Ca2+ Sparks and Ca2+ waves are the subcellular events underlying Ca2+ overload during ischemia and reperfusion in perfused intact hearts (original ) (raw )Calcium-mediated cell death during myocardial reperfusion
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Interplay between Ca2+ cycling and mitochondrial permeability transition pores promotes reperfusion‐induced injury of cardiac myocytes
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2000
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Dyssynchronous Ca 2+ Sparks in Myocytes From Infarcted Hearts
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Arrhythmogenic actions of the Ca2+ channel agonist FPL‐64716 in Langendorff‐perfused murine hearts
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Intracellular Ca2+ transients in isolated perfused rat heart: measurement using the fluorescent indicator
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Cell Calcium, 1994
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Diastolic Ca2+ overload caused by Na+/Ca2+ exchanger during the first minutes of reperfusion results in continued myocardial stunning
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European Journal of Pharmacology, 2007
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Mitochondria regulate inactivation of L‐type Ca2+channels in rat heart
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The Journal of Physiology, 2001
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Cellular Electrophysiologic Mechanisms of Cardiac Arrhythmias
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Annals of the New York Academy of Sciences, 1984
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Proceedings of the National Academy of Sciences, 2004
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Contractile dysfunction and ATP depletion after transient calcium overload in perfused ferret hearts
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The American Journal of Cardiology, 1989
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Increased Ca2+ sparks and sarcoplasmic reticulum Ca2+ stores potentially determine the spontaneous activity of pulmonary vein cardiomyocytes
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Local Ca2+ Signaling and EC Coupling in Heart: Ca2+ Sparks and the Regulation of the [Ca2+]i Transient
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Journal of Molecular and Cellular Cardiology, 2002
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Intracellular Free Ca2+ in the Course of the Ca2+ Paradox and during Poisoning Ca2+ -Selective Microelectrode Measurements in the Perfused Rat Heart
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Clinical Chemistry and Laboratory Medicine, 1986
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Na + /Ca 2+ exchanger inhibition exerts a positive inotropic effect in the rat heart, but fails to influence the contractility of the rabbit heart
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British Journal of Pharmacology, 2008
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Intracellular free Ca2+, Na+, and H+ concentrations in the isolated perfused rat heart during the Ca2+ paradox
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The kinetics of spontaneous calcium oscillations and arrhythmogenesis in the in vivo heart during ischemia/reperfusion
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[Heterogeneity of changes in functional parameters of the heart caused by ischemic-reperfusion injuries of the myocardium]
Jan Slezak
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The Annals of Thoracic Surgery, 2005
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Slow Ca2+ sparks de-synchronize Ca2+ release in failing cardiomyocytes: Evidence for altered configuration of Ca2+ release units?
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Phospholamban phosphorylation sites enhance the recovery of intracellular Ca2+ after perfusion arrest in isolated, perfused mouse heart
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Spontaneous Ca2+ sparks and Ca2+ homeostasis in a minimal model of permeabilized ventricular myocytes
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