Morbidly “Healthy” Obese Are Not Metabolically Healthy but Less Metabolically Imbalanced Than Those with Type 2 Diabetes or Dyslipidemia (original) (raw)
Abstract
Background
We have investigated the differences between metabolically “healthy” morbidly obese patients and those with comorbidities.
Materials and Methods
Thirty-two morbidly obese patients were divided by the absence (“healthy”: DM−DL−) or presence of comorbidities (dyslipidemic: DM−DL+, or dyslipidemic and with type 2 diabetes: DM+DL+). We have studied various plasma parameters and gene expression adipose tissue, before and after gastric bypass.
Results
The group DM+DL+ tends to have lower values than the other two groups for anthropometric parameters. Regarding the satiety parameters, only leptin (p = 0.0024) showed a significant increase with comorbidities. Lipid parameters showed significant differences among groups, except for phospholipids and NEFA. For insulin resistance parameters, only glucose (p < 0.0001) was higher in DM+DL+ patients, but not insulin or homeostasis model assessment of insulin resistance (HOMA-IR). The gene expression of adiponectin, insulin receptor (INSR) and glucose receptor-4 (GLUT4), in the subcutaneous fat, decreased in all groups vs. a non-obese control. Interleukin-6 (IL6) and the inhibitor of plasminogen activator type 1 (PAI-1) genes decreased only in DM−DL+ and DM+DL+, but not in “healthy” patients. Leptin increased in all groups vs. the non-obese control. The visceral fat from DM+DL+ patients showed a sharp decrease in adiponectin, GLUT4, IL6 and PAI-1. All parameters mentioned above improved very significantly by surgery, independent of the occurrence of comorbidities.
Conclusions
The morbidly obese “healthy” individual is not really metabolically healthy, but morbidly obese individuals with diabetes and dyslipidemia are more metabolically imbalanced.
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Abbreviations
RYGBP:
Roux-en-Y gastric bypass
HOMA-IR:
Homeostasis model assessment of insulin resistance
NEFA:
Non-esterified fatty acid
TG:
Triglyceride
TC:
Total cholesterol
SBP:
Systolic blood pressure
DBP:
Diastolic blood pressure
DM:
Diabetes mellitus
DL:
Dyslipidemia
HTA:
Arterial hypertension
PAI-1:
Plasminogen activator inhibitor-1
CRP:
C-Reactive Protein
SAT:
Subcutaneous adipose tissue
VAT:
Visceral adipose tissue
CVD:
Cardiovascular disease
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Acknowledgments
Study concept and design (JAB-F, JP-O), surgery and endocrinology studies of patients (JAB-F, JMF, JMB, EC, OG, RV, AL) performed experiments (RF, EP, JR), tissue analyses (RF, EP, JR), analysis and interpretation of data (JAB-F, JP-O, AL, RF, EP, JR), obtained funding (JP-O), drafting of manuscript (JP-O), critical revision of the manuscript for important intellectual content (JAB-F, JP-O, AL, RF). We thank Dr. Miñarro of the Biostatistics Dept. of Biology Fac. (UB) for their invaluable help. All authors were involved in writing the paper and approved the final version of the manuscript. English grammar and language were corrected by American Journal Experts (www.journalexperts.com).
Ethical Approval
All procedures performed in studies involving human participants were in accordance with the ethical standards of the Institutional and/or National research committee and with the 1964 Helsinki Declaration and its later amendments or comparable ethical standards. Informed consent was obtained from all individual participants included in the study.
Conflict of Interest
The authors declare that no conflicts of interest exist. The authors who have taken part in this study do not have a relationship, past or present, with the manufacturers of the drugs involved and did not receive funding from the manufacturers to conduct their research.
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Authors and Affiliations
- Biochemistry Department, Hospital Universitari Vall D’Hebron, Universitat Autònoma De Barcelona, Barcelona, Spain
Roser Ferrer - Biochemistry and Molecular Biology Department, Biology Faculty, Barcelona University, Barcelona, Spain
Eva Pardina, Joana Rossell, Laura Oller, Anna Viñas & Julia Peinado-Onsurbe - Surgery Unit, Arnau de Vilanova University Hospital (UdL), Lleida, Spain
Juan Antonio Baena-Fustegueras - Endocrinology and Nutrition Department from Arnau de Vilanova University Hospital (UdL) Lleida and Diabetes and Metabolism Research Unit (VHIR, UAB) Barcelona and CIBER de Diabetes y Enfermedades Metabólicas (CIBERDEM-ISCIII), Arnau de Vilanova University Hospital (UdL), Lleida, Spain
Albert Lecube - CIBER de Enfermedades Hepáticas y Digestivas (CIBEREHD) del Instituto de Salud Carlos III (ISCIII), Hospital Universitari Vall D’Hebron, Universitat Autònoma De Barcelona, Barcelona, Spain
Víctor Vargas - Endocrinology Surgery Unit, Hospital Universitari Vall D’Hebron, Universitat Autònoma De Barcelona, Barcelona, Spain
José María Balibrea, Enric Caubet, Oscar González, Ramón Vilallonga & Jose Manuel Fort - Dept. Bioquímica y Biología Molecular, Facultat de Biologia, Universitat de Barcelona, Diagonal 643, 08028, Barcelona, Spain
Julia Peinado-Onsurbe
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- Roser Ferrer
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Corresponding author
Correspondence toJulia Peinado-Onsurbe.
Additional information
Roser Ferrer and Eva Pardina have contributed equally to this study.
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Ferrer, R., Pardina, E., Rossell, J. et al. Morbidly “Healthy” Obese Are Not Metabolically Healthy but Less Metabolically Imbalanced Than Those with Type 2 Diabetes or Dyslipidemia.OBES SURG 25, 1380–1391 (2015). https://doi.org/10.1007/s11695-014-1528-z
- Published: 17 December 2014
- Issue Date: August 2015
- DOI: https://doi.org/10.1007/s11695-014-1528-z