Long-term potentiation enhancing effect of epileptic insult in the CA1 area is dependent on prior-application of primed-burst stimulation (original) (raw)

Contribution of ionotropic glutamate receptors and voltage-dependent calcium channels to the potentiation phenomenon induced by transient pentylenetetrazol in the CA1 region of rat hippocampal slices

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Pentylenetetrazol-induced Seizures in Immature Rats Provoke Long-term Changes in Adult Hippocampal Cholinergic Excitability

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Delayed Impairment of Hippocampal Synaptic Plasticity after Pentylenetetrazole-Induced Seizures in Young Rats

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Repeated administration of pentylenetetrazol alters susceptibility of rat hippocampus to primed-burst stimulation: evidence from in vitro study on CA1 of hippocampal slices

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Changes in phosphorylation of the NMDA receptor in the rat hippocampus induced by status epilepticus

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Stimulation of gene expression of NeuroD-related factor in the mouse brain following pentylenetetrazol-induced seizures

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The NMDA receptor antagonist 2-amino-5-phosphonovalerate blocks stimulus train-induced epileptogenesis but not epileptiform bursting in the rat hippocampal slice

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The Relative Contribution of NMDA Receptor Channels in the Expression of Long-term Potentiation in the Hippocampal CA1 Region

Fredrik Asztely

European Journal of Neuroscience, 1992

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Examination of persistent effects of repeated administration of pentylenetrazol on rat hippocampal CA1: evidence from in vitro study on hippocampal slices

Fereshteh Motamedi

Brain Research, 1997

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Subconvulsant doses of pentylenetetrazol uncover the epileptic phenotype of cultured synapsin-deficient Helix serotonergic neurons in the absence of excitatory and inhibitory inputs

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A Brief Period of Epileptiform Activity Strengthens Excitatory Synapses in the Rat Hippocampus in Vitro

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Imaging the Neural Substrates Involved in the Genesis of Pentylenetetrazol-induced Seizures

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CPP, an NMDA-receptor antagonist, blocks 4-aminopyridine-induced spreading depression episodes but not epileptiform activity in immature rat hippocampal slices

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Transient Morphological Alterations in the Hippocampus After Pentylenetetrazole-Induced Seizures in Rats

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NMDA receptor-mediated long-term alterations in epileptiform activity in experimental chronic epilepsy

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N-methyl-D-aspartate receptor channel blockers prevent pentylenetetrazole-induced convulsions and morphological changes in rat brain neurons

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Synaptic modifications accompanying epileptogenesis in vitro: long-term depression of GABA-mediated inhibition

Robert Wong

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Different NMDA receptor subtypes mediate induction of long-term potentiation and two forms of short-term potentiation at CA1 synapses in rat hippocampus in vitro

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The Journal of Physiology, 2013

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Epileptiform bursts elicited in CA3 hippocampal neurons by a variety of convulsants are not blocked by N-methyl-d-aspartate antagonists

Enrico Cherubini

Brain Research, 1988

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Epileptiform activity and EPSP-spike potentiation induced in rat hippocampal CA1 slices by repeated high-K+: involvement of ionotropic glutamate receptors and Ca2+/calmodulin-dependent protein kinase II

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Neuropharmacology, 2001

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Anticonvulsant effect of neural regeneration peptide 2945 on pentylenetetrazol-induced seizures in rats

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Short-Term Plasticity of Hippocampal Neuropeptides and Neuronal Circuitry in Experimental Status Epilepticus

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WIN 55,212-2 Reverted Pilocarpine-Induced Status Epilepticus Early Changes of the Interaction among 5-HT2C/NMDA/CB1 Receptors in the Rat Hippocampus

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Diphenytoin, riluzole and lidocaine: Three sodium channel blockers, with different mechanisms of action, decrease hippocampal epileptiform activity

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Long-term Potentiation of NMDA Receptor-mediated EPSP in Guinea-pig Hippocampal Slices

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European Journal of Neuroscience, 1991

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Status Epilepticus Enhances Depotentiation after Fully Established LTP in an NMDAR-Dependent but GluN2B-Independent Manner

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