Protein Degradation Pathways Regulate the Functions of Helicases in the DNA Damage Response and Maintenance of Genomic Stability (original) (raw)
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A minimal threshold of FANCJ helicase activity is required for its response to replication stress or double-strand break repair
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BLM and the FANC proteins collaborate in a common pathway in response to stalled replication forks
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BLM helicase–dependent and –independent roles of 53BP1 during replication stress–mediated homologous recombination
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FBH1 promotes DNA double-strand breakage and apoptosis in response to DNA replication stress
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The role of the Fanconi anemia network in the response to DNA replication stress
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