Janus Faced HMGB1 and Post-Aneurysmal Subarachnoid Hemorrhage (aSAH) Inflammation (original) (raw)

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Time-courses of plasma IL-6 and HMGB-1 reflect initial severity of clinical presentation but do not predict poor neurologic outcome following subarachnoid hemorrhage

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Temporal patterns of inflammatory biomarkers measured in the cerebrospinal fluid of patients with aneurysmal subarachnoid hemorrhage using multiplex Proximity Extension Assay technology

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Inflammatory Gene Expression in Cerebral Ischemia and Trauma

Frank Barone

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Gene expression profiling reveals distinct molecular signatures associated with the rupture of intracranial aneurysm

K. Hosomichi

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What Is the Role of Inflammation? Gene Expression Profiles in Human Ruptured and Unruptured Intracranial Aneurysms

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Systemic response to rupture of intracranial aneurysms involves expression of specific gene isoforms

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Inflammation as a predictor for delayed cerebral ischemia after aneurysmal subarachnoid haemorrhage

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Glibenclamide Reduces Inflammation, Vasogenic Edema, and Caspase-3 Activation after Subarachnoid Hemorrhage

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Neurons Are a Primary Driver of Inflammation via Release of HMGB1

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Microglia as target for anti-inflammatory approaches to prevent secondary brain injury after subarachnoid hemorrhage (SAH)

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The HMGB1 Receptor RAGE Mediates Ischemic Brain Damage

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Innate immunity activation in the early brain injury period following subarachnoid hemorrhage

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Reduction of inflammation after administration of interleukin-1 receptor antagonist following aneurysmal subarachnoid hemorrhage: results of the Subcutaneous Interleukin-1Ra in SAH (SCIL-SAH) study

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NLRP3 Inflammasome Overactivation in Patients with Aneurysmal Subarachnoid Hemorrhage

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Inflammatory Mechanisms as Potential Therapeutic Targets in Stroke

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