original ) (raw )Phosphorylation of the Bloom's Syndrome Helicase and Its Role in Recovery from S-Phase Arrest
Alwyn Dart
Molecular and Cellular Biology, 2004
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Regulation and localization of the Bloom syndrome protein in response to DNA damage
Judith Campisi
The Journal of cell …, 2001
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Cell cycle regulation of the endogenous wild type Bloom's syndrome DNA helicase
Bruno Chatton , Jozo Delic
Oncogene, 2000
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Ubiquitin-dependent recruitment of the Bloom Syndrome helicase upon replication stress is required to suppress homologous recombination
Priyanka Modi
The EMBO Journal, 2013
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BLAP75, an essential component of Bloom's syndrome protein complexes that maintain genome integrity
CHANG XU
Embo Journal, 2005
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Point mutations causing Bloom's syndrome abolish ATPase and DNA helicase activities of the BLM protein
Bruno Chatton
Oncogene, 1998
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Bloom's syndrome protein is required for correct relocalization of RAD50/MRE11/NBS1 complex after replication fork arrest
Pietro Pichierri
The Journal of Cell Biology, 2002
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Bloom's Syndrome Helicase and Mus81 are Required to Induce Transient Double-strand DNA Breaks in Response to DNA Replication Stress
Michael Torres
Journal of Molecular Biology, 2008
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The DNA Helicase Activity of BLM Is Necessary for the Correction of the Genomic Instability of Bloom Syndrome Cells
Tianzhang Ye
Molecular Biology of the Cell, 1999
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Dephosphorylation and Subcellular Compartment Change of the Mitotic Bloom's Syndrome DNA Helicase in Response to Ionizing Radiation
lionel tintignac
Journal of Biological Chemistry, 2002
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The Bloom's Syndrome Protein (BLM) Interacts with MLH1 but Is Not Required for DNA Mismatch Repair
Kathleen Goss
Journal of Biological Chemistry, 2001
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Possible anti-recombinogenic role of Bloom's syndrome helicase in double-strand break processing
Bernard Salles
Nucleic Acids Research, 2003
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Bloom helicase is involved in DNA surveillance in early S phase in vertebrate cells
Akira Shimamoto
Oncogene, 2001
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Increased error-prone non homologous DNA end-joining – a proposed mechanism of chromosomal instability in Bloom's syndrome
Terry Gaymes
Oncogene, 2002
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MPS1-dependent mitotic BLM phosphorylation is important for chromosome stability
Hao Luo
Proceedings of the National Academy of Sciences, 2006
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Protein Degradation Pathways Regulate the Functions of Helicases in the DNA Damage Response and Maintenance of Genomic Stability
Joshua Sommers
Biomolecules, 2015
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Colocalization, physical, and functional interaction between Werner and Bloom syndrome proteins
Parimal Karmakar
Journal of Biological …, 2002
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Selective Cleavage of BLM, the Bloom Syndrome Protein, during Apoptotic Cell Death
Terumi Kohwi-Shigematsu
Journal of Biological Chemistry, 2001
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BLM helicase–dependent and –independent roles of 53BP1 during replication stress–mediated homologous recombination
Vivek Tripathi
Journal of Cell Biology, 2007
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MRN complex-dependent recruitment of ubiquitylated BLM helicase to DSBs negatively regulates DNA repair pathways
Priyanka Modi
Nature communications, 2018
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Transcriptomic and protein expression analysis reveals clinicopathological significance of Bloom's syndrome helicase (BLM) in breast cancer
Arvind Arora
Molecular cancer therapeutics, 2015
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BLAP18/RMI2, a novel OB-fold-containing protein, is an essential component of the Bloom helicase–double Holliday junction dissolvasome
Abdullah Ali
Genes & Development, 2008
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Structural basis of Bloom syndrome (BS) causing mutations in the BLM helicase domain
Jouni Väliaho
Molecular medicine (Cambridge, Mass.), 2000
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Complex activities of the human Bloom's syndrome helicase are encoded in a core region comprising the RecA and Zn-binding domains
Kata Sarlós
Nucleic Acids …, 2012
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Chk1Dependent Constitutive Phosphorylation of BLM Helicase at Serine 646 Decreases after DNA Damage
richa mudgal
Molecular Cancer Research, 2010
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Rif1 provides a new DNA-binding interface for the Bloom syndrome complex to maintain normal replication
Keli Agama
The EMBO journal, 2010
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Functional interaction between BLM helicase and 53BP1 in a Chk1-mediated pathway during S-phase arrest
somnath sengupta
Journal of Cell Biology, 2004
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The Bloom's syndrome helicase (BLM) interacts physically and functionally with p12, the smallest subunit of human DNA polymerase
Igor Stagljar
Nucleic Acids Research, 2008
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