ASC, a novel 22-kDa protein, aggregates during apoptosis of human promyelocytic leukemia HL-60 cells - PubMed (original) (raw)
. 1999 Nov 26;274(48):33835-8.
doi: 10.1074/jbc.274.48.33835.
Affiliations
- PMID: 10567338
- DOI: 10.1074/jbc.274.48.33835
Free article
ASC, a novel 22-kDa protein, aggregates during apoptosis of human promyelocytic leukemia HL-60 cells
J Masumoto et al. J Biol Chem. 1999.
Free article
Abstract
The cytoskeletal and/or nuclear matrix molecules responsible for morphological changes associated with apoptosis were identified using monoclonal antibodies (mAbs). We developed mAbs against Triton X-100-insoluble components of HL-60 cells pretreated with all-trans retinoic acid. In particular, one mAb recognized a 22-kDa protein that exhibited intriguing behavior by forming an aggregate and appearing as a speck during apoptosis induced by retinoic acid and other anti-tumor drugs. Cloning and sequencing of its cDNA revealed that this protein comprises 195 amino acids and that its C-terminal half has a caspase recruitment domain (CARD) motif, characteristic of numerous proteins involved in apoptotic signaling. We referred to this protein as ASC (apoptosis-associated speck-like protein containing a CARD). The ASC gene was mapped on chromosome 16p11.2-12. The antisense oligonucleotides of ASC were found to reduce the expression of ASC, and consequently, etoposide-mediated apoptosis of HL-60 cells was suppressed. Our results indicate that ASC is a novel member of the CARD-containing adaptor protein family.
Similar articles
- Interaction between pyrin and the apoptotic speck protein (ASC) modulates ASC-induced apoptosis.
Richards N, Schaner P, Diaz A, Stuckey J, Shelden E, Wadhwa A, Gumucio DL. Richards N, et al. J Biol Chem. 2001 Oct 19;276(42):39320-9. doi: 10.1074/jbc.M104730200. Epub 2001 Aug 9. J Biol Chem. 2001. PMID: 11498534 - Identification and characterization of a novel human cDNA encoding a 21 kDa pRb-associated protein.
Wen H, Ao S. Wen H, et al. Gene. 2001 Jan 24;263(1-2):85-92. doi: 10.1016/s0378-1119(00)00585-0. Gene. 2001. PMID: 11223246 - Pyrin N-terminal homology domain- and caspase recruitment domain-dependent oligomerization of ASC.
Masumoto J, Taniguchi S, Sagara J. Masumoto J, et al. Biochem Biophys Res Commun. 2001 Jan 26;280(3):652-5. doi: 10.1006/bbrc.2000.4190. Biochem Biophys Res Commun. 2001. PMID: 11162571 - Expression of apoptosis-associated speck-like protein containing a caspase recruitment domain, a pyrin N-terminal homology domain-containing protein, in normal human tissues.
Masumoto J, Taniguchi S, Nakayama J, Shiohara M, Hidaka E, Katsuyama T, Murase S, Sagara J. Masumoto J, et al. J Histochem Cytochem. 2001 Oct;49(10):1269-75. doi: 10.1177/002215540104901009. J Histochem Cytochem. 2001. PMID: 11561011 - Human EMR2, a novel EGF-TM7 molecule on chromosome 19p13.1, is closely related to CD97.
Lin HH, Stacey M, Hamann J, Gordon S, McKnight AJ. Lin HH, et al. Genomics. 2000 Jul 15;67(2):188-200. doi: 10.1006/geno.2000.6238. Genomics. 2000. PMID: 10903844
Cited by
- Targeting the NLRP3 inflammasome-IL-1β pathway in type 2 diabetes and obesity.
Meier DT, de Paula Souza J, Donath MY. Meier DT, et al. Diabetologia. 2024 Nov 4. doi: 10.1007/s00125-024-06306-1. Online ahead of print. Diabetologia. 2024. PMID: 39496966 Review. - Therapeutic Targets in Innate Immunity to Tackle Alzheimer's Disease.
Serradas ML, Ding Y, Martorell PV, Kulińska I, Castro-Gomez S. Serradas ML, et al. Cells. 2024 Aug 26;13(17):1426. doi: 10.3390/cells13171426. Cells. 2024. PMID: 39272998 Free PMC article. Review. - Disease tolerance as immune defense strategy in bats: One size fits all?
Pei G, Balkema-Buschmann A, Dorhoi A. Pei G, et al. PLoS Pathog. 2024 Sep 5;20(9):e1012471. doi: 10.1371/journal.ppat.1012471. eCollection 2024 Sep. PLoS Pathog. 2024. PMID: 39236038 Free PMC article. Review. - NLRP3 inflammasome in atherosclerosis: Mechanisms and targeted therapies.
Chen P, Li X. Chen P, et al. Front Pharmacol. 2024 Jul 31;15:1430236. doi: 10.3389/fphar.2024.1430236. eCollection 2024. Front Pharmacol. 2024. PMID: 39144618 Free PMC article. Review. - FDA-approved disulfiram inhibits the NLRP3 inflammasome by regulating NLRP3 palmitoylation.
Xu J, Pickard JM, Núñez G. Xu J, et al. Cell Rep. 2024 Aug 27;43(8):114609. doi: 10.1016/j.celrep.2024.114609. Epub 2024 Aug 7. Cell Rep. 2024. PMID: 39116210 Free PMC article.
Publication types
MeSH terms
Substances
LinkOut - more resources
Full Text Sources
Other Literature Sources
Molecular Biology Databases
Miscellaneous