original ) (raw )Cognitive phenotyping of amyloid precursor protein transgenic J20 mice
David cheng
Behavioural Brain Research, 2012
View PDFchevron_right
Many Neuronal and Behavioral Impairments in Transgenic Mouse Models of Alzheimer's Disease Are Independent of Caspase Cleavage of the Amyloid Precursor Protein
Myo Thwin
Journal of Neuroscience, 2010
View PDFchevron_right
Intraneuronal Aβ Causes the Onset of Early Alzheimer’s Disease-Related Cognitive Deficits in Transgenic Mice
James McGaugh
Neuron, 2005
View PDFchevron_right
Overexpression of wild-type human APP in mice causes cognitive deficits and pathological features unrelated to Aβ levels
Lucio Schiapparelli , Mar Cuadrado-Tejedor
Neurobiology of Disease, 2009
View PDFchevron_right
An in vitro and in vivo study of early deficits in associative learning in transgenic mice that over-express a mutant form of human APP associated with Alzheimer's disease
Jesùs Benavides
European Journal of Neuroscience, 2004
View PDFchevron_right
Age-related learning deficits in transgenic mice expressing the 751-amino acid isoform of human beta-amyloid precursor protein
Paula Moran
Proceedings of the …, 1995
View PDFchevron_right
Progressive cognitive decline in a transgenic mouse model of Alzheimer's disease overexpressing mutant hAPPswe
Silvia Middei
Genes Brain and Behavior, 2006
View PDFchevron_right
Spatial learning and memory impairment and increased locomotion in a transgenic amyloid precursor protein mouse model of Alzheimer's disease
Dennis Miller , Gary Weisman , Grace Sun
Behavioural Brain Research, 2011
View PDFchevron_right
Deficits in synaptic transmission and learning in amyloid precursor protein (APP) transgenic mice require C-terminal cleavage of APP
V. Galvan
The Journal of …, 2006
View PDFchevron_right
Alzheimer's disease-like pathological features in transgenic mice expressing the APP intracellular domain
Jan Lehotský
Proceedings of the National Academy of Sciences, 2009
View PDFchevron_right
Two amyloid precursor protein transgenic mouse models with Alzheimer disease-like pathology
Bernd Sommer
Proceedings of the National Academy of Sciences, 1997
View PDFchevron_right
Intracellular Accumulation of Detergent-Soluble Amyloidogenic Aβ Fragment of Alzheimer's Disease Precursor Protein in the Hippocampus of Aged Transgenic Mice
Janetta Culvenor , Roberto Cappai
Journal of Neurochemistry, 2002
View PDFchevron_right
Amyloid precursor protein processing and A 42 deposition in a transgenic mouse model of Alzheimer disease
Ivan Lieberburg , Kelly Johnson-wood
Proceedings of the National Academy of Sciences, 1997
View PDFchevron_right
An Increase in Aβ42 in the Prefrontal Cortex is Associated with a Reversal-Learning Impairment in Alzheimers Disease Model Tg2576 APPsw Mice
Kumar Sambamurti
Current Alzheimer Research, 2008
View PDFchevron_right
Intracellular Aβ pathology and early cognitive impairments in a transgenic rat overexpressing human amyloid precursor protein: a multidimensional study
Christoph Weise
Acta Neuropathologica Communications, 2014
View PDFchevron_right
Progressive, age-related behavioral impairments in transgenic mice carrying both mutant amyloid precursor protein and presenilin-1
David Diamond
Brain Res, 2001
View PDFchevron_right
Characterization of cognition alteration across the course of the disease in APP751SL mice with parallel estimation of cerebral Aβ deposition
Julie Blanchard
Behavioural Brain Research, 2009
View PDFchevron_right
Progressive, Age-Related Behavioral Impairments In Transgenic Mice Carrying Both Mutant Amyloid Precursor Protein and Presenilin-1 Transgenes
DAVID DIAMOND
Brain research, 2001
View PDFchevron_right
Reversal of Alzheimer's-like pathology and behavior in human APP transgenic mice by mutation of Asp664
V. Galvan
Proceedings of the National Academy of Sciences of the United States of America, 2006
View PDFchevron_right
A transgenic rat expressing human APP with the Swedish Alzheimer’s disease mutation
Nenad Bogdanovic
Biochemical and Biophysical Research Communications, 2007
View PDFchevron_right
Age-related CNS disorder and early death in transgenic FVB/N mice overexpressing Alzheimer amyloid precursor proteins
Wael Yunis
Neuron, 1995
View PDFchevron_right
Biochemical and behavioral characterization of the double transgenic mouse model (APPswe/PS1dE9) of Alzheimer's disease
John Connelly
Neuroscience Bulletin
View PDFchevron_right
Progressive age-related development of Alzheimer-like pathology in APP/PS1 mice
Fabrizio Trinchese
Annals of Neurology, 2004
View PDFchevron_right
An Increase in Aβ42 in the Prefrontal Cortex is Associated with a Reversal-Learning Impairment in Alzheimer's Disease Model Tg2576 APPsw Mice
Michelle Nicolle
Current Alzheimer Research, 2008
View PDFchevron_right
Emergence of synaptic and cognitive impairment in a mature-onset APP mouse model of Alzheimer’s disease
Chrysia-Maria Pegasiou
Acta Neuropathologica Communications, 2019
View PDFchevron_right
Age-Dependent Neuronal and Synaptic Degeneration in Mice Transgenic for the C Terminus of the Amyloid Precursor Protein
Donna McPhie
The Journal of Neuroscience
View PDFchevron_right