Maria Eugenia Cezere - Profile on Academia.edu (original) (raw)
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Gingival overgrowth can be classified as tissue enlargement of inflammatory origin, being charact... more Gingival overgrowth can be classified as tissue enlargement of inflammatory origin, being characterized clinically by the increase of gingival tissue that emerges from the interdental papilla, but which may affect the entire keratinized gingiva; of firm, yet resilient and mobile consistency; staining of pink to erythematous; granular or smooth surface, with a loss of normal appearance of the gingival surface ("orange peel"). Generally asymptomatic, except for inflammation or trauma during mastication, and may still present bleeding during the probing [1-5]. It can cover the coronary portion of affected teeth, creating deep pseudo-pockets that hinder oral hygiene and favor the accumulation of biofilm and dental calculus, causing the promotion of periodontal diseases and the installation of carious lesions [1]. Etiopathogenic factors include poor oral hygiene, accumulation of biofilm and dental calculus, considered as a low-intensity chronic traumatic factor [15]. In additio...
Gingival overgrowth can be classified as tissue enlargement of inflammatory origin, being charact... more Gingival overgrowth can be classified as tissue enlargement of inflammatory origin, being characterized clinically by the increase of gingival tissue that emerges from the interdental papilla, but which may affect the entire keratinized gingiva; of firm, yet resilient and mobile consistency; staining of pink to erythematous; granular or smooth surface, with a loss of normal appearance of the gingival surface ("orange peel"). Generally asymptomatic, except for inflammation or trauma during mastication, and may still present bleeding during the probing [1-5]. It can cover the coronary portion of affected teeth, creating deep pseudo-pockets that hinder oral hygiene and favor the accumulation of biofilm and dental calculus, causing the promotion of periodontal diseases and the installation of carious lesions [1]. Etiopathogenic factors include poor oral hygiene, accumulation of biofilm and dental calculus, considered as a low-intensity chronic traumatic factor [15]. In additio...