawa, Koichi and Willison, Hugh J. ORCID logoORCID: https://orcid.org/0000-0002-5997-1683(2013)The effects of age and ganglioside composition on the rate of motor nerve terminal regeneration following antibody-mediated injury in mice. Synapse, 67(7), pp. 382-389. (doi: 10.1002/syn.21648) (PMID:23401234) (PMCID:PMC4495252)

Rupp, Angie, Yao, Denggao, Meehan, Gavin R. ORCID logoORCID: https://orcid.org/0000-0001-9855-6565 and Willison, Hugh J. ORCID logoORCID: https://orcid.org/0000-0002-5997-1683(2013)Thy-1 Mice Lend Themselves for the Modelling of Anti-ganglioside Antibody-mediated Disorders. PNS Biennial Meeting of the Peripheral Nerve Society, St Malo, France, 11-14 Jun 2013.

Macdonald, F.H., Yao, D., Quinn, J.A. ORCID logoORCID: https://orcid.org/0000-0002-8267-0993 and Greenhalgh, D.A. ORCID logoORCID: https://orcid.org/0000-0002-6737-2744(2013)Activated p-AKT, but not MDM2, drives malignant progression in Ras/Fos/PTENnull skin carcinogenesis via p53/p21 loss and elevated cyclin D1/E2 expression. Journal of Investigative Dermatology, 133(S1), S57-S57. (doi: 10.1038/jid.2013.96)

2009

Greenhalgh, D. ORCID logoORCID: https://orcid.org/0000-0002-6737-2744, Yao, D., Quinn, J. ORCID logoORCID: https://orcid.org/0000-0002-8267-0993 and Macdonald, F.(2009)RasHa activation interdicts compensatory p53/p21WAF expression in Fos/PTENnull skin carcinogenesis via initial p53 loss but retention of p21WAF limits malignant progression. In: American Association for Cancer Research (AACR) 100th Annual Meeting, Denver, USA, 18-22 April 2009,

Macdonald, F., Yao, D., Quinn, J. ORCID logoORCID: https://orcid.org/0000-0002-8267-0993 and Greenhalgh, D. ORCID logoORCID: https://orcid.org/0000-0002-6737-2744(2009)Compensatory p53/p21WAF-induced differentiation in Fos/PTENnull skin carcinogenesis is interdicted by RasHa activation but retention of p21WAF inhibits malignant progression. In: British Society for Investigative Dermatology (BSID) Annual Meeting, Royal Agricultural College, Cirencester, UK, 30 March - 1 April 2009,

2008

Yao, D, Alexander, CL, Quinn, JA ORCID logoORCID: https://orcid.org/0000-0002-8267-0993, Chan, WC, Wu, H and Greenhalgh, DA(2008)Fos cooperation with PTEN loss elicits keratoacanthoma not carcinoma, owing to p53/p21(WAF)-induced differentiation triggered by GSK3 beta inactivation and reduced AKT activity. Journal of Cell Science, 121(10), pp. 1758-1769.

2006

Yao, D., Alexander, C.L., Quinn, J.A., Porter, M.J., Wu, H. and Greenhalgh, D.A. ORCID logoORCID: https://orcid.org/0000-0002-6737-2744(2006)PTEN loss promotes rasHa-mediated papillomatogenesis via dual up-regulation of AKT activity and cell cycle deregulation but malignant conversion proceeds via rasHa-independent pathways. Cancer Research, 66(3), pp. 1302-1312.

Shaw, M., Yao, D., Quinn, J.A. and Greenhalgh, D.A. ORCID logoORCID: https://orcid.org/0000-0002-6737-2744(2006)Paradoxical maintenance of E-cadherin following inducible PTEN ablation in Ras or Fos-mediated skin carcinogenesis identifies sentinel mechanisms geared to combat PTEN dysfunction. In: British Society for Investigative Dermatology Annual Meeting, Manchester, 10-12 April 2006, p. 247. (doi: 10.1111/j.1365-2133.2006.07327.x)

2005

Yao, D., Quinn, J.A. and Greenhalgh, D.A. ORCID logoORCID: https://orcid.org/0000-0002-6737-2744(2005)Inducible cre-mediated N-ras activation and PTEN inactivation in transgenic mouse melanocytes requires keratinocyte hyperplasia to elicit a melanocyte pathology. In: Montagne Symposia, 2004 and 6th International Skin Cancer Conference, 2004, Salishan Resort, Newport, Oregon, USA, Oct 2004, p. 171.

This list was generated on Tue Feb 17 21:07:57 2026 GMT.