Butyricicoccus pullicaecorum in inflammatory bowel disease - PubMed (original) (raw)
. 2013 Dec;62(12):1745-52.
doi: 10.1136/gutjnl-2012-303611. Epub 2012 Dec 22.
Kathleen Machiels, Clémentine Perrier, Carlos Romero, Sofie Maes, Bram Flahou, Marjan Steppe, Freddy Haesebrouck, Benedikt Sas, Richard Ducatelle, Severine Vermeire, Filip Van Immerseel
Affiliations
- PMID: 23263527
- DOI: 10.1136/gutjnl-2012-303611
Butyricicoccus pullicaecorum in inflammatory bowel disease
Venessa Eeckhaut et al. Gut. 2013 Dec.
Abstract
Objective: Many species within the phylum Firmicutes are thought to exert anti-inflammatory effects. We quantified bacteria belonging to the genus Butyricicoccus in stools of patients with ulcerative colitis (UC) and Crohn's disease (CD). We evaluated the effect of Butyricicoccus pullicaecorum in a rat colitis model and analysed the ability to prevent cytokine-induced increases in epithelial permeability.
Design: A genus-specific quantitative PCR was used for quantification of Butyricicoccus in stools from patients with UC or CD and healthy subjects. The effect of B pullicaecorum on trinitrobenzenesulfonic (TNBS)-induced colitis was assessed and the effect of B pullicaecorum culture supernatant on epithelial barrier function was investigated in vitro.
Results: The average number of Butyricicoccus in stools from patients with UC and CD in active (UC: 8.61 log10/g stool; CD: 6.58 log10/g stool) and remission phase (UC: 8.69 log10/g stool; CD: 8.38 log10/g stool) was significantly lower compared with healthy subjects (9.32 log10/g stool) and correlated with disease activity in CD. Oral administration of B pullicaecorum resulted in a significant protective effect based on macroscopic and histological criteria and decreased intestinal myeloperoxidase (MPO), tumour necrosis factor α (TNFα) and interleukin (IL)-12 levels. Supernatant of B pullicaecorum prevented the loss of transepithelial resistance (TER) and the increase in IL-8 secretion induced by TNFα and interferon γ (IFN gamma) in a Caco-2 cell model.
Conclusions: Patients with inflammatory bowel disease have lower numbers of Butyricicoccus bacteria in their stools. Administration of B pullicaecorum attenuates TNBS-induced colitis in rats and supernatant of B pullicaecorum cultures strengthens the epithelial barrier function by increasing the TER.
Keywords: Butyrate; Epithelial Barrier; IBD; Inflammation; Probiotics.
Comment in
- Butyrate-producing bacteria as pharmabiotics for inflammatory bowel disease.
Marteau P. Marteau P. Gut. 2013 Dec;62(12):1673. doi: 10.1136/gutjnl-2012-304240. Epub 2013 Mar 5. Gut. 2013. PMID: 23461897 No abstract available. - Progress towards butyrate-producing pharmabiotics: Butyricicoccus pullicaecorum capsule and efficacy in TNBS models in comparison with therapeutics.
Eeckhaut V, Ducatelle R, Sas B, Vermeire S, Van Immerseel F. Eeckhaut V, et al. Gut. 2014 Feb;63(2):367. doi: 10.1136/gutjnl-2013-305293. Epub 2013 Jun 13. Gut. 2014. PMID: 23766442 No abstract available.
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