Role of reactive oxygen species (ROS) in apoptosis induction (original) (raw)

Activation of apoptosis signalling pathways by reactive oxygen species

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The late increase in intracellular free radical oxygen species during apoptosis is associated with cytochrome c release, caspase activation, and mitochondrial dysfunction

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Differential effects of Bcl-2 and caspases on mitochondrial permeabilization during endogenous or exogenous reactive oxygen species-induced cell death

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Involvement of Mitochondria in Apoptosis

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Apoptosis in mitotic competent undifferentiated cells is induced by cellular redox imbalance independent of reactive oxygen species production

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Redox Control of Apoptosis: An Update

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Reactive oxygen species are involved in FasL-induced caspase-independent cell death and inflammatory responses

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Caspase-8 Mediates Caspase-3 Activation and Cytochrome c Release during Singlet Oxygen-Induced Apoptosis of HL-60 Cells

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Apoptosis Supercedes Necrosis in Mitochondrial DNA-Depleted Jurkat Cells by Cleavage of Receptor-Interacting Protein and Inhibition of Lysosomal Cathepsin

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Caspase inhibition prevents the mitochondrial release of apoptosis-inducing factor

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Role of Reactive Oxygen Species-elicited Apoptosis in the Pathophysiology of Mitochondrial and Neurodegenerative Diseases Associated With Mitochondrial DNA Mutations

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