Corticotropin-Releasing Factor and the Brain-Gut Motor Response to Stress (original) (raw)
A role for corticotropin-releasing factor in functional gastrointestinal disorders
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Role of peripheral CRF signalling pathways in stress-related alterations of gut motility and mucosal function
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Central CRF, urocortins and stress increase colonic transit via CRF1 receptors while activation of CRF2 receptors delays gastric transit in mice
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Differential stress-induced alterations of colonic corticotropin-releasing factor receptors in the Wistar Kyoto rat
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Corticotropin-Releasing Hormone Receptor 1 Antagonist Blocks Brain–Gut Activation Induced by Colonic Distention in Rats
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Differential Actions of Peripheral Corticotropin-Releasing Factor (CRF), Urocortin II, and Urocortin III on Gastric Emptying and Colonic Transit in Mice: Role of CRF Receptor Subtypes 1 and 2
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Journal of Pharmacology and Experimental Therapeutics, 2002
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Corticotropin Releasing Factor Receptor 1–Deficient Mice Display Decreased Anxiety, Impaired Stress Response, and Aberrant Neuroendocrine Development
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Alterations in colonic corticotropin-releasing factor receptors in the maternally separated rat model of irritable bowel syndrome: differential effects of acute psychological and physical stressors
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Cortagine, a CRF1 agonist, induces stresslike alterations of colonic function and visceral hypersensitivity in rodents primarily through peripheral pathways
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Role of Corticotropin-releasing Factor in Gastrointestinal Permeability
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Journal of Neurogastroenterology and Motility, 2015
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Chronic peripheral administration of corticotropin-releasing factor causes colonic barrier dysfunction similar to psychological stress
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CRF 1 receptor signaling pathways are involved in stress-related alterations of colonic function and viscerosensitivity: implications for irritable bowel syndrome
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Intracisternal injection of CRF antagonist blocks surgical stress-induced inhibition of gastric secretion in the rat
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Peptides, 1988
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Cortagine, a CRF1 agonist, induces stresslike alterations of colonic function and visceral hypersensitivity in rodents primarily through peripheral pathways
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American Journal of Physiology-gastrointestinal and Liver Physiology, 2009
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Corticotropin-releasing factor type 1 receptors mediate the visceral hyperalgesia induced by repeated psychological stress in rats
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Plasticity in the brainstem vagal circuits controlling gastric motor function triggered by corticotropin releasing factor
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2014
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The Newly Developed CRF1-Receptor Antagonists, NGD 98-2 and NGD 9002, Suppress Acute Stress-Induced Stimulation of Colonic Motor Function and Visceral Hypersensitivity in Rats
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Pathways mediating CRF-induced inhibition of gastric emptying in rats
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The corticotropin-releasing factor system in inflammatory bowel disease: Prospects for new therapeutic approaches
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Modulation of Urocortin-Induced Hypophagia and Weight Loss by Corticotropin-Releasing Factor Receptor 1 Deficiency in Mice 1
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Modulation of enteric neurons by interleukin-6 and corticotropin-releasing factor contributes to visceral hypersensitivity and altered colonic motility in a rat model of irritable bowel syndrome
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Impaired Emotional Learning and Involvement of the Corticotropin-Releasing Factor Signaling System in Patients With Irritable Bowel Syndrome
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Peripheral injection of a new corticotropin-releasing factor (CRF) antagonist, astressin, blocks peripheral CRF- and abdominal surgery-induced delayed gastric emptying in rats
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Central somatostatin receptor 1 activation reverses acute stress-related alterations of gastric and colonic motor function in mice
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Corticotropin releasing factor in the rat colon: Expression, localization and upregulation by endotoxin
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Abstracts of the International Conference on stress and digestive motility
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Digestive Diseases and Sciences, 1989
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Chronic psychosocial stress induces reversible mitochondrial damage and corticotropin-releasing factor receptor type-1 upregulation in the rat intestine and IBS-like gut dysfunction
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