The DNA Helicase Activity of BLM Is Necessary for the Correction of the Genomic Instability of Bloom Syndrome Cells (original) (raw)
Cell cycle regulation of the endogenous wild type Bloom's syndrome DNA helicase
Bruno Chatton, Jozo Delic
Oncogene, 2000
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Point mutations causing Bloom's syndrome abolish ATPase and DNA helicase activities of the BLM protein
Bruno Chatton
Oncogene, 1998
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Bloom helicase is involved in DNA surveillance in early S phase in vertebrate cells
Akira Shimamoto
Oncogene, 2001
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Regulation and localization of the Bloom syndrome protein in response to DNA damage
Judith Campisi
The Journal of cell …, 2001
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Transfection of BLM into Cultured Bloom Syndrome Cells Reduces the Sister-Chromatid Exchange Rate toward Normal
Tianzhang Ye
The American Journal of Human Genetics, 1999
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Processive translocation mechanism of the human Bloom's syndrome helicase along single-stranded DNA
Kata Sarlós
Nucleic acids research, 2010
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Dephosphorylation and Subcellular Compartment Change of the Mitotic Bloom's Syndrome DNA Helicase in Response to Ionizing Radiation
lionel tintignac
Journal of Biological Chemistry, 2002
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Increased error-prone non homologous DNA end-joining – a proposed mechanism of chromosomal instability in Bloom's syndrome
Terry Gaymes
Oncogene, 2002
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Possible anti-recombinogenic role of Bloom's syndrome helicase in double-strand break processing
Bernard Salles
Nucleic Acids Research, 2003
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Control of Translocations between Highly Diverged Genes by Sgs1, the Saccharomyces cerevisiae Homolog of the Bloom's Syndrome Protein
Joan Wu
Molecular and Cellular Biology, 2006
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The Bloom's syndrome gene product is homologous to RecQ helicases
Tianzhang Ye
Cell, 1995
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The Bloom's Syndrome Protein (BLM) Interacts with MLH1 but Is Not Required for DNA Mismatch Repair
Kathleen Goss
Journal of Biological Chemistry, 2001
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Phosphorylation of the Bloom's Syndrome Helicase and Its Role in Recovery from S-Phase Arrest
Alwyn Dart
Molecular and Cellular Biology, 2004
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Structural basis of Bloom syndrome (BS) causing mutations in the BLM helicase domain
Jouni Väliaho
Molecular medicine (Cambridge, Mass.), 2000
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BLAP75, an essential component of Bloom's syndrome protein complexes that maintain genome integrity
CHANG XU
Embo Journal, 2005
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SGSl, a Homologue of the Bloom's and Werner's Syndrome Genes, Is Required for Maintenance of Genome Stability in Saccharomyces cereuiSiae
Edward Louist, Paul Watt
2000
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Direct association of Bloom's syndrome gene product with the human mismatch repair protein MLH1
Igor Stagljar
Nucleic Acids Research, 2001
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Mitotic phosphorylation of Bloom helicase at Thr182 is required for its proteasomal degradation and maintenance of chromosomal stability
Vivek Srivastava
Oncogene, 2015
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Implications of somatic recombination and sister chromatid exchange in Bloom's syndrome and cells treated with mitomycin C
David Comings
Human Genetics, 1975
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Chromosomal instability associated with a novel BLM frameshift mutation (c.1980-1982delAA) in two unrelated Tunisian families with Bloom syndrome
F. Fakhfakh
Journal of the European Academy of Dermatology and Venereology, 2013
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A role for PML and the nuclear body in genomic stability
Tianzhang Ye
Oncogene, 1999
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Visualization of human Bloom's syndrome helicase molecules bound to homologous recombination intermediates
Mihály Kovács
The FASEB Journal, 2013
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Bloom's and Werner's syndrome genes suppress hyperrecombination in yeast sgs1 mutant: Implication for genomic instability in human diseases
Akira Shimamoto
Proceedings of the National Academy of Sciences, 1998
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A Small Molecule Inhibitor of the BLM Helicase Modulates Chromosome Stability in Human Cells
Csanad Z. Bachrati, Giang Nguyen
Chemistry & Biology, 2013
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Complex activities of the human Bloom's syndrome helicase are encoded in a core region comprising the RecA and Zn-binding domains
Kata Sarlós
Nucleic Acids …, 2012
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Mobile D-loops are a preferred substrate for the Bloom's syndrome helicase
Csanad Z. Bachrati, Rhona Borts
Nucleic Acids Research, 2006
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The distribution and expression of the Bloom's syndrome gene product in normal and neoplastic human cells
Marta Canamero
British journal of cancer, 2001
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Ubiquitin-dependent recruitment of the Bloom Syndrome helicase upon replication stress is required to suppress homologous recombination
Priyanka Modi
The EMBO Journal, 2013
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Stability of microsatellites and minisatellites in Bloom syndrome, a human syndrome of genetic instability
F. Praz
Mutation Research/DNA Repair, 1996
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Mechanisms by Which Bloom Protein Can Disrupt Recombination Intermediates of Okazaki Fragment Maturation
Jeremy Bartos
Journal of Biological Chemistry, 2006
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Genome stability: Failure to unwind causes cancer
Paul Watt
Current Biology, 1996
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Protease inhibitors reduce the frequency of spontaneous chromosome abnormalities in cells from patients with Bloom syndrome
Ann Kennedy
Proceedings of the National Academy of Sciences, 1984
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Binding and Melting of D-Loops by the Bloom Syndrome Helicase
Tianzhang Ye
Biochemistry, 2000
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