The Tumor Suppressor Smad4 Is Required for Transforming Growth Factor β–Induced Epithelial to Mesenchymal Transition and Bone Metastasis of Breast Cancer Cells
Clemens Lowik
Cancer Research, 2006
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The tumor suppressor Smad4 is required for transforming growth factor beta-induced epithelial to mesenchymal transition and bone metastasis of breast cancer cells
Jeroen Buijs
Cancer research, 2006
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Specific interactions between Smad proteins and AP-1 components determine TGFβ-induced breast cancer cell invasion
Carl-henrik Heldin
Oncogene, 2013
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Smad4-expression is decreased in breast cancer tissues: a retrospective study
Jürgen Dippon
BMC cancer, 2006
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Smad4-Independent TGF-β Signaling in Tumor Cell Migration
Andre Menke
Cells Tissues Organs, 2007
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Definition of smad3 phosphorylation events that affect malignant and metastatic behaviors in breast cancer cells
eunjin BAE
Cancer research, 2014
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Breast cancer bone metastasis mediated by the Smad tumor suppressor pathway
Inna Serganova
Proceedings of the National Academy of Sciences, 2005
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Smad3 is a key nonredundant mediator of transforming growth factor beta signaling in Nme mouse mammary epithelial cells
Silvia Cazzola
Molecular Cancer Research, 2009
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Activation of Canonical BMP4-SMAD7 Signaling Suppresses Breast Cancer Metastasis
Robin Anderson
Cancer Research, 2020
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The Dual Role of Smad7 in the Control of Cancer Growth and Metastasis
Veronica De Simone
International Journal of Molecular Sciences, 2013
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Evidence That Smad2 Is a Tumor Suppressor Implicated in the Control of Cellular Invasion
M. Mareel
Journal of Biological Chemistry, 1999
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Reversible Smad-Dependent Signaling between Tumor Suppression and Oncogenesis
Miki Murata
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Attenuation of Smad2 activity shows resistance to TGF-β signalling in mammary adenocarcinoma (MCF-7) cells
Suman Sengupta
Cell Biology International, 2013
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Stable Overexpression of Smad7 in Human Melanoma Cells Impairs Bone Metastasis
Khalid Mohammad
Cancer Research, 2007
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LMO4 can interact with Smad proteins and modulate transforming growth factor-β signaling in epithelial cells
Xiaoli Xu
Oncogene, 2006
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Overexpression of Smad7 Blocks Primary Tumor Growth and Lung Metastasis Development in Osteosarcoma
Marie-cécile Le Deley
Clinical Cancer Research, 2014
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Transforming Growth Factor-β1, Transforming Growth Factor-β2, and Transforming Growth Factor-β3 Enhance Ovarian Cancer Metastatic Potential by Inducing a Smad3-Dependent Epithelial-to-Mesenchymal Transition
Thúy Vy
Molecular Cancer Research, 2008
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TGF-b signaling, Smads, and tumor suppressors
Richard W Padgett
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Transforming growth factor-β1 suppresses bone morphogenetic protein-2/Smad signaling in human oral squamous cell carcinoma cell HSC-4 by downregulation of Smad1/9 expression
Masaharu Kamo
The Molecular Biology Society of Japan, 2016
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Differential effect of the expression of TGF-β pathway inhibitors, Smad-7 and Ski, on invasive breast carcinomas: relation to biologic behavior
Christina Magkou
Apmis, 2011
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Effect of Smad7 Expression on Metastasis of Mouse Mammary Carcinoma JygMC(A) Cells
Teruo Inamoto
JNCI: Journal of the National Cancer Institute, 2005
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Evidence for a Role of Smad3 and Smad2 in Stabilization of the Tumor-derived Mutant Smad2.Q407R
Céline Prunier
Journal of Biological Chemistry, 2003
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Breast cancer metastasis suppressor OTUD1 deubiquitinates SMAD7
Camila Ramirez
Nature communications, 2017
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TGF- and the Smad Signaling Pathway Support Transcriptomic Reprogramming during Epithelial-Mesenchymal Cell Transition
Carl-henrik Heldin
Molecular Biology of The Cell, 2005
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SMAD4 loss enables EGF, TGFβ1 and S100A8/A9 induced activation of critical pathways to invasion in human pancreatic adenocarcinoma cells
Paddy Tighe
Oncotarget, 2016
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Signaling of transforming growth factor-β family members through Smad proteins
Marie-Jose Goumans
European Journal of Biochemistry, 2000
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Transforming growth factor-β1 suppresses bone morphogenetic protein-2-induced mesenchymal-epithelial transition in HSC-4 human oral squamous cell carcinoma cells via Smad1/5/9 pathway suppression
Masaharu Kamo
Oncology Reports, 2016
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Smad4 Regulates Claudin-1 Expression in a Transforming Growth Factor-B-Independent Manner in Colon Cancer Cells
Amar Datta
2007
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Regulation of TGF-β signaling by Smad7
Xiaohua Yan
Acta Biochimica et Biophysica Sinica
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Homozygous deletion of SMAD4 in breast cancer cell lines and invasive ductal carcinomas
Aziza Nassar
Cancer Biology & Therapy, 2006
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A SNAIL1–SMAD3/4 transcriptional repressor complex promotes TGF-β mediated epithelial–mesenchymal transition
Jill Johnson
Nature Cell Biology, 2009
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Breast cancer anti-estrogen resistance 3 inhibits transforming growth factor β/Smad signaling and associates with favorable breast cancer disease outcomes
Jimin Guo, Charles Rajadurai
Breast Cancer Research, 2014
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