Transgenic APP expression during postnatal development causes persistent locomotor hyperactivity in the adult (original) (raw)

Progressive, Age-Related Behavioral Impairments In Transgenic Mice Carrying Both Mutant Amyloid Precursor Protein and Presenilin-1 Transgenes

DAVID DIAMOND

Brain research, 2001

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Progressive, age-related behavioral impairments in transgenic mice carrying both mutant amyloid precursor protein and presenilin-1

David Diamond

Brain Res, 2001

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Wael Yunis

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Dennis Miller, Gary Weisman, Grace Sun

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Juan Troncoso

Neurobiology of Aging, 2009

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Bernd Sommer

Proceedings of the National Academy of Sciences, 1997

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Development of transgenic rats producing human β-amyloid precursor protein as a model for Alzheimer's disease: Transgene and endogenous APP genes are regulated tissue-specifically

James Lah

BMC Neuroscience, 2008

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Data on amyloid precursor protein accumulation, spontaneous physical activity, and motor learning after traumatic brain injury in the triple-transgenic mouse model of Alzheimer׳s disease

Yutaka Kirino

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Neonatal Neurodegeneration in Alzheimer’s Disease Transgenic Mouse Model

Mehmet Ozansoy

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Manfred Windisch

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Donna McPhie

Molecular Brain Research, 1999

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Age-Dependent Neuronal and Synaptic Degeneration in Mice Transgenic for the C Terminus of the Amyloid Precursor Protein

Donna McPhie

The Journal of Neuroscience

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Rita De Gasperi, George Perry

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Thomas Rosahl

2010

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Impaired neurogenesis is an early event in the etiology of familial Alzheimer's disease in transgenic mice

Yuan-Shih Jennifer Hu

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Paula Moran

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Freya Vercauteren

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Modeling Alzheimer's disease in transgenic mice: effect of age and of Presenilin1 on amyloid biochemistry and pathology in APP/London mice

F. Van Leuven

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ENU Mutagenesis Screen to Establish Motor Phenotypes in Wild-Type Mice and Modifiers of a Pre-Existing Motor Phenotype in Tau Mutant Mice

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Differential transgene expression patterns in Alzheimer mouse models revealed by novel human amyloid precursor protein-specific antibodies

Peer-hendrik Kuhn

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Transgenic Expression of the Amyloid-β Precursor Protein-Intracellular Domain Does Not Induce Alzheimer's Disease–Like Traits In Vivo

Luciano Dadamio

PLOS One, 2010

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Synaptotrophic effects of human amyloid β protein precursors in the cortex of transgenic mice

Sonja Forss-petter

Brain Research, 1994

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Selectively reduced expression of synaptic plasticity-related genes in amyloid precursor protein + presenilin-1 transgenic mice

Jeanne Loring

The Journal of neuroscience : the official journal of the Society for Neuroscience, 2003

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The Secreted -Amyloid Precursor Protein Ectodomain APPs Is Sufficient to Rescue the Anatomical, Behavioral, and Electrophysiological Abnormalities of APP-Deficient Mice

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Promoter Mutations That Increase Amyloid Precursor-Protein Expression Are Associated with Alzheimer Disease

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The American Journal of Human Genetics, 2006

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Transgenic APP expression during postnatal development causes persistent locomotor hyperactivity in the adult (original) (raw)

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