An Attenuated Variant of the GDVII Strain of Theiler’s Virus Does Not Persist and Does Not Infect the White Matter of the Central Nervous System (original ) (raw )The Neurovirulence of the DA and GDVII Strains of Theiler’s Virus Correlates with Their Ability To Infect Cultured Neurons
Michel Brahic
Journal of Virology, 1998
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Pathogenesis of early and late disease in mice infected with Theiler's virus, using intratypic recombinant GDVII/DA viruses
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Replacement of loop II of VP1 of the DA strain with loop II of the GDVII strain of Theiler's murine encephalomyelitis virus alters neurovirulence, viral persistence, and demyelination
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Strains from both Theiler's virus subgroups encode a determinant for demyelination
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Theiler's Murine Encephalomyelitis Virus L* Amino Acid Position 93 Is Important for Virus Persistence and Virus-Induced Demyelination
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Distribution of Theiler's virus in the CNS of athymic nude mice: Effect of varying the route of inoculation
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The Immune System Preferentially Clears Theiler's Virus from the Gray Matter of the Central Nervous System
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A three-nucleotide insertion in the H stem-loop of the 5' untranslated region of Theiler's virus attenuates neurovirulence
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Persistent infection of a glioma cell line generates a Theiler's virus variant which fails to induce demyelinating disease in SJL/J mice
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The Leader Polypeptide of Theiler's Murine Encephalomyelitis Virus Is Required for the Assembly of Virions in Mouse L Cells
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