EBF1 binds to EBNA2 and promotes the assembly of EBNA2 chromatin complexes in B cells (original) (raw)

Transcription Profiling of Epstein-Barr Virus Nuclear Antigen (EBNA)-1 Expressing Cells Suggests Targeting of Chromatin Remodeling Complexes

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PLoS ONE, 2010

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EBNA2-EBF1 complexes promote MYC expression and metabolic processes driving S-phase progression of Epstein-Barr virus–infected B cells

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Identification of Epstein-Barr Virus (EBV) Nuclear Antigen 2 (EBNA2) Target Proteins by Proteome Analysis: Activation of EBNA2 in Conditionally Immortalized B Cells Reflects Early Events after Infection of Primary B Cells by EBV

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Journal of Virology, 2004

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The EBNA2-EBF1 complex promotes oncogenic MYC expression levels and metabolic processes required for cell cycle progression of Epstein-Barr virus-infected B cells

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Epstein-Barr virus nuclear protein EBNA3C directly induces expression of AID and somatic mutations in B cells

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Epstein-Barr virus nuclear antigen leader protein localizes to promoters and enhancers with cell transcription factors and EBNA2

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Epstein-Barr virus nuclear antigen 2 (EBNA2) extensively rewires the human chromatin landscape at autoimmune risk loci

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Epstein–Barr virus nuclear antigen 2 extensively rewires the human chromatin landscape at autoimmune risk loci

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Defining B Cell Chromatin: Lessons from EBF1

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Epstein-Barr virus exploits intrinsic B-lymphocyte transcription programs to achieve immortal cell growth

Jon Aster, B. Zhao

Proceedings of the National Academy of Sciences, 2011

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Identification and cloning of a novel chromatin-associated protein partner of Epstein–Barr nuclear protein 2

Tobias Ragoczy

Experimental Cell Research, 2004

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Epstein-Barr virus EBNA3A and EBNA3C proteins both repress RBP-J kappa-EBNA2-activated transcription by inhibiting the binding of RBP-J kappa to DNA

Evelyne Manet

Journal of virology, 1996

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Differential Hyperacetylation of Histones H3 and H4 upon Promoter-Specific Recruitment of EBNA2 in Epstein-Barr Virus Chromatin

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Journal of Virology, 2003

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Host-cell-phenotype-dependent control of the BCR2/BWR1 promoter complex regulates the expression of Epstein-Barr virus nuclear antigens 2-6

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Proceedings of the National Academy of Sciences, 1992

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EBNA1 regulates cellular gene expression by binding cellular promoters

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Proceedings of The National Academy of Sciences, 2009

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EBV genome carrying B lymphocytes that express the nuclear protein EBNA-2 but not LMP-1

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Shared and distinct interactions of type 1 and type 2 Epstein-Barr Nuclear Antigen 2 with the human genome

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Differential Gene Regulation by Epstein-Barr Virus Type 1 and Type 2 EBNA2

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Epstein-Barr Virus Episome Physically Interacts with Active Regions of the Host Genome in Lymphoblastoid Cells

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A multi-omics approach to Epstein-Barr virus immortalization of B-cells reveals EBNA1 chromatin pioneering activities targeting nucleotide metabolism

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Functional Analyses of the EBNA1 Origin DNA Binding Protein of Epstein-Barr Virus

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Journal of Virology, 2000

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Role of epigenetics in EBV regulation and pathogenesis

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Future Microbiology, 2014

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Transcriptional repression by the Epstein-Barr virus EBNA3A protein tethered to DNA does not require RBP-Jj

Evelyne Manet

2000

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The amino terminus of Epstein-Barr Virus (EBV) nuclear antigen 1 contains AT hooks that facilitate the replication and partitioning of latent EBV genomes by tethering them to cellular chromosomes

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Journal of Virology, 2004

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The Epstein–Barr virus nuclear protein encoded by the leader of the EBNA RNAs (EBNA-LP) is important in B-lymphocyte transformation

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Epstein-Barr virus nuclear antigen 2 induces FcRH5 expression through CBF1

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Epstein-Barr virus nuclear antigen 2 inhibits AID expression during EBV-driven B-cell growth

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Role of EBNA-3 Family Proteins in EBV Associated B-cell Lymphomagenesis

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Frontiers in Microbiology, 2016

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EBNA2 Is Required for Protection of Latently Epstein-Barr Virus-Infected B Cells against Specific Apoptotic Stimuli

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The EBV nuclear antigen 1 (EBNA1) enhances B cell immortalization several thousandfold

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An Atlas of the Epstein-Barr Virus Transcriptome and Epigenome Reveals Host-Virus Regulatory Interactions

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The Gly–Ala repeat modulates the interaction of Epstein–Barr virus nuclear antigen-1 with cellular chromatin

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High-resolution Methylation Analysis and In Vivo Protein–DNA Binding at the Promoter of the Viral Oncogene LMP2A in B Cell Lines Carrying Latent Epstein–Barr Virus Genomes

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Virus Genes, 2003

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Epigenetic regulation of latent Epstein–Barr virus promoters

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