Volume-induced increase of K+ and Cl− permeabilities in Ehrlich ascites tumor cells. Role of internal Ca2+ (original) (raw)

Separate, Ca2+-activated K+ and Cl− transport pathways in Ehrlich ascites tumor cells

Else Hoffmann

The Journal of Membrane Biology, 1986

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Ca2+-dependent K+ transport in the Ehrlich ascites tumor cell

Miguel Valdeolmillos

Biochimica et Biophysica Acta (BBA) - Biomembranes, 1982

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Swelling-activated potassium currents of Ehrlich ascites tumour cells

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Basolateral potassium membrane permeability of A6 cells and cell volume regulation

jordi ehrenfeld

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Characterization of the Increase in [Ca2+] i During Hypotonic Shock and the Involvement of Ca2+-activated K+ Channels in the Regulatory Volume Decrease in Human Osteoblast-like Cells

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Stimulation of Na+-dependent amino acid uptake by activation of the Ca2+-dependent K+ channel in the Ehrlich ascites tumor cell

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Biochimica et Biophysica Acta (BBA) - Biomembranes, 1982

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Shrinkage-induced Activation of the Na + /H + Exchanger in Ehrlich Ascites Tumor Cells: Mechanisms Involved in the Activation and a Role for the Exchanger in Cell Volume Regulation

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Journal of Membrane Biology, 1996

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Ca 2+] i oscillations induced by bradykinin in rat glioma cells associated with Ca 2+ store-dependent Ca 2+ influx are controlled by cell volume and by membrane potential

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Cytosolic free Ca2+ in daunorubicin and vincristine resistant ehrlich ascites tumor cells

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Separate Swelling- and Ca 2+ -activated Anion Currents in Ehrlich Ascites Tumor Cells

Jean Prenen, Else Hoffmann, Stine Pedersen

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KCNQ1 channels sense small changes in cell volume

Nanna Macaulay, Thomas Jespersen

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Activation of protein kinase C during cell volume regulation in Ehrlich mouse ascites tumor cells

Bo Jensen

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Calcium permeability of Ehrlich ascites tumour cell plasma membrane in vivo

A Dani

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Interactions among calcium compartments in C6 rat glioma cells: involvement of potassium channels

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Regulation of Blood-Brain Tumor Barrier Permeability by Calcium-Activated Potassium Channels

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Swelling-induced activation of Na+,K+,2Cl− cotransport in C6 glioma cells: kinetic properties and intracellular signalling mechanisms

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Cell volume and drug action: Some interactions and perspectives

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[Ca2+]i elevations detected by BK channels during Ca2+ influx and muscarine-mediated release of Ca2+ from intracellular stores in rat chromaffin cells

Murali Prakriya

The Journal of neuroscience : the official journal of the Society for Neuroscience, 1996

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Mobilization of intracellular calcium by extracellular ATP and by calcium ionophores in the Ehrlich ascites-tumour cell

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Role of AQP2 in activation of calcium entry by hypotonicity: implications in cell volume regulation

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Activation of ion transport pathways by changes in cell volume

Balazs Sarkadi

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The Na+/K+/Cl cotransport in C6 glioma cells. Properties and role in volume regulation

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Membrane Potassium Channels and Human Bladder Tumor Cells: II. Growth Properties

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Deregulation of apoptotic volume decrease and ionic movements in multidrug-resistant tumor cells: role of chloride channels

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Blockage of Intermediate-Conductance Ca2+-Activated K+Channels Inhibit Human Pancreatic Cancer Cell Growth in Vitro

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Molecular and functional aspects of anionic channels activated during regulatory volume decrease in mammalian cells*

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Regulation and possible physiological role of the Ca2-dependent K+ channel of cortical collecting ducts of the rat

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Highly co-operative Ca2+ activation of intermediate-conductance K+ channels in granulocytes from a human cell line

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Regulatory Volume Decrease and Intracellular Ca2+ in Murine Neuroblastoma Cells Studied with Fluorescent Probes

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