Sheila Sait - Academia.edu (original) (raw)
Papers by Sheila Sait
Cancer genetics and cytogenetics, Jun 1, 1987
Cytogenetic and Genome Research, 1995
Modern Pathology, Nov 1, 2009
North American Journal of Medical Sciences, 2013
Cancer genetics and cytogenetics, Mar 1, 2009
American Journal of Human Genetics, Sep 1, 1994
ABSTRACT
American Journal of Clinical Pathology, Sep 1, 2013
PubMed, Apr 1, 1990
To determine the cytogenetic origin of maturing granulocytes in the bone marrow of patients with ... more To determine the cytogenetic origin of maturing granulocytes in the bone marrow of patients with acute myelogenous leukemia, bone marrow cells were studied using a modified cytogenetic technique, which does not disrupt the cell membrane, in conjunction with periodic acid-Schiff (PAS) staining. In four cases successfully studied, myeloblasts were PAS-negative and granulocytes were PAS-positive. In three cases successfully studied following 0-2 days of culture, metaphase spreads with abnormal karyotypes characteristic of the patients' leukemic clones were seen in five of five, six of nine, and four of four PAS-positive cells successfully studied. These patients' bone marrows were AN, AA, and AA, respectively, by standard cytogenetic study. Therefore, the cytogenetic status of PAS-positive cells did not necessarily correlate with presence or absence of normal metaphases determined by standard cytogenetic study. Bone marrow cells which underwent full and partial granulocytic maturation in suspension culture were studied following 2 weeks of culture. Abnormal karyotypes were seen in five of five and two of two metaphases in PAS-positive cells successfully studied in two patients. Therefore, we have demonstrated that when acute myelogenous leukemia cells undergo myeloid maturation in culture, the mature cells may be definitely proven to derive from leukemic progenitors rather than from normal stem cells.
Cancer genetics and cytogenetics, Feb 1, 1989
The involvement of chromosomes 12 and 14 in uterine leiomyomas has been well established. However... more The involvement of chromosomes 12 and 14 in uterine leiomyomas has been well established. However, in a recen! report of only a del(7)(q22.1q31.32) or (q l l.2q22.3) in two cases of typical uterine leiomyoma, Boghosian et al. hypothesized that this could represent a cytagenetic subgroup of uterine leiomyomas. We report a case cff uterine leiomyoma with both the t(12;14) and del(7) in all the cells examined and discuss the implications af this in terms of critical chromosomal rearrangements underlying the route to benign cellular proliferation.
Journal of The National Comprehensive Cancer Network, Feb 1, 2023
Blood, Nov 20, 2009
Abstract 1972 Poster Board I-995 The only successful immunologic approach in acute myeloid leukem... more Abstract 1972 Poster Board I-995 The only successful immunologic approach in acute myeloid leukemia (AML) is allogeneic transplantation. However, this treatment modality is restricted by age and donor availability. It is also associated with significant toxicity. Therefore, autologous approaches are being sought. Among those, harnessing the…
Cancer genetics and cytogenetics, Nov 1, 1989
Cancer genetics and cytogenetics, Jul 1, 1988
Cancer genetics and cytogenetics, 1987
Abnormalities involving the long arm of chromosome #11 have been shown to be involved in a high p... more Abnormalities involving the long arm of chromosome #11 have been shown to be involved in a high proportion of acute nonlymphocytic leukemias, specifically FAB types M4 and M5. In particular, band 11q23 seems to be preferentially affected. Reported herein is a case of acute nonlymphocytic leukemia type M4 showing a t(1;11)(q21;q23).
Cancer genetics and cytogenetics, Oct 1, 1988
Karyotypic analysis of a leiomyosarcoma of the retroperitoneum revealed some structural and numer... more Karyotypic analysis of a leiomyosarcoma of the retroperitoneum revealed some structural and numerical changes. Review of the literature showed that some of these changes, namely involvement of 1p13 and 11p13 and monosomy 9, 18 and 22 seemed to occur frequently. These changes could be characteristic of leiomyosarcoma and define a cytogenetic subgroup within this tumor entity.
