The Epstein-Barr Virus Regulome in Lymphoblastoid Cells (original) (raw)

Epstein-Barr Virus Episome Physically Interacts with Active Regions of the Host Genome in Lymphoblastoid Cells

Journal of Virology, 2020

Epstein-Barr virus exploits intrinsic B-lymphocyte transcription programs to achieve immortal cell growth

Proceedings of the National Academy of Sciences, 2011

Epstein-Barr virus nuclear antigen leader protein localizes to promoters and enhancers with cell transcription factors and EBNA2

Hufeng Zhou

Proceedings of the National Academy of Sciences, 2013

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An Atlas of the Epstein-Barr Virus Transcriptome and Epigenome Reveals Host-Virus Regulatory Interactions

Kevin Tsai

Cell Host & Microbe, 2012

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The EBNA2-EBF1 complex promotes oncogenic MYC expression levels and metabolic processes required for cell cycle progression of Epstein-Barr virus-infected B cells

Lucas Wange

2021

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Epstein-Barr Virus Nuclear Protein EBNA3A Is Critical for Maintaining Lymphoblastoid Cell Line Growth

ANDREW Cooper

Journal of Virology, 2003

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Cellular expression profiles of Epstein-Barr virus-transformed B-lymphoblastoid cell lines

Ratakorn Srisuttee

Biomedical Reports, 2020

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Transcription Profiling of Epstein-Barr Virus Nuclear Antigen (EBNA)-1 Expressing Cells Suggests Targeting of Chromatin Remodeling Complexes

Maria Masucci

PLoS ONE, 2010

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Epstein–Barr virus nuclear antigen 3C regulated genes in lymphoblastoid cell lines

B. Gewurz

2011

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Gene array identification of Epstein Barr virus-regulated cellular genes in EBV-converted Burkitt lymphoma cell lines

Martine Raphael

Laboratory Investigation, 2002

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Functional Implications of Epstein-Barr Virus Lytic Genes in Carcinogenesis

Lee Yap

Cancers

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Carcinoma-risk variant of EBNA1 deregulates Epstein-Barr Virus episomal latency

Troy Messick

Oncotarget, 2017

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Epstein-Barr virus nuclear protein EBNA3C directly induces expression of AID and somatic mutations in B cells

Kostas Paschos, Paul Kellam, Rachael Bashford-Rogers

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EBNA2-EBF1 complexes promote MYC expression and metabolic processes driving S-phase progression of Epstein-Barr virus–infected B cells

Lucas Wange

Proceedings of the National Academy of Sciences

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Pathogenic roles for Epstein–Barr virus (EBV) gene products in EBV-associated proliferative disorders

Jaap Middeldorp

Critical Reviews in Oncology/Hematology, 2003

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Functional interplay of Epstein-Barr virus oncoproteins in a mouse model of B cell lymphomagenesis

Klaus Rajewsky

Proceedings of the National Academy of Sciences, 2020

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Epstein-Barr virus nuclear antigen proteins deploy diverse mechanisms to bind the human genome

Redwanul Haque

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Epstein-Barr virus super-enhancer eRNAs are essential for MYC oncogene expression and lymphoblast proliferation

Kenneth Kaye

Proceedings of the National Academy of Sciences of the United States of America, 2016

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Genome-wide analysis of host-chromosome binding sites for Epstein-Barr Virus Nuclear Antigen 1 (EBNA1)

Louise Showe

Virology Journal, 2010

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Epstein-Barr Virus Nuclear Protein 3A Domains Essential for Growth of Lymphoblasts: Transcriptional Regulation through RBP-J /CBF1 Is Critical

ANDREW Cooper

Journal of Virology, 2005

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Phosphorylation sites of Epstein-Barr virus EBNA1 regulate its function

Dick Burgess

Journal of General Virology, 2009

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Epstein–Barr virus nuclear antigen 1 activates transcription from episomal but not integrated DNA and does not alter lymphocyte growth

Siu Chun Hung

Proceedings of the National Academy of Sciences, 2001

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Genome Diversity of Epstein-Barr Virus from Multiple Tumour Types and Normal Infection

Lawrence Young

Journal of Virology, 2015

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A Genome-Wide Integrative Genomic Study Localizes Genetic Factors Influencing Antibodies against Epstein-Barr Virus Nuclear Antigen 1 (EBNA-1)

Nikhil Dhurandhar

PLoS genetics, 2013

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Epstein–Barr virus and oncogenesis: from latent genes to tumours

Lawrence Young

Oncogene, 2003

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The copy number of Epstein-Barr virus latent genome correlates with the oncogenicity by the activation level of LMP1 and NF-κB

Hongzhuan Wu

Oncotarget, 2015

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RESTRICTED EPSTEIN-BARR-VIRUS PROTEIN EXPRESSION IN BURKITT-LYMPHOMA IS DUE TO A DIFFERENT EPSTEIN-BARR NUCLEAR ANTIGEN-1 TRANSCRIPTIONAL INITIATION SITE

Elliott Kieff

PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1991

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LMP1-deficient Epstein-Barr virus mutant requires T cells for lymphomagenesis

Fotis Asimakopoulos

Journal of Clinical Investigation, 2014

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Repression of the Proapoptotic Cellular BIK/NBK Gene by Epstein-Barr Virus Antagonizes Transforming Growth Factor 1-Induced B-Cell Apoptosis

Eva Campion

Journal of Virology, 2014

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Role of EBNA-3 Family Proteins in EBV Associated B-cell Lymphomagenesis

SHATADRU GHOSH ROY

Frontiers in Microbiology, 2016

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Host Genetic Variants and Gene Expression Patterns Associated with Epstein-Barr Virus Copy Number in Lymphoblastoid Cell Lines

Astrid Gall, Paul Kellam, Charlotte Houldcroft

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A cancer-associated Epstein-Barr virus BZLF1 promoter variant enhances lytic infection

Shannon Kenney

PLoS pathogens, 2018

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Epstein-Barr Virus EBNA-3C Is Targeted to and Regulates Expression from the Bidirectional LMP-1/2B Promoter

Andrew Brooks

Journal of Virology, 2006

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Epstein-Barr virus nuclear antigen 3C putative repression domain mediates coactivation of the LMP1 promoter with EBNA-2

Sam Lin

Journal of virology, 2002

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The Epstein–Barr virus LMP1 amino acid sequence that engages tumor necrosis factor receptor associated factors is critical for primary B lymphocyte growth transformation

Kenneth Kaye

Proceedings of the National Academy of Sciences, 1997

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The Epstein-Barr Virus Regulome in Lymphoblastoid Cells (original) (raw)

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