Meta-analysis of expression signatures of muscle atrophy: gene interaction networks in early and late stages (original) (raw)

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Role for IκBα, but not c-Rel, in skeletal muscle atrophy

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Evidence of MyomiR network regulation of β-myosin heavy chain gene expression during skeletal muscle atrophy

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Aging Affects the Transcriptional Regulation of Human Skeletal Muscle Disuse Atrophy

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Nuclear factor- B signalling and transcriptional regulation in skeletal muscle atrophy

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Introduction to the Special Issue “Skeletal Muscle Atrophy: Mechanisms at a Cellular Level”

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Nuclear factor-kappa B signaling in skeletal muscle atrophy

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Evidence of MyomiR network regulation of -myosin heavy chain gene expression during skeletal muscle atrophy

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TWEAK and TRAF6 regulate skeletal muscle atrophy

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Different atrophy-hypertrophy transcription pathways in muscles affected by severe and mild spinal muscular atrophy

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Foxo transcription factors induce the atrophy-related ubiquitin ligase atrogin-1 and cause skeletal muscle atrophy

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Genome-Wide Atlas of Promoter Expression Reveals Contribution of Transcribed Regulatory Elements to Genetic Control of Disuse-Mediated Atrophy of Skeletal Muscle

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Skeletal muscle wasting with disuse atrophy is multi-dimensional: the response and interaction of myonuclei, satellite cells and signaling pathways

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Review Article: Mechanisms and Strategies to Counter Muscle Atrophy

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JunB transcription factor maintains skeletal muscle mass and promotes hypertrophy

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Recent progress in elucidating signalling proteolytic pathways in muscle wasting: Potential clinical implications

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Role for I B , but not c-Rel, in skeletal muscle atrophy

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The IGF-1/PI3K/Akt pathway prevents expression of muscle atrophy-induced ubiquitin ligases by inhibiting FOXO transcription factors

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