Adenoma-linked barrier defects and microbial products drive IL-23/IL-17-mediated tumour growth (original) (raw)
Change history
07 November 2012
The FACS image in Fig. 1e was corrected; scale bars in Figs 3b, e and f and 4a, b and f were amended.
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Acknowledgements
We thank eBioscience, GeneTex, Santa Cruz, BioLegend and Cell Signaling for antibodies; Genentech and Amgen for Il23 −/− and Il17ra −/− mice, respectively, and S. Reid and E. Southon for the help in generating _Il23r_F/F mice. This work was supported by Crohn’s and Colitis Foundation of America (Career Development Award number 2693), NIH/National Institute of Diabetes and Digestive and Kidney Diseases (K99-DK088589) and a University of California, San Diego, Digestive Disease Research Development Center Pilot Grant (DK080506) to S.I.G.; Croucher Foundation and China Postdoctoral Science Foundation (20110490919) to K.W.; Strategic Young Researcher Overseas Visits Program for Accelerating Brain Circulation to K.T.; SPAR Austria to C.D.; NIH (R01CA082223) to E.R.F.; and NIH (AI043477; DK035108) and American Association for Cancer Research (07-60-21-KARI) grants to M.K., who is an American Cancer Society Research Professor. The content is solely the responsibility of the authors and does not necessarily represent the official views of the NIH.
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Author notes
- Sergei I. Grivennikov and Kepeng Wang: These authors contributed equally to this work.
Authors and Affiliations
- Departments of Pharmacology and Pathology, Laboratory of Gene Regulation and Signal Transduction, School of Medicine, University of California, San Diego, 9500 Gilman Drive, La Jolla, California 92093-0723, USA,
Sergei I. Grivennikov, Kepeng Wang, Dominik Jauch, Koji Taniguchi, Guann-Yi Yu & Michael Karin - Biomedical Research Institute, Shenzhen-PKU-HKUST Medical Center, No. 1120, Lianhua Road, Shenzhen, Guangdong Province, China,
Kepeng Wang - La Jolla Institute for Allergy and Immunology, La Jolla, 92093, California, USA
Daniel Mucida & Hilde Cheroutre - Laboratory of Mucosal Immunology, The Rockefeller University, New York, 10065, New York, USA
Daniel Mucida - Cancer and Inflammation Program, Laboratory of Experimental Immunology, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Frederick, 21702-1201, Maryland, USA
C. Andrew Stewart & Giorgio Trinchieri - Department of Medicine, School of Medicine, University of California, San Diego, 9500 Gilman Drive, La Jolla, California 92093-0723, USA,
Bernd Schnabl, Christoph H. Österreicher & Lars Eckmann - Department of Microbiology and Immunology, Keio University School of Medicine, Tokyo 160-8582, Japan,
Koji Taniguchi - Institute of Pharmacology, Center for Physiology and Pharmacology Medical University of Vienna, Vienna, Austria
Christoph H. Österreicher - Department of Medicine, Tufts Medical Center, Boston, 02111, Massachusetts, USA
Kenneth E. Hung - Department of Internal Medicine, Oberndorf Hospital, Paracelsus Medical University, Salzburg, Austria
Christian Datz - Departments of Internal Medicine, Human Genetics and Pathology, University of Michigan Medical School, Ann Arbor, 48109, Michigan, USA
Ying Feng & Eric R. Fearon - Seattle Children’s Research Institute, Seattle, 98105, Washington, USA
Mohamed Oukka - Mouse Cancer Genetics Program, National Cancer Institute, National Institutes of Health, Frederick, 21702-1201, Maryland, USA
Lino Tessarollo - Department of Molecular Virology, Immunology & Medical Genetics, Ohio State University Comprehensive Cancer Center, Wexner Medical Center, Columbus, 43210, Ohio, USA
Vincenzo Coppola - Department of Immunology, University of Texas Southwestern Medical Center at Dallas, Dallas, 75390, Texas, USA
Felix Yarovinsky
Authors
- Sergei I. Grivennikov
- Kepeng Wang
- Daniel Mucida
- C. Andrew Stewart
- Bernd Schnabl
- Dominik Jauch
- Koji Taniguchi
- Guann-Yi Yu
- Christoph H. Österreicher
- Kenneth E. Hung
- Christian Datz
- Ying Feng
- Eric R. Fearon
- Mohamed Oukka
- Lino Tessarollo
- Vincenzo Coppola
- Felix Yarovinsky
- Hilde Cheroutre
- Lars Eckmann
- Giorgio Trinchieri
- Michael Karin
Contributions
S.G. and M.K. conceived the project. S.I.G., K.W., D.M., B.S., D.J., K.T., G.Y.Y., C.O., Y.F. and K.E.H. performed the experiments. S.I.G., K.W., D.M., D.J., H.C., L.E. and M.K. analysed data. C.A.S., V.C., L.T. and G.T. generated _Il23r_F/F mice. M.O. and F.Y. provided Il23r gfp/gfp and _Tlr2,4,9_−/− bone marrow, respectively, and Y.F. and E.R.F. provided CPC-APC mice and tissues from _Cdx2_ERT-Cre-APC mice. C.A.S., E.R.F., H.C. and G.T. provided conceptual advice. C.D. collected and provided human specimens. S.I.G., K.W. and M.K. wrote the manuscript, with all authors contributing to the writing and providing advice.
Corresponding author
Correspondence toMichael Karin.
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The authors declare no competing financial interests.
Supplementary information
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This file contains Supplementary Table 1 and Supplementary Figures 1-11. Supplementary Figs 1e, 2b and 8a were corrected on 07 November 2012. (PDF 2192 kb)
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Grivennikov, S., Wang, K., Mucida, D. et al. Adenoma-linked barrier defects and microbial products drive IL-23/IL-17-mediated tumour growth.Nature 491, 254–258 (2012). https://doi.org/10.1038/nature11465
- Received: 08 February 2012
- Accepted: 31 July 2012
- Published: 07 November 2012
- Issue date: 08 November 2012
- DOI: https://doi.org/10.1038/nature11465