Cancer genetics and cytogenetics, Jun 1, 2006
A growing body of literature reports therapy-related myelodysplastic syndrome (t-MDS) and acute m... more A growing body of literature reports therapy-related myelodysplastic syndrome (t-MDS) and acute myeloid leukemia (t-AML) in patients treated successfully for acute promyelocytic leukemia (APL). We report a t-MDS with an isochromosome 1q as a sole abnormality, 47,XY,+1,i(1)(q10), in a 46-year-old man with APL 14 years after he was treated with cytosine arabinosine and daunorubicin. The literature on t-MDS/t-AML after APL therapy and on isochromosome 1q is reviewed.
Cancer genetics and cytogenetics, Feb 1, 1988
Cytogenetic studies were performed on human malignant melanoma cells from eight metastatic lesion... more Cytogenetic studies were performed on human malignant melanoma cells from eight metastatic lesions. Five tumors displayed near-triploid and three near-diploid chromosome numbers. Chromosomes #1, #6, #7, followed by #2 and #9, were found to be most frequently involved in structural aberrations. Aberrations involving chromosome #1, with deletions or translocations of 1p, involving region 1p12-1p22 in seven of eight breakpoints of the p arm were observed. Seven of nine breakpoints of 6q were located at region 6q15-6q21. Most of the breakpoints on chromosome #7 occurred near the centromeric region. All tumors had additional chromosome material involving 1q, chromosome #7 (7q in two tumors), and in five tumors an increased dose of chromosome #6 (6p in one tumor). The nonrandom breakpoints of these and other chromosomes involved diverse bands, including loci of oncogenes and fragile sites. The observation of nonrandom chromosomal changes in advanced malignant melanoma suggests that genes important in the progression of melanoma are located on chromosomes #1, #6, and #7.
Cancer genetics and cytogenetics, Sep 1, 1989
Cancer genetics and cytogenetics, Feb 1, 1989
Cancer genetics and cytogenetics, Jun 1, 1987
Cytogenetic and Genome Research, 1995
Modern Pathology, Nov 1, 2009
North American Journal of Medical Sciences, 2013
Cancer genetics and cytogenetics, Mar 1, 2009
American Journal of Human Genetics, Sep 1, 1994
ABSTRACT
American Journal of Clinical Pathology, Sep 1, 2013
PubMed, Apr 1, 1990
To determine the cytogenetic origin of maturing granulocytes in the bone marrow of patients with ... more To determine the cytogenetic origin of maturing granulocytes in the bone marrow of patients with acute myelogenous leukemia, bone marrow cells were studied using a modified cytogenetic technique, which does not disrupt the cell membrane, in conjunction with periodic acid-Schiff (PAS) staining. In four cases successfully studied, myeloblasts were PAS-negative and granulocytes were PAS-positive. In three cases successfully studied following 0-2 days of culture, metaphase spreads with abnormal karyotypes characteristic of the patients' leukemic clones were seen in five of five, six of nine, and four of four PAS-positive cells successfully studied. These patients' bone marrows were AN, AA, and AA, respectively, by standard cytogenetic study. Therefore, the cytogenetic status of PAS-positive cells did not necessarily correlate with presence or absence of normal metaphases determined by standard cytogenetic study. Bone marrow cells which underwent full and partial granulocytic maturation in suspension culture were studied following 2 weeks of culture. Abnormal karyotypes were seen in five of five and two of two metaphases in PAS-positive cells successfully studied in two patients. Therefore, we have demonstrated that when acute myelogenous leukemia cells undergo myeloid maturation in culture, the mature cells may be definitely proven to derive from leukemic progenitors rather than from normal stem cells.
