EBNA2 Is Required for Protection of Latently Epstein-Barr Virus-Infected B Cells against Specific Apoptotic Stimuli (original) (raw)

Differential Gene Regulation by Epstein-Barr Virus Type 1 and Type 2 EBNA2

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Role of a consensus AP-2 regulatory sequence within the Epstein-Barr Virus LMP1 promoter in EBNA2 mediated transactivation

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Expression of EBNA-3 family in fresh B lymphocytes infected with Epstein-Barr virus

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Epstein-Barr virus nuclear protein EBNA3C directly induces expression of AID and somatic mutations in B cells

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Cell target genes of Epstein-Barr virus transcription factor EBNA-2: induction of the p55 regulatory subunit of PI3-kinase and its role in survival of EREB2.5 cells

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Epstein-Barr virus nuclear antigen leader protein localizes to promoters and enhancers with cell transcription factors and EBNA2

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EBNA2-EBF1 complexes promote MYC expression and metabolic processes driving S-phase progression of Epstein-Barr virus–infected B cells

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Epstein - Barr Virus Transforming Protein LMP-1 Alters B Cells Gene Expression by Promoting Accumulation of the Oncoprotein ΔNp73α

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Truncated Form of the Epstein-Barr Virus Protein EBNA-LP Protects against Caspase-Dependent Apoptosis by Inhibiting Protein Phosphatase 2A

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The Latent Membrane Protein 1 (LMP1) Oncogene of Epstein-Barr Virus Can Simultaneously Induce and Inhibit Apoptosis in B Cells

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The EBNA2-EBF1 complex promotes oncogenic MYC expression levels and metabolic processes required for cell cycle progression of Epstein-Barr virus-infected B cells

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Epstein-Barr virus latent membrane protein (LMP1) and nuclear proteins 2 and 3C are effectors of phenotypic changes in B lymphocytes: EBNA-2 and LMP1 cooperatively induce CD23

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Nuclear Factor B Binds to the Epstein-Barr Virus LMP1 Promoter and Upregulates Its Expression

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Phosphorylation sites of Epstein-Barr virus EBNA1 regulate its function

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Role of EBNA-3 Family Proteins in EBV Associated B-cell Lymphomagenesis

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B cells infected with Type 2 Epstein-Barr virus (EBV) have increased NFATc1/NFATc2 activity and enhanced lytic gene expression in comparison to Type 1 EBV infection

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Epstein-Barr virus EBNA3A and EBNA3C proteins both repress RBP-J kappa-EBNA2-activated transcription by inhibiting the binding of RBP-J kappa to DNA

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A monoclonal antibody that recognizes Epstein-Barr virus nuclear antigen 2 (EBNA 2) amino acids 1?58 does not react with EBNA 2 in native form, consistent with the self-association of EBNA 2 through the amino-terminus

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The Epstein–Barr virus nuclear protein encoded by the leader of the EBNA RNAs (EBNA-LP) is important in B-lymphocyte transformation

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