Kobe Jong - Academia.edu (original) (raw)
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Graduate Center of the City University of New York
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Papers by Kobe Jong
Physics Letters B, 2012
A search for diphoton events with large missing transverse momentum has been performed using prot... more A search for diphoton events with large missing transverse momentum has been performed using proton-proton collision data at √ s = 7 TeV recorded with the ATLAS detector, corresponding to an integrated luminosity of 4.8 fb −1 . No excess of events was observed above the Standard Model prediction and model-dependent 95 % confidence level exclusion limits are set. In the context of a generalised model of gauge-mediated supersymmetry breaking with a bino-like lightest neutralino of mass above 50 GeV, gluinos (squarks) below 1.07 TeV (0.87 TeV) are excluded, while a breaking scale Λ below 196 TeV is excluded for a minimal model of gauge-mediated supersymmetry breaking. For a specific model with one universal extra dimension, compactification scales 1/R < 1.40 TeV are excluded. These limits provide the most stringent tests of these models to date.
Virology, 1996
DA strain of TMEV induces a chronic, persistent, demyelinating disease in SJL/J weanling mice, wh... more DA strain of TMEV induces a chronic, persistent, demyelinating disease in SJL/J weanling mice, while inoculation with GDVII strain of TMEV induces an acute, lethal neurovirulent disease. We show that three amino acids in the DA EF loop -DAVP2 141 Lys, 143 Gly, and 173 Thr-are part of a neutralization site of DA monoclonal antibody (mAb), DAmAb1. DA virus with a mutation of VP2 143 from Gly to Asp, like wild-type virus, persists 6 weeks postinfection (PI) and produces white matter disease. DA virus with a mutation of VP2 141 from Lys to Asn persists but does not induce significant white matter disease. DA virus with a mutation of DA VP2 173 from Thr to Phe fails to persist or to induce significant white matter disease. The diversity and complexity of the mutant virus-induced disease phenotype presumably reflects the varied effects of the mutated amino acid residues on the three-dimensional structure of the viral capsid. The localization of DA VP2 141 and VP2 173 near the putative receptor binding region of the virus suggest that a disruption of interactions between the virus and its receptor is important in the late demyelinating disease and for virus neutralization. ᭧
This paper is dedicated to the memory of our ATLAS colleagues who did not live to see the full im... more This paper is dedicated to the memory of our ATLAS colleagues who did not live to see the full impact and significance of their contributions to the experiment.
Physics Letters B, 2012
A search for diphoton events with large missing transverse momentum has been performed using prot... more A search for diphoton events with large missing transverse momentum has been performed using proton-proton collision data at √ s = 7 TeV recorded with the ATLAS detector, corresponding to an integrated luminosity of 4.8 fb −1 . No excess of events was observed above the Standard Model prediction and model-dependent 95 % confidence level exclusion limits are set. In the context of a generalised model of gauge-mediated supersymmetry breaking with a bino-like lightest neutralino of mass above 50 GeV, gluinos (squarks) below 1.07 TeV (0.87 TeV) are excluded, while a breaking scale Λ below 196 TeV is excluded for a minimal model of gauge-mediated supersymmetry breaking. For a specific model with one universal extra dimension, compactification scales 1/R < 1.40 TeV are excluded. These limits provide the most stringent tests of these models to date.
Virology, 1996
DA strain of TMEV induces a chronic, persistent, demyelinating disease in SJL/J weanling mice, wh... more DA strain of TMEV induces a chronic, persistent, demyelinating disease in SJL/J weanling mice, while inoculation with GDVII strain of TMEV induces an acute, lethal neurovirulent disease. We show that three amino acids in the DA EF loop -DAVP2 141 Lys, 143 Gly, and 173 Thr-are part of a neutralization site of DA monoclonal antibody (mAb), DAmAb1. DA virus with a mutation of VP2 143 from Gly to Asp, like wild-type virus, persists 6 weeks postinfection (PI) and produces white matter disease. DA virus with a mutation of VP2 141 from Lys to Asn persists but does not induce significant white matter disease. DA virus with a mutation of DA VP2 173 from Thr to Phe fails to persist or to induce significant white matter disease. The diversity and complexity of the mutant virus-induced disease phenotype presumably reflects the varied effects of the mutated amino acid residues on the three-dimensional structure of the viral capsid. The localization of DA VP2 141 and VP2 173 near the putative receptor binding region of the virus suggest that a disruption of interactions between the virus and its receptor is important in the late demyelinating disease and for virus neutralization. ᭧
This paper is dedicated to the memory of our ATLAS colleagues who did not live to see the full im... more This paper is dedicated to the memory of our ATLAS colleagues who did not live to see the full impact and significance of their contributions to the experiment.