Cancer genetics and cytogenetics, Feb 1, 1989
The involvement of chromosomes 12 and 14 in uterine leiomyomas has been well established. However... more The involvement of chromosomes 12 and 14 in uterine leiomyomas has been well established. However, in a recen! report of only a del(7)(q22.1q31.32) or (q l l.2q22.3) in two cases of typical uterine leiomyoma, Boghosian et al. hypothesized that this could represent a cytagenetic subgroup of uterine leiomyomas. We report a case cff uterine leiomyoma with both the t(12;14) and del(7) in all the cells examined and discuss the implications af this in terms of critical chromosomal rearrangements underlying the route to benign cellular proliferation.
Journal of The National Comprehensive Cancer Network, Feb 1, 2023
Blood, Nov 20, 2009
Abstract 1972 Poster Board I-995 The only successful immunologic approach in acute myeloid leukem... more Abstract 1972 Poster Board I-995 The only successful immunologic approach in acute myeloid leukemia (AML) is allogeneic transplantation. However, this treatment modality is restricted by age and donor availability. It is also associated with significant toxicity. Therefore, autologous approaches are being sought. Among those, harnessing the…
Cancer genetics and cytogenetics, Nov 1, 1989
Cancer genetics and cytogenetics, Jul 1, 1988
Cancer genetics and cytogenetics, 1987
Abnormalities involving the long arm of chromosome #11 have been shown to be involved in a high p... more Abnormalities involving the long arm of chromosome #11 have been shown to be involved in a high proportion of acute nonlymphocytic leukemias, specifically FAB types M4 and M5. In particular, band 11q23 seems to be preferentially affected. Reported herein is a case of acute nonlymphocytic leukemia type M4 showing a t(1;11)(q21;q23).
Cancer genetics and cytogenetics, Oct 1, 1988
Karyotypic analysis of a leiomyosarcoma of the retroperitoneum revealed some structural and numer... more Karyotypic analysis of a leiomyosarcoma of the retroperitoneum revealed some structural and numerical changes. Review of the literature showed that some of these changes, namely involvement of 1p13 and 11p13 and monosomy 9, 18 and 22 seemed to occur frequently. These changes could be characteristic of leiomyosarcoma and define a cytogenetic subgroup within this tumor entity.
Cancer genetics and cytogenetics, Jun 1, 2006
A growing body of literature reports therapy-related myelodysplastic syndrome (t-MDS) and acute m... more A growing body of literature reports therapy-related myelodysplastic syndrome (t-MDS) and acute myeloid leukemia (t-AML) in patients treated successfully for acute promyelocytic leukemia (APL). We report a t-MDS with an isochromosome 1q as a sole abnormality, 47,XY,+1,i(1)(q10), in a 46-year-old man with APL 14 years after he was treated with cytosine arabinosine and daunorubicin. The literature on t-MDS/t-AML after APL therapy and on isochromosome 1q is reviewed.
Cancer genetics and cytogenetics, Feb 1, 1988
Cytogenetic studies were performed on human malignant melanoma cells from eight metastatic lesion... more Cytogenetic studies were performed on human malignant melanoma cells from eight metastatic lesions. Five tumors displayed near-triploid and three near-diploid chromosome numbers. Chromosomes #1, #6, #7, followed by #2 and #9, were found to be most frequently involved in structural aberrations. Aberrations involving chromosome #1, with deletions or translocations of 1p, involving region 1p12-1p22 in seven of eight breakpoints of the p arm were observed. Seven of nine breakpoints of 6q were located at region 6q15-6q21. Most of the breakpoints on chromosome #7 occurred near the centromeric region. All tumors had additional chromosome material involving 1q, chromosome #7 (7q in two tumors), and in five tumors an increased dose of chromosome #6 (6p in one tumor). The nonrandom breakpoints of these and other chromosomes involved diverse bands, including loci of oncogenes and fragile sites. The observation of nonrandom chromosomal changes in advanced malignant melanoma suggests that genes important in the progression of melanoma are located on chromosomes #1, #6, and #7.
Cancer genetics and cytogenetics, Sep 1, 1989
Cancer genetics and cytogenetics, Feb 1, 1989